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13 results for “Rotylenchulus reniformis”
Figure 6 in Evaluation of Meloidogyne incognita and Rotylenchulus reniformis nematode-resistant cotton cultivars with supplemental Corteva Agriscience nematicides
Figure 6: Field trial R. reniformis eggs per gram of root collected from four root systems. LS means for PHY 332 W3FE (R) and PHY 340 W3FE (S) cotton and nematicide combination at 40 DAP in 2021. P-value for Type III fixed effects for the Variety x Nematicide interaction was 0.0441. Nematicide treatments included no-nematicide control, Reklemel (0.28 L/ha) + Vydate C-LV (1.24 L/ha), C-LV (0.28 +1.24 L/ha), Reklemel (0.56 L/ha) + Vydate C-LV (2.5 L/ha), Reklemel (1.13 L/ha) + Vydate C-LV (5.0 L/ha), BIOST Nematicide 100 (0.026 mg ai/seed), BIOST Nematicide 100, Reklemel (0.28 L/ha) + Vydate C-LV (1.24 L/ha), BIOST Nematicide 100 (0.026 mg ai/seed), Reklemel (0.56 L/ha) + Vydate C-LV (2.5 L/ha), BIOST Nematicide 100 (0.026 mg ai/seed), Reklemel (1.13 L/ha) + Vydate C-LV (5.0 L/ha).
Figure 1 in Evaluation of Meloidogyne incognita and Rotylenchulus reniformis nematode-resistant cotton cultivars with supplemental Corteva Agriscience nematicides
Figure 1: M. incognita PHY 340 W3FE susceptible variety on the left and the resistant PHY 360 W3FE on the right at 106 DAP.
Figure 1 in Evaluation of perennial Glycine species for response to Meloidogyne incognita, Rotylenchulus reniformis, and Pratylenchus penetrans
Figure 1: Representative images of soybean and perennial Glycine species roots at 8 weeks post-inoculation of Meloidogyne incognita. Gall index rating is given in parentheses A, susceptible check G. max cv. Pickett 71. B, resistant check G. max cv. Forrest. C, G. tomentella PI 339655. D, G. latifolia PI 559298. E, G. latifolia PI 559300. F, G. tabacina PI 373990.
Figure 5 in Evaluation of Meloidogyne incognita and Rotylenchulus reniformis nematode-resistant cotton cultivars with supplemental Corteva Agriscience nematicides
Figure 5: Field trial R. reniformis eggs per gram of root collected from four root systems LS means for PHY 332 W3FE (R) and PHY 340 W3FE (S) cotton and nematicide combination at 40 DAP in 2020. P-value for Type III fixed effects for the Variety x Nematicide interaction was 0.0178. Nematicide treatments included no-nematicide control, Reklemel (0.28 L/ha) + Vydate C-LV (1.24 L/ha), C-LV (0.28 +1.24 L/ha), Reklemel (0.56 L/ha) + Vydate C-LV (2.5 L/ha), Reklemel (1.13 L/ha) + Vydate C-LV (5.0 L/ha), BIOST Nematicide 100 (0.026 mg ai/seed), BIOST Nematicide 100, Reklemel (0.28 L/ha) + Vydate C-LV (1.24 L/ha), BIOST Nematicide 100 (0.026 mg ai/seed), Reklemel (0.56 L/ha) + Vydate C-LV (2.5 L/ha), BIOST Nematicide 100 (0.026 mg ai/seed), Reklemel (1.13 L/ha) + Vydate C-LV (5.0 L/ha).
Figure 2 in Evaluation of Meloidogyne incognita and Rotylenchulus reniformis nematode-resistant cotton cultivars with supplemental Corteva Agriscience nematicides
Figure 2: Field trial M. incognita eggs per gram of root collected from four root systems LS means for PHY 360 W3FE (R) and PHY 340 W3FE (S) cotton and nematicide combination at 40 DAP in 2020. P-value for Type III fixed effects for the Variety x Nematicide interaction was 0.0368. Nematicide treatments included no-nematicide control, Reklemel (0.28 L/ha) + Vydate C-LV (1.24 L/ha), C-LV (0.28 +1.24 L/ha), Reklemel (0.56 L/ha) + Vydate C-LV (2.5 L/ha), Reklemel (1.13 L/ha) + Vydate C-LV (5.0 L/ha), BIOST Nematicide 100 (0.026 mg ai/seed), BIOST Nematicide 100, Reklemel (0.28 L/ha) + Vydate C-LV (1.24 L/ha), BIOST Nematicide 100 (0.026 mg ai/seed), Reklemel (0.56 L/ha) + Vydate C-LV (2.5 L/ha), BIOST Nematicide 100 (0.026 mg ai/seed), Reklemel (1.13 L/ha) + Vydate C-LV (5.0 L/ha).
