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1,242 results for “Cell proliferation”
Data: DEAE-Dextran enhances the lentiviral transduction of primary human mesenchymal stromal cells from all major tissue sources without affecting their proliferation and phenotype
<p>This data set includes all the raw data collected for the following article: "DEAE-Dextran enhances the lentiviral transduction of primary human mesenchymal stromal cells from all major tissue sources without affecting their proliferation and phenotype"</p>
Underlying data for: "Capturing the mechanosensitivity of cell proliferation in models of epithelium"
<p>For our publication "Capturing the mechanosensitivity of cell proliferation in models of epithelium" (available as a preprint at <a title="BioRXiv Link" href="https://doi.org/10.1101/2023.01.31.526438" target="_blank" rel="noopener">DOI: 10.1101/2023.01.31.526438 )</a> we here provide the raw data for the included plots and the code used to generate the Delayed Fisher Kolmogorov (DFK) data referenced in the main publication</p> <p>The archive '<em>underlying_data.zip</em>' contains raw data underlying the plots in the publication. <br>The archive '<em>puls_proliferation_rate-1.0.zip</em>' contains the code for generating DFK trajectories referenced in the publication and its SI. <br>The archive '<em>ddesolver-1.0.zip</em>' contains the python code for solving delayed differential equations used by the puls_proliferation_rate project. It is included to ensure completeness and reproducibility of the simulations. </p>
Code and data from "Mother cells control daughter cell proliferation in intestinal organoids to minimize proliferation fluctuations"
<p>Includes the microscopy images, cell tracking data and scripts used in the publication Huelsz-Prince, Guizela, et al. "Mother cells control daughter cell proliferation in intestinal organoids to minimize proliferation fluctuations." <em>eLife </em> 11:e80682 (2022). <a href="https://doi.org/10.7554/eLife.80682"> https://doi.org/10.7554/eLife.80682</a> .</p>
Data_Figure 2_Impact of 17β‑HSD12, the 3‑ketoacyl‑CoA reductase of long‑chain fatty acid synthesis, on breast cancer cell proliferation and migration
<p>Data of figure 2 from Impact of 17β‑HSD12, the 3‑ketoacyl‑CoA reductase of long‑chain fatty acid synthesis, on breast cancer cell proliferation and migration</p> <p>Dataset (doi: 10.1007/s00018-019-03227-w) contains the original figure as TIF-format (10.1007_s00018-019-03227-w_CMLS_Fig2). Corresponding raw data obtained from a) Migration potential as four files in CSV format (31003A-179400_ date_examiner_17BHSD12_16_1_1-4. b) mRNA content analyzed by RT-PCR provided as ten files in CSV format (31003A-179400_date_examiner_17BHSD12_1_1-2_1-6) and proliferation investigation on xCELLigence provided as six files in CSV format (31003A-179400_date_examiner_17BHSD12_9_2_1-6). All further experiment related information and subsequent data analysis provided as four meta-data-files (31003A-179400_ date_examiner_17BHSD12_16/1/9_dataset_M_1) as TXT format.</p>
Data_Figure 6_Impact of 17β‑HSD12, the 3‑ketoacyl‑CoA reductase of long‑chain fatty acid synthesis, on breast cancer cell proliferation and migration
<p>Data of figure 6 from Impact of 17β‑HSD12, the 3‑ketoacyl‑CoA reductase of long‑chain fatty acid synthesis, on breast cancer cell proliferation and migration</p> <p>Dataset (doi: 10.1007/s00018-019-03227-w) contains the original figure as TIF-format (10.1194_jlr.M092908_Fig. 6). Corresponding raw data obtained from a1/2) cellomics HTC array scan analysis provided as six files in CSV format (31003A-179400_Date_examiner_17BHSD12_8_6_1-6), b1/2) oxygen consumption rate (OCR) and extracellular acidification rate (ECAR) provided as 10 files in CSV format (31003A-179400_20190521_MT_17BHSD12_10_3-4_1-5); c 1/2 ), cellomics HTC array scan analysis provided as 12 files in CSV format (31003A-179400_Date_examiner_17BHSD12_8_7-8_1-8). d) Western blot and densitometry provided as eight files in CSV format (31003A-179400_Date_examiner_17BHSD12_2_3-4_1-5). All further experiment related information protocols and subsequent data analysis provided as meta-data-files (31003A-179400_date_examiner_17BHSD12_8/10/2_dataset_M_1) as TXT format and (31003A-179400_date_examiner_17BHSD12_2_dataset_M_2-3) as PNG format.</p>
Data_Figure 7_Impact of 17β‑HSD12, the 3‑ketoacyl‑CoA reductase of long‑chain fatty acid synthesis, on breast cancer cell proliferation and migration
