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2,849 results for “Adiposity”

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ClinicalTrials.gov36/100

Growth Hormone, Cardiovascular Risk, and Visceral Adiposity

ClinicalTrials.gov study NCT00131378. IPD Sharing: Not stated. Countries: 1. Publications: 7.

restrictedIPD-UNDECIDEDFeb 2026View details →
ClinicalTrials.gov36/100

Adipose-derived SVF for the Treatment of Knee OA

ClinicalTrials.gov study NCT02726945. IPD Sharing: NO. Countries: 1. Publications: 2.

closedIPD-NOFeb 2026View details →
ClinicalTrials.gov36/100

Effect of Time-restricted Eating on Catecholamine-sensitivity of Adipose Tissue in Obese Adults

ClinicalTrials.gov study NCT04916730. IPD Sharing: NO. Countries: 1. Publications: 0.

closedIPD-NOFeb 2026View details →
ClinicalTrials.gov36/100

Investigating Brown Adipose Tissue Activation in Humans

ClinicalTrials.gov study NCT01935791. IPD Sharing: NO. Countries: 1. Publications: 1.

closedIPD-NOFeb 2026View details →
ClinicalTrials.gov36/100

Adipose Tissue Extract and Platelet-rich Plasma Use for Wound Healing

ClinicalTrials.gov study NCT02799290. IPD Sharing: NO. Countries: 1. Publications: 2.

closedIPD-NOFeb 2026View details →
ClinicalTrials.gov36/100

Effectiveness of Autologous Adipose-derived Stem Cells in the Treatment of Knee Cartilage Injury

ClinicalTrials.gov study NCT03955497. IPD Sharing: NO. Countries: 1. Publications: 4.

closedIPD-NOFeb 2026View details →
dryad36/100

Lifecourse genome-wide association study meta-analysis refines the critical life stages for adiposity’s influence on breast cancer risk

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publicDec 2025View details →
dryad36/100

Data from: Brown adipose tissue and skeletal muscle coordinately contribute to thermogenesis in mice

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publicOct 2025View details →
dryad36/100

Maternal progesterone and adipose mPRε in pregnancy regulate the embryonic nutritional state

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publicFeb 2025View details →
dryad36/100

Data from: Adiposity is related to cerebrovascular and brain volumetry outcomes in the RUN DMC Study

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publicApr 2020View details →
dryad36/100

Data from: Adipose tissue explant culture using PDMS flow chambers: an alternative to static explant culture

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publicDec 2025View details →
dryad36/100

Data from: Development of an adipose-tropic AAV capsid ablating liver tropism

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publicMar 2025View details →
dryad36/100

Metabolomics data from: Dietary caloric input and tumor growth accelerate senescence and modulate liver and adipose tissue crosstalk

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publicDec 2024View details →
dryad36/100

Adipose-derived leptin and complement factor D mediate osteoarthritis severity and pain

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publicApr 2025View details →
dryad36/100

The association between long-term air pollution exposure and Chinese visceral adiposity index: A nationwide study of middle-aged and older adults

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publicJun 2025View details →
dryad36/100

Whole mount 3D imaged adipose tissue demonstrating sympathetic neurons and blood vessels during cold exposure and thermoneutrality

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publicFeb 2025View details →
dryad36/100

Adipose-derived stromal cells preserve pancreatic islet function in a transplantable 3D bioprinted scaffold

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publicJun 2022View details →
zenodo32/100

Neural priming of adipose-derived stem cells by cell-imprinted substrates

<p>Raw data and figure evaluation scripts for the manuscript <strong>Neural Priming of Adipose-Derived Stem Cells by Cell-Imprinted Substrates</strong></p>

opencc-by-4.0May 2020View details →
dryad32/100

Results from: Angiogenic property of silk fibroin scaffolds with adipose-derived stem cells on chick chorioallantoic membrane

<p>Angiogenesis is a key step in tissue regeneration and repair. Biomaterials that allow or promote angiogenesis are thus beneficial. In this study, angiogenic properties of salt-leached silk fibroin (SF) scaffolds seeded with human adipose stem cells (hADSCs) were studied using chick chorioallantoic membrane (CAM) as a model. The hADSC-seeded SF scaffolds (SF-hADSC) with porosity of 77.34 ± 6.96 % and pore diameter of 513.95 ± 4.99 µm were implanted on CAM of chick embryos that were on embryonic day 8 (E8) of development. The SF-hADSC scaffolds induced a spoke-wheel pattern of capillary network indicative of angiogenesis, which was evident since E11. Moreover, ingrowth of blood vessels into the scaffolds was seen in histological sections. The unseeded scaffolds induced the same extent of angiogenesis later on E14. In contrast, the control group could not induce angiogenesis to the same extent even as late. <i>In vitro</i> cytotoxicity tests and <i>in vivo</i> angioirritative study reaffirmed the biocompatibility of the scaffolds. This work highlighted that the biocompatible SF-hADSC scaffolds accelerates angiogenesis, and hence they can be a promising biomaterial for regeneration of tissues that require angiogenesis.</p>

opencc-zeroSep 2020View details →
dryad32/100

Magnetic resonance imaging reveals human brown adipose tissue is rapidly activated in response to cold

<p class="MsoNoSpacing"><b>Context.</b> In rodents, cold exposure induces the activation of brown adipose tissue (BAT) and the induction of intracellular triacylglycerol (TAG) lipolysis. However, in humans, the kinetics of supraclavicular (SCV) BAT activation and the potential importance of TAG stores remain poorly defined.</p> <p class="MsoNoSpacing"><b>Objective.</b> To determine the time course of BAT activation and changes in intracellular TAG using magnetic resonance imaging (MRI) assessment of the SCV (i.e. BAT depot) and fat in the posterior neck region (i.e. non BAT).</p> <p class="MsoNoSpacing"><b>Design.</b> Cross-sectional.</p> <p class="MsoNoSpacing"><b>Setting.</b> Clinical research centre.</p> <p class="MsoNoSpacing"><b>Patients or Other Participants.</b> Twelve healthy male volunteers ages 18-29 years [BMI=24.7±2.8kg/m<sup>2</sup> and body fat percentage = 25.0±7.4% (both mean±SD)].</p> <p class="MsoNoSpacing"><b>Intervention(s).</b> Standardized whole-body cold exposure (180 minutes at 18<span>°</span>C) and immediate re-warming (30 minutes at 32°C).</p> <p class="MsoNoSpacing"><b>Main Outcome Measure(s).</b> Proton density fat fraction (PDFF) and T2* of the SCV and posterior neck fat pads. Acquisitions occurred at 5-15 minute intervals during cooling and subsequent warming.</p> <p class="MsoNoSpacing"><b>Results.</b> SCV PDFF declined significantly after only 10 minutes of cold exposure [-1.6% (standard error (SE) 0.44%), <i>p</i>=0.007) and continued to decline until 35 minutes after which time it remained stable until 180 minutes. A similar time course was also observed for SCV T2*. In the posterior neck fat (non-BAT) there were no cold-induced changes in PDFF or T2*. Re-warming did not result in a change in SCV PDFF or T2*.</p> <p class="MsoNoSpacing"><b>Conclusions.</b> The rapid cold-induced decline in SCV PDFF suggests that in humans, BAT is activated quickly in response to cold and that TAG is a primary substrate.</p>

opencc-zeroOct 2019View details →

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International Brain Laboratory public data

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Last verified 2026-04-29Open record

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Last verified 2026-04-29Open record