Figure 3 in Evaluation of Meloidogyne incognita and Rotylenchulus reniformis nematode-resistant cotton cultivars with supplemental Corteva Agriscience nematicides
Figure 3: Field trial M. incognita eggs per gram of root collected from four root systems LS means for PHY 360 W3FE (R) and PHY 340 W3FE (S) cotton and nematicide combination at 40 DAP in 2021. P-value for Type III fixed effects for the Variety x Nematicide interaction was 0.0272. Nematicide treatments included no-nematicide control, Reklemel (0.21 L/ha) + Vydate C-LV (0.88 L/ha) Reklemel (0.28 L/ha) + Vydate C-LV (1.24 L/ha, Reklemel (0.56 L/ha) + Vydate C-LV (2.5 L/ha), BIOST Nematicide 100 (0.026 mg ai/seed), BIOST Nematicide 100, Reklemel (0.21 L/ ha) + Vydate C-LV (0.88 L/ha), BIOST Nematicide 100 (0.026 mg ai/seed), Reklemel (0.28 L/ha) + Vydate C-LV (1.24 L/ha), BIOST Nematicide 100 (0.026 mg ai/seed), Reklemel (0.56 L/ha) + Vydate C-LV (2.50 L/ha).
Figure 4 in Evaluation of Meloidogyne incognita and Rotylenchulus reniformis nematode-resistant cotton cultivars with supplemental Corteva Agriscience nematicides
Figure 4: Auburn University's Tennessee Valley Research Extension Center showing the nematode susceptible PHY 340 W3FE on the left and the R. reniformis-resistant PHY 332 W3FE on the right 102 DAP.
Figure 2 in Toxicity of Tioxazafen to MelOIDOgYNe INCOgNITa and ROTYleNCHUlUS reNIfOrmIS
Figure 2: Effect of tioxazafen on hatch of M. incognita and R. reniformis. Different letters over bars per sample day indicate a significant difference at α = 0.05 according to Fisher's LSD procedure. LSD, least significant difference.
Figure 1 in Toxicity of Tioxazafen to MelOIDOgYNe INCOgNITa and ROTYleNCHUlUS reNIfOrmIS
Figure 1: Relationship between the paralyses of M. incognita and R. reniformis treated for 24 hr and 48 hr with water solutions of tioxazafen. Equations were derived by nonlinear regression of probit analysis. For each equation, the R2 value was 0.99 (P = 0.0001).
Figure 4 in Toxicity of Tioxazafen to MelOIDOgYNe INCOgNITa and ROTYleNCHUlUS reNIfOrmIS
Figure 4: Effect of low concentrations of tioxazafen on infectivity of M. incognita and R. reniformis on tomato roots. Different letters over bars indicate a significant difference at α = 0.05 according to Fisher's LSD procedure. LSD, least significant difference.
Figure 3 in Toxicity of Tioxazafen to MelOIDOgYNe INCOgNITa and ROTYleNCHUlUS reNIfOrmIS
Figure 3: Recovery of M. incognita and R. reniformis treated with tioxazafen. Each species was treated with water solutions corresponding to its 48-hr EC50 value of tioxazafen for 24 hr, and then rinsed and transferred to distilled water. Different letters over bars indicate a significant difference at α = 0.05 according to Fisher's LSD procedure. LSD, least significant difference.
Data from: Functional C-terminally encoded peptide (CEP) plant hormone domains evolved de novo in the plant parasite Rotylenchulus reniformis
Sedentary plant-parasitic nematodes (PPNs) induce and maintain an intimate relationship with their host, stimulating cells adjacent to root vascular tissue to re-differentiate into unique and metabolically active 'feeding sites'. The interaction between PPNs and their host is mediated by nematode effectors. We describe the discovery of a large and diverse family of effector genes, encoding C-TERMINALLY ENCODED PEPTIDE (CEP) plant hormone mimics (RrCEPs), in the syncytia-forming plant parasite Rotylenchulus reniformis. The particular attributes of RrCEPs distinguish them from all other CEPs, regardless of origin. Together with the distant phylogenetic relationship of R. reniformis to the only other CEP-encoding nematode genus identified to date (Meloidogyne), this suggests that CEPs probably evolved de novo in R. reniformis. We have characterized the first member of this large gene family (RrCEP1), demonstrating its significant up-regulation during the plant–nematode interaction and expression in the effector-producing pharyngeal gland cell. All internal CEP domains of multi-domain RrCEPs are followed by di-basic residues, suggesting a mechanism for cleavage. A synthetic peptide corresponding to RrCEP1 domain 1 is biologically active and capable of up-regulating plant nitrate transporter (AtNRT2.1) expression, whilst simultaneously reducing primary root elongation. When a non-CEP-containing, syncytia-forming PPN species (Heterodera schachtii) infects Arabidopsis in a CEP-rich environment, a smaller feeding site is produced. We hypothesize that CEPs of R. reniformis represent a two-fold adaptation to sustained biotrophy in this species: (i) increasing host nitrate uptake, whilst (ii) limiting the size of the syncytial feeding site produced.
Data from: Functional C-terminally encoded peptide (CEP) plant hormone domains evolved de novo in the plant parasite Rotylenchulus reniformis
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