<p>Data of figure 7 from Impact of 17β‑HSD12, the 3‑ketoacyl‑CoA reductase of long‑chain fatty acid synthesis, on breast cancer cell proliferation and migration</p> <p>Dataset (doi: 10.1007/s00018-019-03227-w) contains the original figure as TIF-format (10.1194_jlr.M092908_Fig. 7). Corresponding raw data obtained from a1/2) Western blot and densitometry provided as eight files in CSV format (31003A-179400_date_examiner_17BHSD12_2_5-6_1-6); b) mRNA content analyzed by RT-PCR provided as four files in CSV format (31003A-179400_date_examiner_17BHSD12_1_6_1-4); c 1/2) cellomics HTC array scan analysis provided as 11 files in CSV format (31003A-179400_Date_examiner_17BHSD12_8_9-10_1-6); d1/2); Western blot and densitometry provided as eight files in CSV format (31003A-179400_date_examiner_17BHSD12_2_7-8_1-6). All further experiment related information protocols and subsequent data analysis provided as meta-data-files (31003A-179400_date_examiner_17BHSD12_2/1/8_dataset_M_1) as TXT format and (31003A-179400_date_examiner_17BHSD12_2_dataset_M_2-3) as PNG format.</p>
Data_Figure 5_Impact of 17β‑HSD12, the 3‑ketoacyl‑CoA reductase of long‑chain fatty acid synthesis, on breast cancer cell proliferation and migration
<p>Data of figure 5 from Impact of 17β‑HSD12, the 3‑ketoacyl‑CoA reductase of long‑chain fatty acid synthesis, on breast cancer cell proliferation and migration</p> <p>Dataset (doi: 10.1007/s00018-019-03227-w) contains the original figure as TIF-format (10.1194_jlr.M092908_Fig. 5). Corresponding raw data obtained from oxygen consumption rate (OCR) and extracellular acidification rate (ECAR) provided as 10 files in CSV format (31003A-179400_20190521_MT_17BHSD12_10_1-2_1-5). All further experiment related information and subsequent data analysis provided as two meta-data-file: (31003A-179400_20190521_MT_17BHSD12_10_1-2_1) as TXT format.</p>
Data_Figure 4_Impact of 17β‑HSD12, the 3‑ketoacyl‑CoA reductase of long‑chain fatty acid synthesis, on breast cancer cell proliferation and migration
<p>Data of figure 4 from Impact of 17β‑HSD12, the 3‑ketoacyl‑CoA reductase of long‑chain fatty acid synthesis, on breast cancer cell proliferation and migration</p> <p>Dataset (doi: 10.1007/s00018-019-03227-w) contains the original figure as TIF-format (10.1194_jlr.M092908_Fig. 4). Corresponding raw data obtained from: a1/2) Migration potential as five files in CSV format (31003A-179400_date_examiner_17BHSD12_16_3_1-5); b 1/2) Migration potential as four files in CSV format (31003A-179400_date_examiner_17BHSD12_16_4_1-4); c1/2/3) mRNA content analyzed by RT-PCR provided as four files in CSV format (31003A-179400_date_examiner_17BHSD12_1_4_1-4); cellomics HTC array scan analysis provided as three files in CSV format (31003A-179400_Date_examiner_17BHSD12_8_3-4_1-4); d) Migration potential as five files in CSV format (31003A-179400_ date_examiner_17BHSD12_16_5_1-5); e) mRNA content analyzed by RT-PCR provided as six files in CSV format (31003A-179400_date_examiner_17BHSD12_1_5_1-6); f) Migration potential as four files in CSV format (31003A-179400_date_examiner_17BHSD12_16_6_1-4), cellomics HTC array scan analysis provided as three files in CSV format (31003A-179400_Date_examiner_17BHSD12_8_5_1-5); g) ELISA measurement provided as four files in CSV format (31003A-179400_date_examiner_17BHSD12_20_1_1-4). All further experiment related information protocols and subsequent data analysis provided as 10 meta-data-files (31003A-179400_date_examiner_17BHSD12_8/16/1/20_dataset_M_1) as TXT format.</p>
Data_supplemental figure 2_Impact of 17β‑HSD12, the 3‑ketoacyl‑CoA reductase of long‑chain fatty acid synthesis, on breast cancer cell proliferation and migration
<p>Data of supplemental figure 2 from Impact of 17β‑HSD12, the 3‑ketoacyl‑CoA reductase of long‑chain fatty acid synthesis, on breast cancer cell proliferation and migration</p> <p>Dataset (doi: 10.1007/s00018-019-03227-w) contains the original figure as TIF format (10.1194_jlr.M092908_Fig. S2). Corresponding raw data obtained from cellomics HTC array scan analysis provided as seven files in CSV format (31003A-179400_Date_examiner_17BHSD12_8_11-12_1-4) All further experiment related information protocols as meta-data-files (31003A-179400_date_examiner_17BHSD12_8_11-12_M_1) as TXT format.</p>
Data from: Predation drives the evolution of brain cell proliferation and brain allometry in male Trinidadian killifish, Rivulus hartii
