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1,501 results for “fatty acid”
Strategic delivery of Omega-3 fatty acids for modulating inflammatory neurodegenerative diseases
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Integrating pigment and fatty acid profiles for enhanced estimation of seston community composition
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Data from: Microbial composition play the leading role in volatile fatty acid production in the fermentation of different scale of corn stover with rumen fluid
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Phospholipid fatty acids (PFLA) on decomposed litter: Nitrogen limitation in decomposition
Modern agriculture and fossil fuel combustion contribute to the transfer of N from largely inert pools (atmospheric N2, fossil fuel reserves) to biologically reactive forms that can be transported downwind from agricultural or industrial areas to ecosystems that historically may have experienced low levels of N inputs. Understanding how increased N inputs alter the cycling of another biologically important element, C, has been impeded by uncertainties about N effects on the process of decomposition. To date, ecologists remain unable to predict when, where, and in what forms N addition stimulates rates of decomposition. For example, recent work showed that in eight low-N sites in Central Minnesota, litter N was positively correlated with decomposition, suggesting N limitation of decomposition, yet addition of inorganic N fertilizer increased decomposition in only two of eight sites. These paradoxical results call into question the assumption that the often-observed correlation between substrate N concentration and decomposition arises because N limits decomposition. Research is addressing three interrelated questions:* (1) Why do litter N and externally supplied N have contrasting effects on decomposition in low-N ecosystems? (2) Do different forms of N (organic vs. inorganic; substrate vs. externally supplied) affect the activity, function and composition of the decomposer community differently, and, if so, what are the consequences for decomposition? (3) What are temporal dynamics of the activity, function, and composition of the decomposer community and do these dynamics depend upon the amount and forms of N supplied to the decomposer community?* These questions will be addressed using a 4-y decomposition experiment manipulating the quantity and form of N available to decomposers via use of substrates ranging in N concentrations and of inorganic (ammonium nitrate) and organic (amino acids) N fertilizers. The response of microbial biomass, stoichiometry, efficiency
SGS-LTER Graduate Student Research: Phospholipid fatty acid (PFLA) as Biochemical Responses of US Great Plains Grasslands to Regional and Interannual Variability in Precipitation (1999-2001)
This data package was produced by researchers working on the Shortgrass Steppe Long Term Ecological Research (SGS-LTER) Project, administered at Colorado State University. Long-term datasets and background information (proposals, reports, photographs, etc.) on the SGS-LTER project are contained in a comprehensive project collection within the Digital Collections of Colorado (http://digitool.library.colostate.edu/R/?func=collections&collection_id=3429). The data table and associated metadata document, which is generated in Ecological Metadata Language, may be available through other repositories serving the ecological research community and represent components of the larger SGS-LTER project collection. Additional information and referenced materials can be found: http://hdl.handle.net/10217/85531. Carbon (C) sequestration potential in grasslands is thought to be high due to the large soil organic carbon pools characteristic of these ecosystems. Inputs of C (aboveground net primary productivity) are highly correlated to precipitation across the Great Plains region; however, changes in C pool size at a specific site are governed by the relative input and output rates across time. Our objective was to quantify the ecosystem C response of three grassland community