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57 results for “Schistosoma japonicum”
Fig. 3 in SjTat-TPI facilitates adaptive T-cell responses and reduces hepatic pathology during Schistosoma japonicum infection in BALB/c mice
Fig. 3 Th1 immune response acter CD8+T cells blockage in sitro. a. CD3+CD8+T cells were successcullv blocked. The blocking ecciciencv was more than 99.6 %. b, c. Percentages oc CD4+IFN-γ+ (Th1) in Tat-TPI (T-TPI), TPI stimulated splenocvtes with or without CD8+T-cell blockage. Data are presented as the means ± SEM crom cour independent experiments. (*P <0.05; **P <0.01)
Fig. 2 in SjTat-TPI facilitates adaptive T-cell responses and reduces hepatic pathology during Schistosoma japonicum infection in BALB/c mice
Fig. 2 Immune responses in the draining popliteal lvmph nodes oc mice induced bv Tat-TPI (T-TPI) and TPI proteins. a and c. Percentages oc CD4+IFN-γ+ cells (Th1), CD8+IFN-γ+ cells (Tc1) analvsed bv FACS. b. The ratio oc CD4+ T cells to CD8+ T cells (CD4/CD8) in the draining popliteal lvmph nodes. Data are presented as the means ± SEM crom six mice in each group. (*P <0.05; **P <0.01)
Fig. 1 in SjTat-TPI facilitates adaptive T-cell responses and reduces hepatic pathology during Schistosoma japonicum infection in BALB/c mice
Fig. 1 Expression, puricication and identicication oc the cusion proteins Tat-TPI and TPI. a. Puricication oc two cusion proteins detected bv Protein Gel Electrophoresis. M: molecular weight marker, Lane 1: recombinant SjTat-TPI, Lane 2: recombinant SjTPI, Lane 3: the recombinant plasmid without puricication. b. Fusion proteins recognised bv His-Ab with Western blotting. Lane 1: recombinant SjTat-TPI, Lane 2: recombinant SjTPI. c. Fusion proteins recognised bv S. japonicum incected-mice serum with Western blotting. Lane 1: recombinant SjTat-TPI, Lane 2: recombinant SjTPI
Fig. 5 in SjTat-TPI facilitates adaptive T-cell responses and reduces hepatic pathology during Schistosoma japonicum infection in BALB/c mice
Fig. 5 Parasite burden and immune response were obsersed at 6 weeks acter S. japonicum incection in mice saccinated with T-TPI + IFA, TPI + IFA, IFA and PBS. a. Aserage number oc worms recosered. b. Aserage number oc eggs per gram (EPG) in the liser. c. Representatise granulomas with a single egg crom each group (100×). d. Aserage area oc single egg granulomas crom each group. e, f. Percentages oc CD3+CD4+IFN-γ+(Th1) and CD3+CD8+IFN-γ+(Tc1) gated crom CD3+ T cells analvsed bv FACS. Each bar represents the means ± SEM crom twelse mice per group. (*P <0.05; **P <0.01)
Fig. 4 T cell and antibodies responses acter three immunisations with T in SjTat-TPI facilitates adaptive T-cell responses and reduces hepatic pathology during Schistosoma japonicum infection in BALB/c mice
Fig. 4 T cell and antibodies responses acter three immunisations with T-TPI + IFA, TPI + IFA, IFA and PBS. a and b: Percentages oc CD3+CD4+IFN-γ+ (Th1) and CD3+CD8+IFN-γ+ (Tc1) gated crom CD3+ cells analvsed bv FACS. c. IgG, IgG1 and IgG2a lesels in mice sera were detected. Data are presented as the means ± SEM crom eight mice in each group. (*P <0.05; **P <0.01)
Figure 5 in Genetic difference between two Schistosoma japonicum isolates with contrasting cercarial shedding patterns revealed by whole genome sequencing
Figure 5. Gene ontology enrichment analysis of genes from genome regions with strong selective signals.
Figure 2 in Genetic difference between two Schistosoma japonicum isolates with contrasting cercarial shedding patterns revealed by whole genome sequencing
Figure 2. Box plots of diversity, Tajima's D of S. japonicum and their FST. (A) Nucleotide diversity estimated in 100-kb windows sliding in 10-kb steps throughout the genome. (B) Tajima's D estimated within a nonoverlapping 100-kb window throughout the genome. (C) pairwise FST computed in 100-kb windows sliding in 10-kb steps throughout the genome.
Figure 4 in Genetic difference between two Schistosoma japonicum isolates with contrasting cercarial shedding patterns revealed by whole genome sequencing
Figure 4. Distribution of p ratios (pST/pHX) and FST values calculated in 100-kb windows sliding in 10-kb steps throughout the genome. Data points colored red and blue were identified as selected regions in ST (red dots) and in HX (blue dots), respectively. These points correspond to the 5% left and right tails of the empirical p ratio distribution, where the p ratios are 0.092 and 1.643, respectively (vertical dashed lines), and the 5% right tail of the empirical FST distribution, where FST is 0.965 (horizontal dashed line).
