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datasets available to search
ShareScore release 0.9.0
Dataset results
56 results for “serum albumin”
Recombinant Human Serum Albumin in Patients With Liver Cirrhosis and Ascites Subjects
ClinicalTrials.gov study NCT05249374. IPD Sharing: NO. Countries: 1. Publications: 0.
Recombinant Human Serum Albumin in Healthy Subjects
ClinicalTrials.gov study NCT05179265. IPD Sharing: NO. Countries: 1. Publications: 0.
Phase II/III Clinical Trial of Recombinant Human Serum Albumin in Cirrhotic Patients With Ascites
ClinicalTrials.gov study NCT06553456. IPD Sharing: NO. Countries: 1. Publications: 0.
Succinylated Human Serum Albumin (Suc-HSA) for HIV-1 Infection
ClinicalTrials.gov study NCT00128063. IPD Sharing: Not stated. Countries: 1. Publications: 0.
Correlation Between Serum Albumin Level and Severity of Hepatic Encephalopathy in Patients With Chronic Liver Disease in Sohag University Hospital
ClinicalTrials.gov study NCT06953921. IPD Sharing: UNDECIDED. Countries: 1. Publications: 0.
The Effect of Branched-chain Amino Acid on the Improvement of Serum Albumin Level in Cirrhotic Patients With Ascites
ClinicalTrials.gov study NCT02755701. IPD Sharing: NO. Countries: 2. Publications: 0.
Recombinant Human Serum Albumin in the Treatment of AD (Alzheimer Disease) Exploratory Clinical Trials
ClinicalTrials.gov study NCT06489015. IPD Sharing: NO. Countries: 1. Publications: 0.
Serum Albumin Ratios Can Improve Sepsis Mortality Prediction
ClinicalTrials.gov study NCT06640504. IPD Sharing: Not stated. Countries: 1. Publications: 0.
Transcriptomic fingerprints of C. elegans exposed to superparamagnetic iron oxide nanoparticles coated with a monolayer of bovine serum albumin (BSA-SPIONs)
GEO Series GSE93186. Caenorhabditis elegans. 6 samples. Type: Expression profiling by array.
RNAseqencing of wild type (WT) and Ripk3-/- (KO) AML-12 cells in the presence (PA) of absence (C) of 125 µM palmitate-bovine serum albumin for 24 h
GEO Series GSE199155. Mus musculus. 20 samples. Type: Expression profiling by high throughput sequencing.
Hormone Therapy on Serum Ischemia Modified Albumin in Menopausal Women
ClinicalTrials.gov study NCT04424108. IPD Sharing: Not stated. Countries: 0. Publications: 0.
CRP to Serum Albumin Ratio in Type 2 DM é Diabetic Nephropathy
ClinicalTrials.gov study NCT05681286. IPD Sharing: Not stated. Countries: 0. Publications: 0.
High Dose Hyperoncotic Serum Albumin for the Treatment of the Acute Phase of Severe Head Injury
ClinicalTrials.gov study NCT00152685. IPD Sharing: Not stated. Countries: 1. Publications: 0.
mRNA transcriptome data of granulosa cells cultured either with BSA (bovine serum albumin) or NEFA (non estrified fatty acid) mix
GEO Series GSE302841. Bos taurus. 8 samples. Type: Expression profiling by array.
Microarray of PBMC from patients with decompensated cirrhosis (AD) and ACLF stimulated with CpG-DNA and treated with human serum albumin (HSA)
GEO Series GSE146462. Homo sapiens. 24 samples. Type: Expression profiling by array.
Dataset related to the article : "The Burden of Impaired Serum Albumin Antioxidant Properties and Glyco-Oxidation in Coronary Heart Disease Patients with and without Type 2 Diabetes Mellitus"
<p>This record contains raw data related to the article: The Burden of Impaired Serum Albumin Antioxidant Properties and Glyco-Oxidation in Coronary Heart Disease Patients with and without Type 2 Diabetes Mellitus</p> <p>Abstract</p> <p>Human serum albumin (HSA) has an important antioxidant activity due to the presence of the reduced cysteine at position 34, which represents the most abundant free thiol in the plasma. In oxidative-based diseases, HSA undergoes S-thiolation (THIO-HSA) with changes in the antioxidant function of albumin that could contribute to the progression of the disease. The aim of this study was to verify, for the first time, the different burdens of THIO-HSA, glycated HSA (GLY-HSA), and advanced glycation end products (AGE) accumulation both in type 2 diabetes mellitus (T2DM) patients and in non-diabetic patients, with or without coronary heart disease (CHD). In this study, we assessed the presence of modified forms of HSA, THIO-HSA, and GLY-HSA by means of mass spectrometry in 33 patients with both T2DM and CHD, in 31 patients with T2DM and without CHD, in 30 patients without diabetes with a history of CHD, and 27 subjects without diabetes and CHD. All the patients' anthropometric and clinical data were recorded including age, sex, duration of diabetes, body mass index (BMI), blood pressure, and history of CHD defined with anamnestic data. Metabolic parameters, such as fasting plasma glucose (FPG), glycated hemoglobin (HbA1c), lipids, pentosidine, AGE, receptor for advanced glycation end-products (RAGE) and its soluble form (sRAGE), were measured. AGE and pentosidine are significantly higher in T2DM patients with and without CHD with respect to non-diabetic patients with CHD and control subjects. RAGE levels are significantly higher in T2DM patients with respect to non-diabetic patients, and among T2DM patients, the group with CHD showed significantly higher RAGE levels than those without CHD (217 ± 171 pg/mL and 140 ± 61 pg/mL, respectively). Albumin isoforms discriminate between non-diabetic patients with CHD and T2DM patients with and without CHD and control subjects, with GLY-HSA levels higher in T2DM with and without CHD, and THIO-HSA higher in CHD patients without T2DM. Finally, we demonstrated that the oxidized forms of HSA can increase the expression of the inflammatory cytokine Tumor Necrosis Factor-alpha (TNFα) in monocytic cells. In patients with CHD, GLY-HSA and THIO-HSA have a different prevalent distribution, the first one prevailing in patients with T2DM and the second one in patients without T2DM. These findings suggest that albumin quality and homeostasis balance between glyco-oxidation and thiolation might have an impact on the antioxidant defense system in cardiovascular diseases.</p>
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