types (shortgrass steppe, mixed grass and tallgrass prairie) to interannual variation in precipitation. At five sites across a precipitation gradient in the Great Plains, we measured net primary production (NPP), soil respiration (SRESP), and litter decomposition rates for three consecutive years. NPP, SRESP, and litter decomposition increased from shortgrass steppe (175, 454, and 47 g C m-2 yr-1) to tallgrass prairie (408, 1221, and 348 g C m-2 yr-1 for NPP, SRESP, and litter decomposition respectively). Increased growing season precipitation between study years resulted in increased NPP, SRESP, and litter decomposition at almost all sites. However, the regional patterns of the interannual NPP, SRESP, and lit
Data_Figure 3_Impact of 17β‑HSD12, the 3‑ketoacyl‑CoA reductase of long‑chain fatty acid synthesis, on breast cancer cell proliferation and migration
<p>Data of figure 3 from Impact of 17β‑HSD12, the 3‑ketoacyl‑CoA reductase of long‑chain fatty acid synthesis, on breast cancer cell proliferation and migration</p> <p>Dataset (doi: 10.1007/s00018-019-03227-w) contains the original figure as TIF-format (10.1194_jlr.M092908_Fig. 3). Corresponding raw data obtained from a) cellomics HTC array scan analysis provided as three files in CSV format (31003A-179400_Date_examiner_17BHSD12_8_2_1-3). Migration potential as three files in CSV format (31003A-179400_ date_examiner_17BHSD12_16_2_1-3); b) mRNA content analyzed by RT-PCR provided as six files in CSV format (31003A-179400_date_examiner_17BHSD12_1_3_1-6); c) western blot and densitometry provided as eight files in CSV format 31003A-179400_date_examiner_2_1-2_1-8). All further experiment related information protocols and subsequent data analysis provided as meta-data-files (31003A-179400_date_examiner_17BHSD12_8/16/1_dataset_M_1) as TXT format and (31003A-179400_date_examiner_17BHSD12_2_dataset_M_2-3) as PNG format.</p>
Data_supplemental figure 7_Impact of 17β‑HSD12, the 3‑ketoacyl‑CoA reductase of long‑chain fatty acid synthesis, on breast cancer cell proliferation and migration
<p>Data of supplemental figure 7 from Impact of 17β‑HSD12, the 3‑ketoacyl‑CoA reductase of long‑chain fatty acid synthesis, on breast cancer cell proliferation and migration</p> <p>Dataset (doi: 10.1007/s00018-019-03227-w) contains the original figure as TIF-format (10.1194_jlr.M092908_Fig. S7). Corresponding raw data obtained from xCELLigence provided as six files in CSV format (31003A-179400_date_examiner_17BHSD12_9_3-4_1-3). All further experiment related information and subsequent data analysis provided as a) two meta-data-file 31003A-179400_ date_examiner_17BHSD12_9_3-4_M_1) as TXT format.</p>
Data_supplemental figure 3_Impact of 17β‑HSD12, the 3‑ketoacyl‑CoA reductase of long‑chain fatty acid synthesis, on breast cancer cell proliferation and migration
<p>Data of supplemental figure 3 from Impact of 17β‑HSD12, the 3‑ketoacyl‑CoA reductase of long‑chain fatty acid synthesis, on breast cancer cell proliferation and migration</p> <p>Dataset (doi: 10.1007/s00018-019-03227-w) contains the original figure as TIF-format (10.1194_jlr.M092908_Fig. S3). Corresponding raw data from a) western blot and densitometry provided as 11 files in CSV format (31003A-179400_date_examiner_17BHSD12_2_18-20_1-3) b) cellomics HTC array scan analysis provided as four files in CSV format (31003A-179400_Date_examiner_17BHSD12_8_13_1-4). All further experiment related information protocols and subsequent data analysis provided as meta-data-files (31003A-179400_date_examiner_17BHSD12_2/8_dataset_M_1) as TXT format and (31003A-179400_date_examiner_17BHSD12_2_dataset_M_2-3) as PNG format.</p>
Data_supplemental figure 6_Impact of 17β‑HSD12, the 3‑ketoacyl‑CoA reductase of long‑chain fatty acid synthesis, on breast cancer cell proliferation and migration