Figure 3 in Genetic difference between two Schistosoma japonicum isolates with contrasting cercarial shedding patterns revealed by whole genome sequencing
Figure 3. Box plots of diversity and relationship of two sample groups. (A) Nucleotide diversity estimated in 100-kb windows sliding in 10-kb steps throughout the genome. (B) Tajima's D estimated within a nonoverlapping 100-kb window throughout the genome. (C) FST between computed in 100-kb windows sliding in 10-kb steps throughout the genome.
Fig. 3 in Exorchis sp. in the catfish Silurus asotus and Oncomelania hupensis in marshlands of Poyang Lake, China: A potential biological control tool for Schistosoma japonicum
Fig. 3. The prevalence of Exorchis sp. in S. asotus collected from the marshland of Poyang Lake from 2012 to 2016. (A) The adult S. asotus fish collected from Shi Li Hu were kept in plastic containers supplied with water. (B) The intestine of collected S. asotus fish were dissected in a glass Petri dish containing 0.75% saline solution. (C) The isolated Exorchis sp. adult trematodes isolated from the intestine of infected S. asotus. (D) The infection rate of Exorchis sp. in S. asotus is 56.82%, 75.56%, 67.09%, 63.81% and 72.18% from 2012 to 2016, respectively. (E) The intensity of infection of Exorchis sp. in S. asotus is 14.45, 15.24, 16.87, 14.18 and 12.22 per fish from 2012 to 2016, respectively. (F) The average infection rate of Exorchis sp. in S. asotus collected from the marshland of Poyang Lake from 2012 to 2016 was 65.79%. (G) The average intensity of infection of Exorchis sp. in S. asotus collected from the marshland of Poyang Lake from 2012 to 2016 was 14.21 per fish.
Fig. 2 in Exorchis sp. in the catfish Silurus asotus and Oncomelania hupensis in marshlands of Poyang Lake, China: A potential biological control tool for Schistosoma japonicum
Fig. 2. The infection rate of Exorchis sp. in O. hupensis collected from the marshland of Poyang Lake from 2012 to 2015. (A) The natural habitat of O. hupensis. (B) High grass region inhabited by large numbers of O. hupensis are shown, and the snails were marked with red arrows. (C) The cercaria of Exorchis sp. collected from O. hupensis. (D) The infection rate of Exorchis sp. in O. hupensis was 1.87%, 0.51%, 1.06% and 0.14% from 2012 to 2015, respectively. (E) The average infection rate of Exorchis sp. in O. hupensis collected from the marshland of Poyang Lake from 2012 to 2015 was 1.11%. (For interpretation of the references to colour in this figure legend, the reader is referred to the Web version of this article.)
Fig. 1 in Exorchis sp. in the catfish Silurus asotus and Oncomelania hupensis in marshlands of Poyang Lake, China: A potential biological control tool for Schistosoma japonicum
Fig. 1. Geographical location of the study area. Poyang Lake is located in the middle and lower reaches of the Yangtze River and in the north of Jiangxi Province. This study was conducted at Shi Li Hu (29◦ 25′ N, 116◦ 01′ E) in Xingzi county, Jiujiang City, Jiangxi Province, which is located on the western bank of the Poyang Lake in southern China.
Figure 1 in Genetic difference between two Schistosoma japonicum isolates with contrasting cercarial shedding patterns revealed by whole genome sequencing
Figure 1. Map of geographical locations of research sites.
Transcriptome analysis of females and males following knockdown serine/threonine protein phosphatase 1 catalytic subunit encoding genes(Sj-pp1c) in Schistosoma japonicum
GEO Series GSE108738. Schistosoma japonicum. 12 samples. Type: Expression profiling by high throughput sequencing.
Single-cell RNA sequencing profiles of schistosoma japonicum during reproductive development
GEO Series GSE293642. Schistosoma japonicum. 7 samples. Type: Expression profiling by high throughput sequencing.
Tissue specific profiling of the human pathogen Schistosoma japonicum
GEO Series GSE12706. Schistosoma japonicum; Schistosoma mansoni. 8 samples. Type: Expression profiling by array.
Altered levels of circulating miRNAs are associated with Schistosoma japonicum infection in mice
GEO Series GSE63135. Cytomegalovirus; JC polyomavirus; Betapolyomavirus macacae; Rattus norvegicus; Human alphaherpesvirus 2; Lymphocryptovirus; Merkel cell polyomavirus; Mus musculus; Human alphaherpesvirus 1; Human betaherpesvirus 5; Human immunodeficiency virus 1; Homo sapiens; Rhadinovirus; Murid gammaherpesvirus 4; Betapolyomavirus hominis. 4 samples. Type: Non-coding RNA profiling by array.
Single-cell RNA sequencing to dissect the immunological network of liver fibrosis in Schistosoma japonicum-infected mice
GEO Series GSE220286. Mus musculus. 2 samples. Type: Expression profiling by high throughput sequencing.
Comparison and analysis of gene expression between cattle and buffalo after infection with schistosoma japonicum
GEO Series GSE34021. Bubalus bubalis; Bos taurus. 12 samples. Type: Expression profiling by array.
Migrating Schistosoma japonicum schistosomula induce type-2 inflammation in the murine lung
GEO Series GSE27171. Mus musculus. 6 samples. Type: Expression profiling by array.
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