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646 results for “Migration data”
Data from: Indirect genetic control of migration in a salmonid fish
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Data from: Neutrophils actively swell to potentiate rapid migration
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Data_Figure 3_Impact of 17β‑HSD12, the 3‑ketoacyl‑CoA reductase of long‑chain fatty acid synthesis, on breast cancer cell proliferation and migration
<p>Data of figure 3 from Impact of 17β‑HSD12, the 3‑ketoacyl‑CoA reductase of long‑chain fatty acid synthesis, on breast cancer cell proliferation and migration</p> <p>Dataset (doi: 10.1007/s00018-019-03227-w) contains the original figure as TIF-format (10.1194_jlr.M092908_Fig. 3). Corresponding raw data obtained from a) cellomics HTC array scan analysis provided as three files in CSV format (31003A-179400_Date_examiner_17BHSD12_8_2_1-3). Migration potential as three files in CSV format (31003A-179400_ date_examiner_17BHSD12_16_2_1-3); b) mRNA content analyzed by RT-PCR provided as six files in CSV format (31003A-179400_date_examiner_17BHSD12_1_3_1-6); c) western blot and densitometry provided as eight files in CSV format 31003A-179400_date_examiner_2_1-2_1-8). All further experiment related information protocols and subsequent data analysis provided as meta-data-files (31003A-179400_date_examiner_17BHSD12_8/16/1_dataset_M_1) as TXT format and (31003A-179400_date_examiner_17BHSD12_2_dataset_M_2-3) as PNG format.</p>
Data_supplemental figure 7_Impact of 17β‑HSD12, the 3‑ketoacyl‑CoA reductase of long‑chain fatty acid synthesis, on breast cancer cell proliferation and migration
<p>Data of supplemental figure 7 from Impact of 17β‑HSD12, the 3‑ketoacyl‑CoA reductase of long‑chain fatty acid synthesis, on breast cancer cell proliferation and migration</p> <p>Dataset (doi: 10.1007/s00018-019-03227-w) contains the original figure as TIF-format (10.1194_jlr.M092908_Fig. S7). Corresponding raw data obtained from xCELLigence provided as six files in CSV format (31003A-179400_date_examiner_17BHSD12_9_3-4_1-3). All further experiment related information and subsequent data analysis provided as a) two meta-data-file 31003A-179400_ date_examiner_17BHSD12_9_3-4_M_1) as TXT format.</p>
Data_supplemental figure 3_Impact of 17β‑HSD12, the 3‑ketoacyl‑CoA reductase of long‑chain fatty acid synthesis, on breast cancer cell proliferation and migration
<p>Data of supplemental figure 3 from Impact of 17β‑HSD12, the 3‑ketoacyl‑CoA reductase of long‑chain fatty acid synthesis, on breast cancer cell proliferation and migration</p> <p>Dataset (doi: 10.1007/s00018-019-03227-w) contains the original figure as TIF-format (10.1194_jlr.M092908_Fig. S3). Corresponding raw data from a) western blot and densitometry provided as 11 files in CSV format (31003A-179400_date_examiner_17BHSD12_2_18-20_1-3) b) cellomics HTC array scan analysis provided as four files in CSV format (31003A-179400_Date_examiner_17BHSD12_8_13_1-4). All further experiment related information protocols and subsequent data analysis provided as meta-data-files (31003A-179400_date_examiner_17BHSD12_2/8_dataset_M_1) as TXT format and (31003A-179400_date_examiner_17BHSD12_2_dataset_M_2-3) as PNG format.</p>
Data_supplemental figure 6_Impact of 17β‑HSD12, the 3‑ketoacyl‑CoA reductase of long‑chain fatty acid synthesis, on breast cancer cell proliferation and migration
<p>Data of supplemental figure 6 from Impact of 17β‑HSD12, the 3‑ketoacyl‑CoA reductase of long‑chain fatty acid synthesis, on breast cancer cell proliferation and migration</p> <p>Dataset (doi: 10.1007/s00018-019-03227-w) contains the original figure as TIF-format (10.1194_jlr.M092908_Fig. S6). Corresponding raw data from a-c) cellomics HTC array scan analysis provided as 19 files in CSV format (31003A-179400_Date_examiner_17BHSD12_8_14-17_1-5), d) western blot and densitometry provided as five files in CSV format (31003A-179400_date_examiner_17BHSD12_2_21_1-5). All further experiment related information protocols and subsequent data analysis provided as meta-data-files (31003A-179400_date_examiner_17BHSD12_2/8_dataset_M_1) as TXT format and (31003A-179400_date_examiner_17BHSD12_2_dataset_M_2-3) as PNG format.</p>