<p>Data of supplemental figure 6 from Impact of 17β‑HSD12, the 3‑ketoacyl‑CoA reductase of long‑chain fatty acid synthesis, on breast cancer cell proliferation and migration</p> <p>Dataset (doi: 10.1007/s00018-019-03227-w) contains the original figure as TIF-format (10.1194_jlr.M092908_Fig. S6). Corresponding raw data from a-c) cellomics HTC array scan analysis provided as 19 files in CSV format (31003A-179400_Date_examiner_17BHSD12_8_14-17_1-5), d) western blot and densitometry provided as five files in CSV format (31003A-179400_date_examiner_17BHSD12_2_21_1-5). All further experiment related information protocols and subsequent data analysis provided as meta-data-files (31003A-179400_date_examiner_17BHSD12_2/8_dataset_M_1) as TXT format and (31003A-179400_date_examiner_17BHSD12_2_dataset_M_2-3) as PNG format.</p>
Data_Figure 1_Impact of 17β‑HSD12, the 3‑ketoacyl‑CoA reductase of long‑chain fatty acid synthesis, on breast cancer cell proliferation and migration
<p>Data of figure 1 from Impact of 17β‑HSD12, the 3‑ketoacyl‑CoA reductase of long‑chain fatty acid synthesis, on breast cancer cell proliferation and migration</p> <p>Dataset (doi: 10.1007/s00018-019-03227-w) contains the original figure as TIF-format (10.1007_s00018-019-03227-w_CMLS_Fig1). Corresponding raw data obtained from a) cellomics HTC array scan analysis provided as seven files in CSV format (31003A-179400_Date_examiner_17BHSD12_8_1_1-7), b) raw data obtained from proliferation investigation on xCELLigence provided as one (31003A-179400_date_examiner_17BHSD12_9_1_1) file in CSV format. All further experiment related information and subsequent data analysis provided as two meta-data-files as TXT format (31003A-179400_date_examiner_17BHSD12_8/9_1_M_1).</p>
Data_supplemental figure 10_Impact of 17β‑HSD12, the 3‑ketoacyl‑CoA reductase of long‑chain fatty acid synthesis, on breast cancer cell proliferation and migration
<p>Data of supplemental figure 10 from Impact of 17β‑HSD12, the 3‑ketoacyl‑CoA reductase of long‑chain fatty acid synthesis, on breast cancer cell proliferation and migration</p> <p>Dataset (doi: 10.1007/s00018-019-03227-w) contains the original figure as TIF-format (10.1194_jlr.M092908_Fig. S10). Corresponding raw data obtained from a/b) western blot and densitometry provided as 20 files in CSV format (31003A-179400_date_examiner_17BHSD12_2_27-30_1-5), c) mRNA content analyzed by RT-PCR provided as 10 files in CSV format (31003A-179400_date_examiner_17BHSD12_1_11-12_1-6). d) RT-PCR provided as 10 files in CSV format (31003A-179400_date_examiner_17BHSD12_1_13_1-4). All further experiment related information protocols and subsequent data analysis provided as meta-data-files (31003A-179400_date_examiner_17BHSD12_1/2_dataset_M_1) as TXT format and (31003A-179400_date_examiner_17BHSD12_2_dataset_M_2-3) as PNG format.</p>
Data_supplemental figure 9_Impact of 17β‑HSD12, the 3‑ketoacyl‑CoA reductase of long‑chain fatty acid synthesis, on breast cancer cell proliferation and migration
<p>Data of supplemental figure 9 from Impact of 17β‑HSD12, the 3‑ketoacyl‑CoA reductase of long‑chain fatty acid synthesis, on breast cancer cell proliferation and migration</p> <p>Dataset (doi: 10.1007/s00018-019-03227-w) contains the original figure as TIF-format (10.1194_jlr.M092908_Fig. S9). Corresponding raw data obtained from a) western blot and densitometry provided as four files in CSV format (31003A-179400_date_examiner_17BHSD12_2_23_1-5); b) Cellomics HTC array scan analysis provided as eight files in CSV format (31003A-179400_Date_examiner_17BHSD12_8_19-20_1-5); c) western blot and densitometry provided as four files in CSV format (31003A-179400_date_examiner_17BHSD12_2_26_1-3), d) mRNA content analyzed by RT-PCR provided as 10 files in CSV format (31003A-179400_date_examiner_17BHSD12_1_9-10_1-6), e) western blot and densitometry provided as four files in CSV format (31003A-179400_date_examiner_17BHSD12_2_25_1-3), f) western blot and densitometry provided as four files in CSV format (31003A-179400_date_examiner_17BHSD12_2_24_1-3) All further experiment related information protocols and subsequent data analysis provided as meta-data-files (31003A-179400_date_examiner_17BHSD12_1/2/8_dataset_M_1) as TXT format and (31003A-179400_date_examiner_17BHSD12_2_dataset_M_2-3) as PNG format.</p>
Data_supplemental figure 8_Impact of 17β‑HSD12, the 3‑ketoacyl‑CoA reductase of long‑chain fatty acid synthesis, on breast cancer cell proliferation and migration
