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563 results for “psychosis”
Kufor-Rakeb Syndrome-Associated Psychosis: A Novel Loss-of-Function ATP13A2 Variant and Response to Antipsychotic Therapy
Open the record for dataset details and reuse information.
FIGURES 84–87 in Facial affect recognition in individuals at clinical high risk for psychosis
FIGURES 84–87. Mesoceration, aedeagi of holotypes. —84. M. brevigranum. —85. M. granulovestum. —86. M. truncatum. —87. M. umbrosum.
FIGURES 78–81 in Facial affect recognition in individuals at clinical high risk for psychosis
FIGURES 78–81. Mesoceration, aedeagi of holotypes. —78. M. concessum. —79. M. tabulare. —80. M. littlekarroo. —81. M. piceum.
FIGURES 71–74 in Facial affect recognition in individuals at clinical high risk for psychosis
FIGURES 71–74. Mesoceration, aedeagi of holotypes. —71. M. bispinum. —72. M. curvosum. —73. M. longipennis. —74. M. natalensis.
FIGURES 55–58 in Facial affect recognition in individuals at clinical high risk for psychosis
FIGURES 55–58. Mesoceration, aedeagi of holotypes. —55. M. barriotum. —56. M. incarinum. —57. M. semicarinulum. —58. M. compressum.
FIGURES 47–50 in Facial affect recognition in individuals at clinical high risk for psychosis
FIGURES 47–50. Mesoceration, aedeagi of holotypes. —47. M. periscopum. —48. M. splendorum. —49. M. maluti. —50. M. rapidensis.
FIGURES 32–35 in Facial affect recognition in individuals at clinical high risk for psychosis
FIGURES 32–35. Parasthetops, aedeagi of holotypes. —32. P. benefossus. —33. P. parallelus. —34. P. camurus. —35. P. reflexus.
FIGURES 63–66 in Facial affect recognition in individuals at clinical high risk for psychosis
FIGURES 63–66. Mesoceration, aedeagi of holotypes. —63. M. integer. —64. M. drakensbergensis. —65. M. concavum. —66. M. bicurvum.
FIGURES 26–29 in Facial affect recognition in individuals at clinical high risk for psychosis
FIGURES 26–29. Parasthetops, aedeagi of holotypes. —26. P. confluentus. —27. P. namibiensis. —28. P. rufulus. —29. P. striatus.
FIGURES 19–22 in Facial affect recognition in individuals at clinical high risk for psychosis
FIGURES 19–22. Parasthetops, aedeagi of holotypes. —19. P. retinaculus. —20. P. lemniscus. —21. P. semiplanus. —22. P. pampinus.
FIGURE 90 in Facial affect recognition in individuals at clinical high risk for psychosis
FIGURE 90. Allopatric geographical distributions, lateral habitus, and aedeagi of three species in the Mesoceration barriotum group.
FIGURES 88–89.—88 in Facial affect recognition in individuals at clinical high risk for psychosis
FIGURES 88–89.—88. Geographical distribution records for all species of Parasthetops. —89. Geographical distribution records for all species of Mesoceration.
FIGURES 40–43 in Facial affect recognition in individuals at clinical high risk for psychosis
FIGURES 40–43. Mesoceration, aedeagi of holotypes. —40. M. durabilis. —41. M. disjunctum. —42. M. repandum. —43. M. reticulatum.
FIGURES 11–14 in Facial affect recognition in individuals at clinical high risk for psychosis
FIGURES 11–14. Parasthetops, aedeagi of holotypes. —11. P. unicornus. —12. P. sebastiani. —13. P. buunicornus. —14. P. propitius.
FIGURES 3–6 in Facial affect recognition in individuals at clinical high risk for psychosis
FIGURES 3–6. Aedeagi of holotypes. —3. Prosthetops gladiator. —4. Prosthetops grandiceps. —5. Pterosthetops hawequas. —6. Pterosthetops equestrius.
Supplementary information for: Clinical, neuroimmunologic, and CSF investigations in first episode psychosis
<p><strong>Objectives: </strong>To report the neuropsychiatric features and frequency of N-methyl-d-aspartate receptor (NMDAR) and other neuronal IgG antibodies in patients with first episode psychosis (FEP), and assess the performance of reported warning signs and criteria for autoimmune psychosis (AP).</p> <p><strong>Methods:</strong> Prospective observational study of patients with FEP assessed for neuropsychiatric symptoms, serum and CSF neuronal antibodies (brain immunohistochemistry, cell-based assays, live neurons), and warning signs and criteria of AP. Previous autoimmune FEP series were reviewed.</p> <p><strong>Findings: </strong>105 patients were included; median age 30 years (range 14-75), 44 (42%) female. None had neuronal antibodies. Two/105 (2%) had CSF pleocytosis, 4/100 (4%) brain MRI abnormalities, and 3/73 (4%) EEG alterations. Thirty-four (32%) and 39 (37%) patients fulfilled two sets of warning signs of AP, and 21 (20%) fulfilled criteria of possible or probable AP, yet none developed AP. The cause of FEP was psychiatric in 101 (96%) and non-psychiatric in 4 (4%). During this study, 3 patients with psychosis caused by anti-NMDAR encephalitis were transferred to our center; 2 did not meet criteria for possible AP. Of 1159 reported FEP patients, only 7 (1%) had CSF studies; 36 (3%) had serum NMDAR-antibodies (without definite diagnosis of AP), and 4 had CSF NMDAR-antibodies (3 classic anti-NMDAR encephalitis and 1 with isolated psychiatric features).</p> <p><strong>Conclusions:</strong> NMDAR-antibodies were not found in patients with FEP unless they had anti-NMDAR encephalitis. Warning signs and criteria for AP have limited utility when neurological symptoms are absent or paraclinical tests are normal. A diagnostic algorithm for autoimmune FEP is provided.</p>
Cannabidiol for Treatment of Non-affective Psychosis and Cannabis Use
ClinicalTrials.gov study NCT04105231. IPD Sharing: YES. Countries: 1. Publications: 1.
Tracking Outcomes in Psychosis
ClinicalTrials.gov study NCT02882204. IPD Sharing: NO. Countries: 1. Publications: 4.
Longitudinal Long-term Study (10 Years) of the Sample of First Episode of Non-affective Psychosis: PAFIP
ClinicalTrials.gov study NCT02200588. IPD Sharing: NO. Countries: 1. Publications: 1.
Cognitive Remediation and Social Recovery in Early Psychosis
ClinicalTrials.gov study NCT04273685. IPD Sharing: NO. Countries: 1. Publications: 15.
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International Brain Laboratory public data
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OpenNeuro
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