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Dataset results
76 results for “copeptin”
Fetal Copeptin After Oxytocin Challenge Test
ClinicalTrials.gov study NCT01962701. IPD Sharing: UNDECIDED. Countries: 1. Publications: 1.
Diagnostic Utility of Copeptin in Addition to High-sensitivity Cardiac Troponin for the Early Diagnosis of NSTE-ACS
ClinicalTrials.gov study NCT01962506. IPD Sharing: Not stated. Countries: 1. Publications: 1.
Copeptin in the Differential Diagnosis of Dysnatremia in Hospitalized Patients
ClinicalTrials.gov study NCT01456533. IPD Sharing: Not stated. Countries: 1. Publications: 2.
Prognostic Value of Copeptin for Infarct Size and Prognosis in Patients With ST-elevation Myocardial Infarction
ClinicalTrials.gov study NCT03074214. IPD Sharing: NO. Countries: 1. Publications: 2.
Copeptin in Differentiation of Polyuria and Polydipsia
ClinicalTrials.gov study NCT01056887. IPD Sharing: Not stated. Countries: 1. Publications: 1.
Place of Copeptin-troponin Assay in the Elimination Diagnosis of Non-ST+ ACS
ClinicalTrials.gov study NCT05902117. IPD Sharing: Not stated. Countries: 1. Publications: 10.
Copeptin as a Stress Marker During a Written Examination
ClinicalTrials.gov study NCT02051647. IPD Sharing: Not stated. Countries: 1. Publications: 1.
Value of Von Willebrand Factor and Copeptin Assay in Children With Febrile Seizures at Sohag University Hospital
ClinicalTrials.gov study NCT05008458. IPD Sharing: YES. Countries: 1. Publications: 3.
Copeptin Registry (proCORE) Biomarkers in Cardiology (BIC)-19
ClinicalTrials.gov study NCT02490969. IPD Sharing: UNDECIDED. Countries: 1. Publications: 1.
Prediction of Inflammatory Response and Hypotensive Syndrome After Cardiac Surgery by Preoperative Copeptin Level
ClinicalTrials.gov study NCT03288610. IPD Sharing: NO. Countries: 1. Publications: 2.
Assessment of Circulating Copeptin Levels in Healthy Subjects and Patients With Renal Insufficiency After Oral Water Load and Hypertonic Saline Infusion
ClinicalTrials.gov study NCT00696605. IPD Sharing: Not stated. Countries: 1. Publications: 1.
Investigation of the Biomarker Copeptin in Patients With Acute Myocardial Infarction
ClinicalTrials.gov study NCT00952744. IPD Sharing: Not stated. Countries: 1. Publications: 2.
Copeptin for Risk Stratification in Non-traumatic Headache in the Emergency Setting - The CoHead Study
ClinicalTrials.gov study NCT01174901. IPD Sharing: Not stated. Countries: 1. Publications: 6.
Use of Copeptin in Diabetes Insipidus
ClinicalTrials.gov study NCT01940614. IPD Sharing: Not stated. Countries: 1. Publications: 3.
Data from: Is high-sensitivity troponin, alone or in combination with copeptin, sensitive enough for ruling out NSTEMI in very early presenters at admission? A post-hoc analysis performed in emergency departments
Objectives: Copeptin and high-sensitivity cardiac troponin (HS-cTn) assays improve the early detection of NSTEMI. Their sensitivities may however be reduced in very early presenters. Setting: We performed a post-hoc analysis of three prospective studies that included patients who presented to the Emergency Department for chest pain onset (CPO) of less than 6 hours. Participants: 449 patients were included, in whom 12% had NSTEMI. CPO occurred <2h from ED presentation in 160, between 2-4h in 143, and >4h in 146 patients. The prevalence of NSTEMI was similar in all groups (9%, 13% and 12%, respectively, p=0.281). Measures: Diagnostic performances of HS-cTn and copeptin at presentation were examined according to CPO. The discharge diagnosis was adjudicated by 2 experts, including cTnI. HS-cTn and copeptin were blindly measured. Results: Diagnostic accuracies of cTnI, cTnI+copeptin and HS-cTnT (but not HS-cTnT+copeptin) lower through CPO categories. For patients with CPO<2h, the choice of a threshold value of 14 ng/L for HS-cTnT resulted in 3 false negative (Sensitivity 80% [95%CI: 51-95]; specificity 85% [78-90]; 79% of correctly ruled-out patients) and that of 5 ng/L in 2 false negative ( Sensitivity 87% [59-98]; specificity 58% [50-66]; 52% of correctly ruled-out patients). The addition of copeptin to HS-cTnT induced a decrease of misclassified patients to 1 in patients with CPO<2h (Sensitivity 93% [66-100]; specificity 41% [33-50]). Conclusion: A single measurement of HS-cTn, alone or in combination with copeptin at admission, seems not safe enough for ruling out NSTEMI in very early presenters (with CPO<2h). Trial registrations: French Health Ministry (no. DC-2009-1052), French Local Ethic comity « Comité de Protection des Personnes Ile-de-France » III (Hôpital Cochin) et VI (CHU Pitié-Salpétrière).
Data from: Serum copeptin and neuron specific enolase are markers of neonatal distress and long-term neurodevelopmental outcome
The objective of this study was to evaluate the early changes in serial serum levels of copeptin and neuron-specific enolase (NSE) in neonates diagnosed with birth asphyxia, and to determine whether these biomarkers measured in the first 168 hours after birth are predictive of long-term neurodevelopmental outcome. Copeptin and NSE levels were measured from serum samples collected 6, 12, 24, 48, 72, and 168 hours after birth from 75 term neonates diagnosed with hypoxic-ischemic encephalopathy (HIE) and treated with therapeutic hypothermia for 72 hours. In addition, serum copeptin levels after birth were measured from 10 HIE diagnosed neonates, who were randomized to the normothermic arm of the TOBY cohort. All neonates underwent neurodevelopmental assessment using the Bayley Scales of Infant and Toddler Development-II at two years of age. Copeptin levels were highest at 6 hours after birth and steadily decreased, whereas the highest NSE levels were measured at 24 hours after birth. The biomarker levels correlated with blood-gas parameters (base excess, pH and lactate) at 6 and 12 hours after birth. Copeptin and NSE levels in the early postnatal period were significantly higher in neonates with poor outcome compared to those with favorable outcome at two years of age. Furthermore, in the TOBY cohort, copeptin levels were significantly lower in hypothermic compared to normothermic neonates. To conclude, copeptin and NSE measured in the early postnatal period are potential prognostic biomarkers of long-term neurodevelopmental outcome in term neonates diagnosed with HIE and treated with therapeutic hypothermia.
Supplemetary material- Apelin and Copeptin Levels in Patients With Chronic SIAD Treated With Empagliflozin
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Copeptin Changes After Food Intake
ClinicalTrials.gov study NCT01772914. IPD Sharing: Not stated. Countries: 1. Publications: 1.
Pilot Study of Using Copeptin to Predict Response to Tolvaptan
ClinicalTrials.gov study NCT01346072. IPD Sharing: Not stated. Countries: 1. Publications: 0.
Data from: Serum copeptin and neuron specific enolase are markers of neonatal distress and long-term neurodevelopmental outcome
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