<p>Data of supplemental figure 8 from Impact of 17β‑HSD12, the 3‑ketoacyl‑CoA reductase of long‑chain fatty acid synthesis, on breast cancer cell proliferation and migration</p> <p>Dataset (doi: 10.1007/s00018-019-03227-w) contains the original figure as TIF-format (10.1194_jlr.M092908_Fig. S8). Corresponding raw data obtained from a) cellomics HTC array scan analysis provided as five files in CSV format (31003A-179400_Date_examiner_17BHSD12_8_18_1-5); b) western blot and densitometry provided as four files in CSV format (31003A-179400_date_examiner_17BHSD12_2_22_1-4), investigation of migration on xCELLigence provided as four (31003A-179400_date_examiner_17BHSD12_9_5_1) files in CSV format. All further experiment related information protocols and subsequent data analysis provided as meta-data-files (31003A-179400_date_examiner_17BHSD12_2/8/9_dataset_M_1) as TXT format and (31003A-179400_date_examiner_17BHSD12_2_dataset_M_2-3) as PNG format.</p>
Data_supplemental figure 5_Impact of 17β‑HSD12, the 3‑ketoacyl‑CoA reductase of long‑chain fatty acid synthesis, on breast cancer cell proliferation and migration
<p>Data of supplemental figure 5 from Impact of 17β‑HSD12, the 3‑ketoacyl‑CoA reductase of long‑chain fatty acid synthesis, on breast cancer cell proliferation and migration</p> <p>Dataset (doi: 10.1007/s00018-019-03227-w) contains the original figure as TIF-format (10.1194_jlr.M092908_Fig. S5). Corresponding raw data from immunofluorescence measurements provided as three files (31003A-179400_20190528_MT, PST, ADU_17BHSD12_13_2_1-3) in png format. All further experiment related information protocols and subsequent data analysis provided as meta-data-file (31003A-179400_date_examiner_17BHSD12_13_2_M_1) as TXT format.</p>
Data_supplemental figure 1_Impact of 17β‑HSD12, the 3‑ketoacyl‑CoA reductase of long‑chain fatty acid synthesis, on breast cancer cell proliferation and migration
<p>Data of supplemental figure 1 from Impact of 17β‑HSD12, the 3‑ketoacyl‑CoA reductase of long‑chain fatty acid synthesis, on breast cancer cell proliferation and migration</p> <p>Dataset (doi: 10.1007/s00018-019-03227-w) contains the original figure as TIF-format (10.1194_jlr.M092908_Fig. S1). (31003A-179400_date_examiner_17BHSD12_2_14-17) PNG format. All further experiment related information protocols as meta-data-files (31003A-179400_date_examiner_17BHSD12_2_dataset_M_1) as TXT format.</p>
Data_Figure 8_Impact of 17β‑HSD12, the 3‑ketoacyl‑CoA reductase of long‑chain fatty acid synthesis, on breast cancer cell proliferation and migration
<p>Data of figure 8 from Impact of 17β‑HSD12, the 3‑ketoacyl‑CoA reductase of long‑chain fatty acid synthesis, on breast cancer cell proliferation and migration</p> <p>Dataset (doi: 10.1007/s00018-019-03227-w) contains the original figure as TIF-format (10.1194_jlr.M092908_Fig. 8). Corresponding raw data obtained from a1/2) Western blot and densitometry provided as three files in CSV format (31003A-179400_date_examiner_17BHSD12_2_9_1-3), mRNA content analyzed by RT-PCR provided as four files in CSV format (31003A-179400_date_examiner_17BHSD12_1_7_1-4); b) Western blot and densitometry provided as three files in CSV format (31003A-179400_date_examiner_17BHSD12_2_10-11_1-3); c1/2) mRNA content analyzed by RT-PCR provided as four files in CSV format (31003A-179400_date_examiner_17BHSD12_1_8_1-4), d1/2) Western blot and densitometry provided as seven files in CSV format (31003A-179400_date_examiner_17BHSD12_2_12-13_1-4). All further experiment related information protocols and subsequent data analysis provided as meta-data-files (31003A-179400_date_examiner_17BHSD12_2/1_dataset_M_1) as TXT format and (31003A-179400_date_examiner_17BHSD12_2_dataset_M_2-3) as PNG format.</p>
Data_supplemental figure 4_Impact of 17β‑HSD12, the 3‑ketoacyl‑CoA reductase of long‑chain fatty acid synthesis, on breast cancer cell proliferation and migration
