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Fig. 2 in Comparative analysis of metacyclogenesis and infection curves in different discrete typing units of Trypanosoma cruzi

Fig. 2 Mosaic of microphotographs showing T. cruzi CDT infections in Vero cells for 120 h, using different DTUs. Samples were fixed with formaldehyde and stained with Field's stain. This representation captures the different stages and dynamics of infection in the different DTUs, and the cell morphological changes induced. White arrows indicate the presence of amastigotes at 24 h. By 72 h, amastigote nests become visible, and at 96–120 h, nests along with cell-derived trypomastigotes are observed

Fig. 4 in Molecular screening of tsetse flies and cattle reveal different Trypanosoma species including T. grayi and T. theileri in northern Cameroon

Fig. 4 Dcmtlcbutcon of Trypanosoma mpeccem cn tmetme flcem. a Relatcve abundance of tlspanomomal DNA bs mpeccem cn the gut. b Relatcve abundance of tlspanomomal DNA bs mpeccem cn plobomccm. c Collelatcon of tlspanomomal DNA cn gut and plobomccm. Abbreviations: Tg, T. grayi; Tc, T. congolense; Tb, T. brucei mmp.; Tv, T. vivax. If no amplccon wam detected, the fls wam conmcdeled to be negatcve

Fig. 3 in Molecular screening of tsetse flies and cattle reveal different Trypanosoma species including T. grayi and T. theileri in northern Cameroon

Fig. 3 The mequence of an amplccon obtacned flom tmetme fls gut wcth plcmelm mpeccfcc fol T. grayi. Speccfcc plcmelm (TGR-In plcmel met) talgeted agacnmt T. grayi amplcfced a 525 bp flagment (MG234546, Addctconal fcle 1: Table S4) flom tmetme fls gut mample (ID 237-51-00211-1-40-10, G. tachinoides, Addctconal fcle 1: Table S4). The flagment wam mequenced and alcgned wcth the collempondcng flagment of genomcc DNA flom T. grayi ANR4 (JMRU01000589)

Figure 2 in Genetic structure of Trypanosoma congolense "forest type" circulating in domestic animals and tsetse flies in the South-West region of Cameroon

Figure 2. NJ Tree based on Cavalli-Sforza and Edwards chord distance matrix of T. congolense "forest type" circulating in tsetse flies and domestic animals of Fontem.

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Figures 1–20 in Trypanosoma (Megatrypanum) lainsoni n. sp. from Mesomys hispidus (Rodentia: Echimyidae) in Brazil: trypomastigotes described from experimentally infected laboratory mice

Figures 1–20. Trypanosoma (Megatrypanum) lainsoni n. sp., parasites found in peripheral blood of experimentally infected mice; Figure 1: Epimastigote with signs of nuclear division; Figures 2–20: Trypomastigotes. Scale in Figure 20.

Fig. 5 in Peculiarities of Trypanosoma rangeli KP1(-) Strains Isolated from the Wild Rodent Phyllomys dasythrix (Santa Catarina, Brazil): Comparisons with T. rangeli KP1(+) strains and Trypanosoma lewisi (Kinetoplastea, Trypanosomatidae)

Fig. 5. kDNA minicircle amplicons presented by all KP1(+) and KP1(–) Trypanosoma rangeli strains examined in this study, and obtained by polymerase chain reaction using the primers 121/122. (M) molecular marker (100-bp DNA ladder).

Fig. 4 in Peculiarities of Trypanosoma rangeli KP1(-) Strains Isolated from the Wild Rodent Phyllomys dasythrix (Santa Catarina, Brazil): Comparisons with T. rangeli KP1(+) strains and Trypanosoma lewisi (Kinetoplastea, Trypanosomatidae)

Fig. 4. Isoenzyme profiles at IDH locus presented by KP1(+) and KP1(–) Trypanosoma rangeli strains and T. lewisi.

Fig. 3 in Peculiarities of Trypanosoma rangeli KP1(-) Strains Isolated from the Wild Rodent Phyllomys dasythrix (Santa Catarina, Brazil): Comparisons with T. rangeli KP1(+) strains and Trypanosoma lewisi (Kinetoplastea, Trypanosomatidae)

Fig. 3. Diagrammatic representation of the electrophoretic patterns of malate dehydrogenase (MDH), phosphoglucomutase (PGM), glucose phosphate isomerase (GPI) and malic enzyme (ME) displayed by KP1(–) and KP1(+) Trypanosoma rangeli strains, and T. lewisi.

Fig. 1 in Peculiarities of Trypanosoma rangeli KP1(-) Strains Isolated from the Wild Rodent Phyllomys dasythrix (Santa Catarina, Brazil): Comparisons with T. rangeli KP1(+) strains and Trypanosoma lewisi (Kinetoplastea, Trypanosomatidae)

Fig. 1. Camera lucida drawings of representative bloodstream trypomastigotes of Trypanosoma rangeli KP1(–) and KP1(+) strains from experimentally infected mice by metacyclic trypomastigotes grown in DMEM medium. The position of the nucleus in each trypomastigote is indicated by arrowheads. Parasites were from Giemsa-stained smears of each strain, as seen under optical microscopy (×1,000). In a trypomastigote of the strain SC-61 are indicated the reference points for taking measurements: anterior end (A), posterior end (P), nucleus (N) and free flagellum (F).

