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730 results for “biochemicals”
Fig. 3 in Transcriptomic investigation of the biochemical function of 7-dehydro- cholesterol reductase 1 from the traditional Chinese medicinal plant Anemarrhena asphodeloides Bunge
Fig. 3. Content analyses of steroidal saponins (A) and phytosterols in different organs of A. asphodeloides Bunge. Corresponding histograms indicate the difference in concentration among the different organs. Three biological replicates were performed for each sample.
Fig. 7 in Transcriptomic investigation of the biochemical function of 7-dehydro- cholesterol reductase 1 from the traditional Chinese medicinal plant Anemarrhena asphodeloides Bunge
Fig. 7. Phylogenetic tree of 7-dehydrocholesterol reductase. Sequences from the following species were represented: A. asphodeloides Bunge (Aa), Arabidopsis thaliana L. (Brassicaceae) (At), Brachypodium distachyon L. (Poaceae) (Bd), Capsicum annuum L. (Solanaceae) (Ca), Chlamydomonas reinhardtii (Cr), Homo sapiens (Hs), Medicago truncatula Gaetn (Leguminosae) (Mt), Nicotiana. Benthamiana Domin (Solanaceae) (Nb), Ostreococcus lucimarinus (Ol), Oryza sativa L. (Poaceae) (Os), Ostreococcus tauri (Ot), Physcomitrella patens (Pp), Sorghum bicolor L. (Poaceae) (Sb), Saccharomyces cerevisiae (Sc), Solanum lycopersicum L. (Solanaceae) (Sl), Solanum melongena L. (Solanaceae) (Sm), Solanum tuberosum L. (Solanaceae) (St), Volvax carteri (Vc), Vitis vinifera L. (Vitaceae) (Vv) and Zea mays L. (Poaceae) (Zm).
Fig. 4 in Biochemical and molecular insights of PGPR application for the augmentation of carotenoids, tocopherols, and folate in the foliage of Moringa oleifera
Fig. 4. HPLC Chromatograms of 1–5,6,7,8 tetrahydro folic acid (THF) in Control and 937b- B. subtilis IN937b treated M. oleifera foliage.
Fig. 3 in Biochemical and molecular insights of PGPR application for the augmentation of carotenoids, tocopherols, and folate in the foliage of Moringa oleifera
Fig. 3. HPLC Chromatograms of 1- Violaxanthin, 2- Lutein, 3- Chl b, 4-Chl a, 5- β-carotene in control and GBO3- B. subtilis GB03 treated M. oleifera foliage.
Fig. 5 in Biochemical and molecular insights of PGPR application for the augmentation of carotenoids, tocopherols, and folate in the foliage of Moringa oleifera
Fig. 5. Differential expression of mRNA levels of γ-tocopherol methyltransferase (γ-TMT), phytoene synthase (PSY), phytoene desaturase (PDS), lycopene β-cyclase (LBC) and dihydrofolate reductase thymidylate (DHFR-TS) in M. oleifera foliage after treatment with GB03-B. subtilis GB03, T4-B. pumilus T4, COM1- Combination 1, COM2- Combination 2, COM3-Combination 3. Experiments were repeated three times, each with three replicates. Columns without a common lowercase letter indicate significantly different values among treatments based on Tukey's test (P value ≤ 0.05).
Fig. 2 in Biochemical and molecular insights of PGPR application for the augmentation of carotenoids, tocopherols, and folate in the foliage of Moringa oleifera
Fig. 2. HPLC Chromatograms of 1- δ-tocopherol, 2- γ-tocopherol and 3- α-tocopherol in control, 937a- B. amyloliquefaciens IN937a and SE34- B. pumilus SE34 treated M. oleifera foliage.
Fig. 1. M. oleifera treated with 1 in Biochemical and molecular insights of PGPR application for the augmentation of carotenoids, tocopherols, and folate in the foliage of Moringa oleifera
Fig. 1. M. oleifera treated with 1) B. amyloliquefaciens IN937a, 2) B. subtilis IN937b, 3) Brevibacillus brevis IPC 11, 4) B. subtilis GB03, 5) B. pumilus INR7, 6) B. pumilus SE34, 7) B. pumilus T4, 8) P. fluorescens UOM14 shown compared to control.
Fig. 5 in L-DOPA synthesis in Mucuna pruriens (L.) DC. is regulated by polyphenol oxidase and not CYP 450/tyrosine hydroxylase: An analysis of metabolic pathway using biochemical and molecular markers
Fig. 5. The amplicons generated using degenerate primer approach. (a) Lane M-DNA ladder, Lane 1–250 bp amplicon generated using MTH –F and MTH-R primer pairs of TH gene (b) Lane M-DNA ladder, Lane 1 and 2–800 bp amplicon using primers deduced from the peptide sequence derived through LCMS/MS.