<p>Data of supplemental figure 4 from Impact of 17β‑HSD12, the 3‑ketoacyl‑CoA reductase of long‑chain fatty acid synthesis, on breast cancer cell proliferation and migration</p> <p>Dataset (doi: 10.1007/s00018-019-03227-w) contains the original figure as TIF-format (10.1194_jlr.M092908_Fig. S4). Corresponding raw data from immunofluorescence measurements provided as three files (31003A-179400_date_examiner_17BHSD12_13_1_1-3) in png format. All further experiment related information protocols and subsequent data analysis provided as meta-data-file (31003A-179400_date_examiner_17BHSD12_13_1_M_1) as TXT format.</p>
Data from: Regulation of fatty acid composition related to ontogenetic changes and niche differentiation of a common aquatic consumer
<p>Fatty acids (FAs) are key nutrients for fitness which take part in multiple physiological processes over the ontogeny of organisms. Yet, we lack evidence on how FA nutrition mediates life-history trade-offs and ontogenetic niche shifts in natural populations. In a field study, we analyzed ontogenetic changes in the FAs of Eurasian perch (<i>Perca fluviatilis</i> L.), a widespread fish that goes through ontogenetic niche shifts and can have high individual niche specialization. Diet explained most of the variation in the FA composition of perch dorsal muscle over early ontogeny (28%), while the total length explained 23%, suggesting that perch significantly regulated FA composition over early ontogeny. Condition explained 1% of the remaining variation. 18:3n-3 (ALA) and 18:4n-3 (SDA) indicated planktivory; 18:1n-7, benthivory; and 22:6n-3 (DHA), piscivory in perch diet. Conversely, perch regulated long-chained polyunsaturated fatty acids (PUFAs), such as 20:5n-3 (EPA), 20:4n-6 (ARA) and 22:6n-3 (DHA) over ontogeny, emphasizing the role of such FAs in early growth and sexual maturation. Adult perch increasingly retained 16:1n-7 and 18:1n-9 suggesting higher energy storage in older perch. Furthermore, differences in DHA availability in diet correlated with intra-cohort differences in perch growth, potentially hindering the overall use of benthic resources and promoting earlier shifts to piscivory in littoral habitats. Overall, this study indicates that in addition to diet, internal regulation may be more important for FA composition than previously thought. Differences between FA needs and FA availability may lead to life-history trade-offs that affect the ecology of consumers, including their niche.</p>
Solution properties of anionic sesame fatty acid methyl esters sulphonate surfactant and sodium dodecyl sulphate standard
<p>The relationship between specific conductivity and surfactant concentration was used to determine the solution properties of synthesized anionic sesame fatty acid methyl esters sulphonate (SEFAMESO) surfactant and sodium dodecyl sulphate (SDS) standard. The solution properties evaluated included critical micelle concentration (CMC) values, counter-ion binding degree (<em>β</em>) and thermodynamic parameters namely, Gibbs free energy (<i>∆G°</i><sub><sub><i><sub>mic</sub></i></sub></sub>), enthalpy (<i>∆H°</i><sub><sub><i><sub>mic</sub></i></sub></sub>) and entropy (<i>∆S°</i><sub><sub><i><sub>mic</sub></i></sub></sub><i>)</i> of micellization.</p>
Data from: Fatty acid profiles of five farmed Brazilian freshwater fish species from different families
The proximate composition and fatty acid (FA) profiles of five Brazilian freshwater fish species, namely Brycon cephalus (BC), Cichla ocellaris (CO), Prochilodus lineatus (PL), Leporinus friderici (LF) and Pseudoplatystoma corruscans (PCO), were investigated. CO and LF exhibited the highest (p < 0.05) moisture content, as well as one of the lowest (p < 0.05) lipid values, whereas BC presented the lowest (p < 0.05) moisture and, alongside PL, the highest (p < 0.05) lipid content. The predominant FAs in the evaluated fish species were palmitic, oleic, linoleic and docosahexaenoic acids. BC and CO presented high n-3 PUFA content, especially DHA, and demonstrated superior nutritional quality indices compared to the other evaluated fish species. Furthermore, a significant relationship was observed among these species, suggesting they possess similar nutritional lipid values. Thus, BC and CO were proven to be an excellent matrix with relevant lipid quality, desirable for human health.
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