Data_Figure 1_Impact of 17β‑HSD12, the 3‑ketoacyl‑CoA reductase of long‑chain fatty acid synthesis, on breast cancer cell proliferation and migration
<p>Data of figure 1 from Impact of 17β‑HSD12, the 3‑ketoacyl‑CoA reductase of long‑chain fatty acid synthesis, on breast cancer cell proliferation and migration</p> <p>Dataset (doi: 10.1007/s00018-019-03227-w) contains the original figure as TIF-format (10.1007_s00018-019-03227-w_CMLS_Fig1). Corresponding raw data obtained from a) cellomics HTC array scan analysis provided as seven files in CSV format (31003A-179400_Date_examiner_17BHSD12_8_1_1-7), b) raw data obtained from proliferation investigation on xCELLigence provided as one (31003A-179400_date_examiner_17BHSD12_9_1_1) file in CSV format. All further experiment related information and subsequent data analysis provided as two meta-data-files as TXT format (31003A-179400_date_examiner_17BHSD12_8/9_1_M_1).</p>
Data_supplemental figure 10_Impact of 17β‑HSD12, the 3‑ketoacyl‑CoA reductase of long‑chain fatty acid synthesis, on breast cancer cell proliferation and migration
<p>Data of supplemental figure 10 from Impact of 17β‑HSD12, the 3‑ketoacyl‑CoA reductase of long‑chain fatty acid synthesis, on breast cancer cell proliferation and migration</p> <p>Dataset (doi: 10.1007/s00018-019-03227-w) contains the original figure as TIF-format (10.1194_jlr.M092908_Fig. S10). Corresponding raw data obtained from a/b) western blot and densitometry provided as 20 files in CSV format (31003A-179400_date_examiner_17BHSD12_2_27-30_1-5), c) mRNA content analyzed by RT-PCR provided as 10 files in CSV format (31003A-179400_date_examiner_17BHSD12_1_11-12_1-6). d) RT-PCR provided as 10 files in CSV format (31003A-179400_date_examiner_17BHSD12_1_13_1-4). All further experiment related information protocols and subsequent data analysis provided as meta-data-files (31003A-179400_date_examiner_17BHSD12_1/2_dataset_M_1) as TXT format and (31003A-179400_date_examiner_17BHSD12_2_dataset_M_2-3) as PNG format.</p>
Data_supplemental figure 9_Impact of 17β‑HSD12, the 3‑ketoacyl‑CoA reductase of long‑chain fatty acid synthesis, on breast cancer cell proliferation and migration
<p>Data of supplemental figure 9 from Impact of 17β‑HSD12, the 3‑ketoacyl‑CoA reductase of long‑chain fatty acid synthesis, on breast cancer cell proliferation and migration</p> <p>Dataset (doi: 10.1007/s00018-019-03227-w) contains the original figure as TIF-format (10.1194_jlr.M092908_Fig. S9). Corresponding raw data obtained from a) western blot and densitometry provided as four files in CSV format (31003A-179400_date_examiner_17BHSD12_2_23_1-5); b) Cellomics HTC array scan analysis provided as eight files in CSV format (31003A-179400_Date_examiner_17BHSD12_8_19-20_1-5); c) western blot and densitometry provided as four files in CSV format (31003A-179400_date_examiner_17BHSD12_2_26_1-3), d) mRNA content analyzed by RT-PCR provided as 10 files in CSV format (31003A-179400_date_examiner_17BHSD12_1_9-10_1-6), e) western blot and densitometry provided as four files in CSV format (31003A-179400_date_examiner_17BHSD12_2_25_1-3), f) western blot and densitometry provided as four files in CSV format (31003A-179400_date_examiner_17BHSD12_2_24_1-3) All further experiment related information protocols and subsequent data analysis provided as meta-data-files (31003A-179400_date_examiner_17BHSD12_1/2/8_dataset_M_1) as TXT format and (31003A-179400_date_examiner_17BHSD12_2_dataset_M_2-3) as PNG format.</p>
Data_supplemental figure 8_Impact of 17β‑HSD12, the 3‑ketoacyl‑CoA reductase of long‑chain fatty acid synthesis, on breast cancer cell proliferation and migration
<p>Data of supplemental figure 8 from Impact of 17β‑HSD12, the 3‑ketoacyl‑CoA reductase of long‑chain fatty acid synthesis, on breast cancer cell proliferation and migration</p> <p>Dataset (doi: 10.1007/s00018-019-03227-w) contains the original figure as TIF-format (10.1194_jlr.M092908_Fig. S8). Corresponding raw data obtained from a) cellomics HTC array scan analysis provided as five files in CSV format (31003A-179400_Date_examiner_17BHSD12_8_18_1-5); b) western blot and densitometry provided as four files in CSV format (31003A-179400_date_examiner_17BHSD12_2_22_1-4), investigation of migration on xCELLigence provided as four (31003A-179400_date_examiner_17BHSD12_9_5_1) files in CSV format. All further experiment related information protocols and subsequent data analysis provided as meta-data-files (31003A-179400_date_examiner_17BHSD12_2/8/9_dataset_M_1) as TXT format and (31003A-179400_date_examiner_17BHSD12_2_dataset_M_2-3) as PNG format.</p>