Fig. 4 in L-DOPA synthesis in Mucuna pruriens (L.) DC. is regulated by polyphenol oxidase and not CYP 450/tyrosine hydroxylase: An analysis of metabolic pathway using biochemical and molecular markers
Fig. 4. Effect of enzyme inhibitors on L-DOPA production in callus cultures of M. pruriens was estimated using HPTLC. The culture without inhibitor was treated as negative control and cultures with different concentration of the inhibitor were the test samples. (Control-untreated, C = Cimetidine at 1.98 μM and 19.8 μM; Q = Quinidine at 1.46 μM and 14.6 μM; A = L-ascorbic acid at 567 μM and 851 μM; K = Kojic acid at 703 μM and 1055 μM).
Fig. 3 in L-DOPA synthesis in Mucuna pruriens (L.) DC. is regulated by polyphenol oxidase and not CYP 450/tyrosine hydroxylase: An analysis of metabolic pathway using biochemical and molecular markers
Fig. 3. Effect of substrate concentration on partially purified enzymes. The assay was performed for PPO activity with 50 mM catechol as substrate at pH 6.0 while keeping the temperature for reaction at 30 ◦ C. For TH activity, 30 mM L-tyrosine was the substrate and assay done at pH 7.0 and 25 ◦ C.
Fig. 2 in L-DOPA synthesis in Mucuna pruriens (L.) DC. is regulated by polyphenol oxidase and not CYP 450/tyrosine hydroxylase: An analysis of metabolic pathway using biochemical and molecular markers
Fig. 2. Effect of pH on the activity of partially purified enzymes from Mucuna pruriens. The assay was performed using 50 mM catechol and 30 mM L-tyrosine as substrates for the PPO and TH enzyme activity, respectively. Four different buffers with their optimal buffering capacity in the pH range of 3–10 were used in separate assays.
Fig. 7 in L-DOPA synthesis in Mucuna pruriens (L.) DC. is regulated by polyphenol oxidase and not CYP 450/tyrosine hydroxylase: An analysis of metabolic pathway using biochemical and molecular markers
Fig. 7. Homology modelling and secondary structure prediction of PPO enzyme from Mucuna pruriens (a) Predicted secondary structure of PPO (b) Phyre2 protein model for PPO with 3D model dimensions (in Å) (X:49.941 Y:64.463 Z:57.979). Image colored by rainbow N → C terminus (c) Three dimensional SWISS protein model for PPO enzyme with two active copper binding ligands (copper ions bridging oxygen moiety is illustrated as small yellow spheres highlighted in the box), conserved histidine residues and metal complex interactions (in dotted lines). Chain A for Ligand 1: H.183, H.204, H.213, F.367, H.371; metal interactions: A:H.183, A:H.204, A:H.213. Chain A for Ligand 2: H.337, H.341, F.367, H.370, H.371; metal interactions: A:H.337, A:H.341, A:H.371). (For interpretation of the references to color in this figure legend, the reader is referred to the Web version of this article.)
Fig. 6. The 1800 in L-DOPA synthesis in Mucuna pruriens (L.) DC. is regulated by polyphenol oxidase and not CYP 450/tyrosine hydroxylase: An analysis of metabolic pathway using biochemical and molecular markers
Fig. 6. The 1800 bp amplicon of full-length PPO cDNA obtained after deducing the 5′and 3′ ends through RACE analysis. Lane 1- 1 Kb DNA marker, Lane 2 and 3 the amplicon in duplicate after amplification using gene specific primers.
Role of Some Biochemical Indices for Prediction of Acute Kidney Injury in Intensive Care Unit Patients in Upper Egypt
ClinicalTrials.gov study NCT06791200. IPD Sharing: NO. Countries: 1. Publications: 1.
Clinical, Morphometric and Biochemical Effects on Adiposopathy Associated With the Use of GLP-1RA in CKD
ClinicalTrials.gov study NCT07309094. IPD Sharing: NO. Countries: 1. Publications: 15.
The Dose Response of Prednisone on Biochemical and Clinical Makers in Adult Healthy Volunteers
ClinicalTrials.gov study NCT02767089. IPD Sharing: Not stated. Countries: 1. Publications: 1.
Effects of Kefir Consumption on Health Outcomes: Gastrointestinal System, Immunity, Biochemical Parameters, Body Composition, Sleep Quality and Mental Well Being in Healthy Adults
ClinicalTrials.gov study NCT06612164. IPD Sharing: UNDECIDED. Countries: 1. Publications: 1.
Could Non-invasive Biochemical, Image or Physiological Index Predict Significant Coronary Arterial Stenosis in Symptomatic Adults?
ClinicalTrials.gov study NCT01645228. IPD Sharing: Not stated. Countries: 1. Publications: 6.
Effect of CoQ10 Plus Selenium Supplementation on Clinical Outcomes and Biochemical Markers in ME/CFS (CoSeME Study)
ClinicalTrials.gov study NCT05128292. IPD Sharing: NO. Countries: 1. Publications: 12.
Effect of Citrulline on the Clinical and Biochemical Evolution of Patients With Sepsis.
ClinicalTrials.gov study NCT02370030. IPD Sharing: Not stated. Countries: 1. Publications: 9.
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Allen Brain Atlas
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DANDI Archive for NWB datasets
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International Brain Laboratory public data
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OpenNeuro
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