<p>The external environment influences brain cell proliferation, and this might contribute to brain plasticity underlying adaptive behavioural changes. Additionally, internal genetic factors influence brain cell proliferation rate. However, to date, researchers have not examined the importance of environmental vs. genetic factors in causing natural variation in brain cell proliferation. Here, we examine brain cell proliferation and brain growth trajectories in free-living populations of Trinidadian killifish, Rivulus hartii, exposed to contrasting predation environments. Compared to populations without predators, populations in high predation environments exhibited higher rates of brain cell proliferation and a steeper brain growth trajectory (relative to body size). To test whether these differences in the wild persist in a common garden environment, we reared first generation fish originating from both predation environments in uniform laboratory conditions. Just as in the wild, brain cell proliferation and brain growth in the common garden were greater in high predation populations than in no predation populations. The similar results in field and common garden studies indicate that population differences in these brain features are intrinsic, probably genetic, differences arising from natural selection acting on overall brain growth and life history rather than differences arising through phenotypic plasticity.</p>
Figure 4 in Investigation the effects of vitreous humor on proliferation and dedifferentiation of differentiated NTERA2 cells
Figure 4. Western blot analysis show that the NT2 cells are known as perfectly positive cells for OCT4 protein expression.
Figure 2 in Investigation the effects of vitreous humor on proliferation and dedifferentiation of differentiated NTERA2 cells
Figure 2. Cell cycle analysis of NT2-RA cells after treatment with different amounts of VH at (A) 3 days and (B) 6 days post-treatment. After treatment with RA the number of cells at S phase decreased. These numbers were recovered after three and six days treatment with different amounts of VH.
◂Fig. 5 Gametogenesis in male and female Veneriserva pygoclava. A–D Semi-thin histological sections of female Veneriserva pygoclava, stained with toluidine blue. A Cross-section of a female Veneriserva. B Close-up of large mature oocytes without discernible nurse cells. C Developing oocytes attached to mesenteries (mes), and oogonia proliferating from the ventral side of the dorsal blood vessel (bv). D Details of vitellogenic oocytes and nurse cells. Arrowheads indicate brownstained yolk platelets and yolk bodies. E Live sperm cells captured in a light micrograph. F–G Cross-sections of male Veneriserva. Note the absence of a gut in the cross-sections. Abbreviations—ac acicula, acr acrosome, bv blood vessel, coe coelomic cavity, mes mesentery, nc nurse cell, nn nurse cell nucleus, nu sperm cell nucleus, Oo oocyte, on oocyte nucleus, sp spermatogonia, vnc ventral nerve cord in Hardly Venus's servant-morphological adaptations of Veneriserva to an endoparasitic lifestyle and its phylogenetic position within Dorvilleidae (Annelida)
◂Fig. 5 Gametogenesis in male and female Veneriserva pygoclava. A–D Semi-thin histological sections of female Veneriserva pygoclava, stained with toluidine blue. A Cross-section of a female Veneriserva. B Close-up of large mature oocytes without discernible nurse cells. C Developing oocytes attached to mesenteries (mes), and oogonia proliferating from the ventral side of the dorsal blood vessel (bv). D Details of vitellogenic oocytes and nurse cells. Arrowheads indicate brownstained yolk platelets and yolk bodies. E Live sperm cells captured in a light micrograph. F–G Cross-sections of male Veneriserva. Note the absence of a gut in the cross-sections. Abbreviations—ac acicula, acr acrosome, bv blood vessel, coe coelomic cavity, mes mesentery, nc nurse cell, nn nurse cell nucleus, nu sperm cell nucleus, Oo oocyte, on oocyte nucleus, sp spermatogonia, vnc ventral nerve cord
Pyruvate transamination and NAD biosynthesis enable proliferation of succinate dehydrogenase-deficient cells by supporting aerobic glycolysis
<p>Data supporting results published by Ricci et al. Pyruvate transamination and NAD biosynthesis enable proliferation of succinate dehydrogenase-deficient cells by supporting aerobic glycolysis. Cell Death and Disease (2023) 14:403 (https://doi.org/10.1038/s41419-023-05927-5).</p>
Fig. 9. T. thermophila cells immunostained with monoclonal anti-cdc14A in Effects of Roscovitine on Schedule of Divisional Morphogenesis, Basal Bodies Proliferation and Cell Divisions in Tetrahymena thermophila
Fig. 9. T. thermophila cells immunostained with monoclonal anti-cdc14A antibody. Cs and nCS – apical couplets of basal bodies for proter and opisthe, respectively; Ma – macronucleus. Other explanations as in Figs 1 and 8. Bar: 10 µm, bar in D for B–D.