Data_supplemental figure 5_Impact of 17β‑HSD12, the 3‑ketoacyl‑CoA reductase of long‑chain fatty acid synthesis, on breast cancer cell proliferation and migration
<p>Data of supplemental figure 5 from Impact of 17β‑HSD12, the 3‑ketoacyl‑CoA reductase of long‑chain fatty acid synthesis, on breast cancer cell proliferation and migration</p> <p>Dataset (doi: 10.1007/s00018-019-03227-w) contains the original figure as TIF-format (10.1194_jlr.M092908_Fig. S5). Corresponding raw data from immunofluorescence measurements provided as three files (31003A-179400_20190528_MT, PST, ADU_17BHSD12_13_2_1-3) in png format. All further experiment related information protocols and subsequent data analysis provided as meta-data-file (31003A-179400_date_examiner_17BHSD12_13_2_M_1) as TXT format.</p>
Data_supplemental figure 1_Impact of 17β‑HSD12, the 3‑ketoacyl‑CoA reductase of long‑chain fatty acid synthesis, on breast cancer cell proliferation and migration
<p>Data of supplemental figure 1 from Impact of 17β‑HSD12, the 3‑ketoacyl‑CoA reductase of long‑chain fatty acid synthesis, on breast cancer cell proliferation and migration</p> <p>Dataset (doi: 10.1007/s00018-019-03227-w) contains the original figure as TIF-format (10.1194_jlr.M092908_Fig. S1). (31003A-179400_date_examiner_17BHSD12_2_14-17) PNG format. All further experiment related information protocols as meta-data-files (31003A-179400_date_examiner_17BHSD12_2_dataset_M_1) as TXT format.</p>
Data_Figure 8_Impact of 17β‑HSD12, the 3‑ketoacyl‑CoA reductase of long‑chain fatty acid synthesis, on breast cancer cell proliferation and migration
<p>Data of figure 8 from Impact of 17β‑HSD12, the 3‑ketoacyl‑CoA reductase of long‑chain fatty acid synthesis, on breast cancer cell proliferation and migration</p> <p>Dataset (doi: 10.1007/s00018-019-03227-w) contains the original figure as TIF-format (10.1194_jlr.M092908_Fig. 8). Corresponding raw data obtained from a1/2) Western blot and densitometry provided as three files in CSV format (31003A-179400_date_examiner_17BHSD12_2_9_1-3), mRNA content analyzed by RT-PCR provided as four files in CSV format (31003A-179400_date_examiner_17BHSD12_1_7_1-4); b) Western blot and densitometry provided as three files in CSV format (31003A-179400_date_examiner_17BHSD12_2_10-11_1-3); c1/2) mRNA content analyzed by RT-PCR provided as four files in CSV format (31003A-179400_date_examiner_17BHSD12_1_8_1-4), d1/2) Western blot and densitometry provided as seven files in CSV format (31003A-179400_date_examiner_17BHSD12_2_12-13_1-4). All further experiment related information protocols and subsequent data analysis provided as meta-data-files (31003A-179400_date_examiner_17BHSD12_2/1_dataset_M_1) as TXT format and (31003A-179400_date_examiner_17BHSD12_2_dataset_M_2-3) as PNG format.</p>
Data_supplemental figure 4_Impact of 17β‑HSD12, the 3‑ketoacyl‑CoA reductase of long‑chain fatty acid synthesis, on breast cancer cell proliferation and migration
<p>Data of supplemental figure 4 from Impact of 17β‑HSD12, the 3‑ketoacyl‑CoA reductase of long‑chain fatty acid synthesis, on breast cancer cell proliferation and migration</p> <p>Dataset (doi: 10.1007/s00018-019-03227-w) contains the original figure as TIF-format (10.1194_jlr.M092908_Fig. S4). Corresponding raw data from immunofluorescence measurements provided as three files (31003A-179400_date_examiner_17BHSD12_13_1_1-3) in png format. All further experiment related information protocols and subsequent data analysis provided as meta-data-file (31003A-179400_date_examiner_17BHSD12_13_1_M_1) as TXT format.</p>
CCoDaMiC: A framework for Coherent Coordination of Data Migration and Computation platforms