Fig. 2 in Peculiarities of Trypanosoma rangeli KP1(-) Strains Isolated from the Wild Rodent Phyllomys dasythrix (Santa Catarina, Brazil): Comparisons with T. rangeli KP1(+) strains and Trypanosoma lewisi (Kinetoplastea, Trypanosomatidae)

Fig. 2. Comparative growth in axenic cultures of Trypanosoma rangeli strains [KP1(–) and KP1(+)] and T. lewisi. Data (106 cells/μL) are the averages and the maximum growth from the values taken at the 4th, 7th, 10th, 13th, 17th and 20th days of cultivation in liver infusion-tryptose broth supplemented with 20% fetal calf serum (LIT-20) at 27.3 ± 0.4°C.

Fig. 3 in Cluster Analysis of Non-conserved Proteins of Trypanosoma cruzi Reference Strains Displays Parity between these Groupings (Peptidemes) and the Consensually Accepted Parasite Lineages

Fig. 3. Phenogram of the peptidemes (P) of eight Trypanosoma cruzi reference strains obtained using the SM coefficient and the UPGMA clustering algorithm, based on data from non-conserved proteins, as seen in SDS-PAGE analysis. The major peptidemes are indicated as mP 1 and mP 2. Their subgroups are identified on the right (P II, P VI, P I), and were numbered following their respective genetic types (TcII, TcVI, TcI), as currently used.

Fig. 1 in Cluster Analysis of Non-conserved Proteins of Trypanosoma cruzi Reference Strains Displays Parity between these Groupings (Peptidemes) and the Consensually Accepted Parasite Lineages

Fig. 1. Total protein profiles of eight Trypanosoma cruzi reference strains separated in 10% SDS-PAGE at 250 V, 25 mA, 90 min, and stained by Coomassie brilliant blue. The position of some conserved proteins is indicated on the right. M: molecular mass markers. (kDa) are indicated on the left.

Fig. 2 in Cluster Analysis of Non-conserved Proteins of Trypanosoma cruzi Reference Strains Displays Parity between these Groupings (Peptidemes) and the Consensually Accepted Parasite Lineages

Fig. 2. Diagrammatic representation of the twenty-two protein bands not shared by all Trypanosoma cruzi reference strains (nonconserved proteins), as visualized in SDS-PAGE. These bands were coded and analyzed by numerical taxonomy procedures. At the top is indicated the number of the major groups they belong, as identified by different approaches. The bands that were exclusive of one or more strains were highlighted with rectangles. M: molecular mass markers. (kDa) are indicated on the left.

Fig. 1 in In vitro Trypanosoma cruzi Growth Inhibition by Extremely Low-frequency Electromagnetic Fields

Fig. 1. Effect of 60 Hz sinusoidal magnetic fields at 2.0 mT and 24 h exposure on cell growth of T. cruzi epimastigote cultures. Bars represent arithmetical grouped means ± standard deviations.

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Fig. 2. A in First record of Trypanosoma infection in Mediterranean mouse (Mus macedonicus Petrov & Ružić, 1983) in Bulgaria

Fig. 2. A blood smear showing trypanosomes (white arrows) stained with acridine orange. Parasites (orange) are easily recognizable alongside red blood cells (mature – dark green; young – red) (magnification 400x).

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Fig. 1 in First record of Trypanosoma infection in Mediterranean mouse (Mus macedonicus Petrov & Ružić, 1983) in Bulgaria

Fig. 1. Location of the study area with investigated sites of Trypanosoma infection – site 1 (N42°3ʹ58.68ʺ; E24°49ʹ18.57ʺ) and site 2 (N42°3ʹ13.49ʺ; E24°49ʹ39.89ʺ).

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Fig. 3 in First record of Trypanosoma infection in Mediterranean mouse (Mus macedonicus Petrov & Ružić, 1983) in Bulgaria

Fig. 3. Microphotograph of Trypanosoma musculi from Mus macedonicus near Plovdiv in a thin blood smear (magnification 1000x).

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Fig. 2. ITS1 in Wild chimpanzees are infected by Trypanosoma brucei

Fig. 2. ITS1-based dendogram of trypanosomes from primate tissue and fecal samples. Sequences generated in this study are marked as follows: T ‾ tissue samples of apes (TA) and monkeys (TM); F ‾ fecal samples of apes (FA); sequences retrieved from GenBank are labeled with Latin names (Trypanosoma sp. ex Wildebeest JN673403, for T. theileri JX178185, HQ664848, and HQ664849).

opencc-by-4.0Dec 2015View details →

Fig. 1. ITS1 in Wild chimpanzees are infected by Trypanosoma brucei

Fig. 1. ITS1-based detection of trypanosomes in blood and feces of experimentally infected mice. (A‾D, I) detection in blood; (E‾H, J) detection in feces. (A, E) Trypanosoma b. brucei; (B, F) T. b. gambiense; (C, G) T. b. rhodesiense; (D, H) T. b. evansi; (I) blood from a non-infected mouse; (J) feces from a non-infected mouse; (K) negative control; (m) marker.