Fig. 7. T. thermophila cells immunostained with the antiserum against component B in Effects of Roscovitine on Schedule of Divisional Morphogenesis, Basal Bodies Proliferation and Cell Divisions in Tetrahymena thermophila
Fig. 7. T. thermophila cells immunostained with the antiserum against component B of epiplasm. DF – staining around the deep fibers. Other explanations as in Fig. 1. Bar: 10 µm.
Fig. 2 in Effects of Roscovitine on Schedule of Divisional Morphogenesis, Basal Bodies Proliferation and Cell Divisions in Tetrahymena thermophila
Fig. 2. Localization of nuclei in control and roscovitine treated T. tetrmophila. The cells were immunostained with the anti-centrin 20H5 antibody and stained with DAPI. A and B – control cells in the VI stage of morphogenesis, and early cytokinesis, respectively; C and D – roscovitine treated cells (4 h) in the VI stage of morphogenesis and in cytokinesis respectively. Arrows – micronuclei (in the C micronucleus in opisthe is out of focus). Bar: 10 µm.
Fig. 4. Postdivider cells after 5.5 h in Effects of Roscovitine on Schedule of Divisional Morphogenesis, Basal Bodies Proliferation and Cell Divisions in Tetrahymena thermophila
Fig. 4. Postdivider cells after 5.5 h of roscovitine treatment. Cells were immunostained with anti-centrin 20H5 antibody. A and A' – ventral and dorsal views of the same proter cell with remnants of the OA2; B–C opisthe cells, B – opisthe with ARF on dorsal side, C – opisthe with remnants of oral structures; D and D' – ventral and dorsal views of the same cell; E and E' – ventral and dorsal views of the same opisthe during pinching off the OA. Other explanations as in Fig. 1. Bar: 10 µm for A–E'.
Fig. 5 in Effects of Roscovitine on Schedule of Divisional Morphogenesis, Basal Bodies Proliferation and Cell Divisions in Tetrahymena thermophila
Fig. 5. Mean cell sizes, positions of OA1, OA2 and fission zone in control and in roscovitine treated cells. Upper panel: early and late control dividers, lower panel: early dividers after 3.5 h roscovitine treatment, late dividers after 3.5 and 5.5 h roscovitine treatment. The shape of cells is represented by elipses. Solid horizontal lines – cells "equator", dotted horizontal lines – positions of the fission furrow. All sizes and distances were drawn to the same scale. Bar: 10 µm. The measurements represent means of at least 10 specimens for each cell sample.
Fig. 6 in Effects of Roscovitine on Schedule of Divisional Morphogenesis, Basal Bodies Proliferation and Cell Divisions in Tetrahymena thermophila
Fig. 6. Correlation of length of proters and shift of the fission zone in relation to OA2 induced by roscovitine in late dividers. The shift of the fission zone was measured as a difference between length of the proters and distance from cell apex to the anterior end of the OA2 (ordinate). Each point in this diagram represent individual cell. Open symbols – untreated cells, close symbols – cells treated with roscovitine for 3.5 and 5.5 h (pooled).
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Allen Brain Atlas
Allen Brain Atlas is an Allen Institute collection of brain map atlases, datasets, APIs, and analysis tools covering mouse, human, and non-human primate brain resources.
Annotated Behaviour and Observability Dataset (ABODe)
ABODe is a University of Edinburgh DataShare dataset for behavior classification in group-housed mice using home-cage video, identities, bounding boxes, ground-plate positions, and annotator labels.
DANDI Archive for NWB datasets
DANDI is a BRAIN Initiative archive for publishing and sharing neurophysiology data, including electrophysiology, optophysiology, and behavioral data packaged as NWB and related standards.
International Brain Laboratory public data
The International Brain Laboratory public data releases expose standardized mouse decision-making experiments, including Neuropixels recordings, widefield calcium imaging, behavior, and session metadata accessed through the ONE API.
OpenNeuro
OpenNeuro is a free, open platform for sharing neuroimaging datasets, with public search, dataset pages, and download paths for web, S3, DataLad, and the OpenNeuro CLI.