<p>This dataset release supports the results presented in the paper</p> <p><strong>Chinmaya Kumar Dehury</strong>, Satish Narayana Srirama, Tek Raj Chhetri, CCoDaMiC: A Framework for Coherent Coordination of Data Migration and Computation Platforms. Future Generation Computer Systems</p> <p>URL: https://www.sciencedirect.com/science/article/pii/S0167739X19330924</p> <p> </p>
Data from: Ovarian BDNF promotes survival, migration, and attachment of tumor precursors originated from p53 mutant fallopian tube epithelial cells
<p class="CxSpFirst"><span>High-grade serous ovarian carcinoma (HGSOC) is the most lethal gynecological malignancy. New evidence supports a hypothesis that HGSOC can originate from fallopian tube epithelium (FTE). It is unclear how genetic alterations and pathophysiological processes drive the progression of FTE tumor precursors into widespread HGSOCs. In this study, we uncovered that brain-derived neurotrophic factor (BDNF) in the follicular fluid stimulates the tropomyosin receptor kinase B (TrkB)-expressing FTE cells to promote their survival, migration, and attachment. Using <i>in vitro</i> and <i>in vivo</i> models, we further identified that the acquisition of common TP53 gain-of-function (GOF) mutations in FTE cells led to enhanced BDNF/TrkB signaling comparing to that of FTE cells with <i>TP53</i> loss-of-function (LOF) mutations. Different mutant p53 proteins can either increase TrkB transcription or enhance TrkB endocytic recycling. Our findings have demonstrated possible interplays between genetic alterations in FTE tumor precursors (i.e., p53 gain-of-function mutations) and pathophysiological processes (i.e., the release of follicular fluid upon ovulation) during the initiation of HGSOC from the fallopian tube. Our data revealed molecular events underlying the link between HGSOC tumorigenesis and ovulation, a physiological process that has been associated with risk factors of HGSOC. </span></p>
Data from: Characterizing population and individual migration patterns among native and restored bighorn sheep (Ovis canadensis)
Migration evolved as a behavior to enhance fitness through exploiting spatially and temporally variable resources and avoiding predation or other threats. Globally, landscape alterations have resulted in declines to migratory populations across taxa. Given the long time periods over which migrations evolved in native systems, it is unlikely restored populations embody the same migratory complexity that existed before population reductions or regional extirpation. 2. We used GPS location data collected from 209 female bighorn sheep (Ovis canadensis) to characterize population and individual migration patterns along elevational and geographic continuums for 18 populations of bighorn sheep with different management histories (i.e., restored, augmented, and native) across the western United States. 3. Individuals with resident behaviors were present in all management histories. Elevational migrations were the most common population-level migratory behavior. There were notable differences in the degree of individual variation within a population across the three management histories. Relative to native populations, restored and augmented populations had less variation among individuals with respect to elevational and geographic migration distances. Differences in migratory behavior were most pronounced for geographic distances, where the majority of native populations had a range of variation that was 2 to 4 times greater than restored or augmented populations. 4. Synthesis and applications. Migrations within native populations include a variety of patterns that translocation efforts have not been able to fully recreate within restored and augmented populations. Theoretical and empirical research has highlighted the benefits of migratory diversity in promoting resilience and population stability. Limited migratory diversity may serve as an additional factor limiting demographic performance and range expansion. We suggest preserving native systems with intact migratory portfolios and a more nuanced approach to restoration and augmentation in which source populations are identified based on a suite of criteria that includes matching migratory patterns of source populations with local landscape attributes.
Wrong-way migrations of benthic species driven by ocean warming and larval transport: Data
<p>All data used to generate figures, tables, and other results in the publication cited below. All data are derived from publicly available data archives as indicated in the publication's Data Availability Statement. Archived data may change over time, and results in the paper are only relevant to data as originally downloaded. These condensed data were used in the analyses and are provided to ensure that analyses are replicable.</p> <p>If using any code or data, please cite the following publication:<br> Heidi L. Fuchs, Robert J. Chant, Elias J. Hunter, Enrique N. Curchitser, Gregory P. Gerbi, and Emily A. Chen. 2020. Wrong-way migrations of benthic species driven by ocean warming and larval transport. Nature Climate Change. DOI: 10.1038/s41558-020-0894-x</p> <p>The paper is available at: <a href="https://nam02.safelinks.protection.outlook.com/?url=https%3A%2F%2Fwww.nature.com%2Farticles%2Fs41558-020-0894-x&data=02%7C01%7Chfuchs%40marine.rutgers.edu%7C8f5bf7d8e5744a5353e408d84e6e924e%7Cb92d2b234d35447093ff69aca6632ffe%7C1%7C0%7C637345584096517620&sdata=Jy%2FD%2FyrfSX6Z0EU1rriqL%2FHnz7KLxpmlqjQmhM6ACAg%3D&reserved=0">https://www.nature.com/articles/s41558-020-0894-x</a></p>
Data from: Broad-front migration leads to strong migratory connectivity in the lesser kestrel (Falco naumanni)
Aim: Migratory animals regularly move between often distant breeding and non-breeding ranges. Knowledge about how these ranges are linked by movements of individuals from different populations is crucial for unravelling temporal variability in population spatial structuring and for identifying environmental drivers of population dynamics acting at different spatio-temporal scales. We performed a large-scale individual-based migration tracking study of the lesser kestrel (Falco naumanni), an Afro-Palearctic migratory raptor, to determine the patterns of migratory connectivity of European breeding populations. Location: Europe, Africa. Methods: Migration data were recorded using different devices (geolocators, Argos PTTs, GPS loggers) from 87 individuals breeding in the three core European populations, located in the Iberian, Italian and Balkan peninsulas. We estimated connectivity by the Mantel correlation coefficient (rM), and computed both the degree of separation between the non-breeding areas of individuals from the same population (i.e., the population spread) and the relative size of the non-breeding range (i.e., the non-breeding range spread). Results: European lesser kestrels migrated on a broad-front across the Mediterranean Sea and Sahara Desert, with different populations using different routes. Iberian birds migrated to western Sahel (Senegal, Mauritania, western Mali), Balkan birds migrated chiefly to central-eastern Sahel (Niger, Nigeria, Chad), whereas Italian ones spread from eastern Mali to Nigeria. Spatial differentiation of non-breeding areas led to a strong migratory connectivity (rM = 0.58), associated with a relatively high population (637 km) and non-breeding range (1149 km) spread. Main conclusions: Our comprehensive analysis of the non-breeding distribution of European lesser kestrel populations revealed a strong migratory connectivity, a rare occurrence in long-distance avian migrants. The geographic conformation of the species' breeding and non-breeding ranges, together with broad-front migration across ecological barriers, promoted the differentiation of migratory routes and non-breeding areas. Strong connectivity could then arise because of both high population spread and broad non-breeding range.
Data from: Experimental migration upward in elevation is associated with strong selection on life history traits
One of the strongest biological impacts of climate change has been the movement of species poleward and upward in elevation. Yet, what is not clear, is the extent to which the spatial distribution of locally adapted lineages and ecologically important traits may also shift with continued climate change. Here, we take advantage of a transplant experiment mimicking up-slope seed dispersal for a suite of ecologically-diverse populations of yellow monkeyflower (Mimulus guttatus sensu lato) into a high-elevation common garden during an extreme drought period in the Sierra Nevada mountains, California, USA. We use a demographic approach to quantify fitness and test for selection on life history traits in local vs. lower-elevation populations and in normal vs. drought years to test the potential for up-slope migration and phenotypic selection to alter the distribution of key life history traits in montane environments. We find that lower-elevation populations tend to outperform local populations, confirming the potential for up-slope migration. Although selection generally favored some local montane traits, including larger flowers and larger stem size at flowering, drought conditions tended to select for earlier flowering typical of lower elevation genotypes. Taken together, this suggests that monkeyflower lineages moving upward in elevation could experience selection for novel trait combinations, particularly under warmer and drier conditions that are predicted to occur with continued climate change.
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Allen Brain Atlas
Allen Brain Atlas is an Allen Institute collection of brain map atlases, datasets, APIs, and analysis tools covering mouse, human, and non-human primate brain resources.
Annotated Behaviour and Observability Dataset (ABODe)
ABODe is a University of Edinburgh DataShare dataset for behavior classification in group-housed mice using home-cage video, identities, bounding boxes, ground-plate positions, and annotator labels.
DANDI Archive for NWB datasets
DANDI is a BRAIN Initiative archive for publishing and sharing neurophysiology data, including electrophysiology, optophysiology, and behavioral data packaged as NWB and related standards.
International Brain Laboratory public data
The International Brain Laboratory public data releases expose standardized mouse decision-making experiments, including Neuropixels recordings, widefield calcium imaging, behavior, and session metadata accessed through the ONE API.
OpenNeuro
OpenNeuro is a free, open platform for sharing neuroimaging datasets, with public search, dataset pages, and download paths for web, S3, DataLad, and the OpenNeuro CLI.