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Dataset results
13 results for “baroreflex sensitivity;”
Both hypoxia and hypobaria impair baroreflex sensitivity but through different mechanisms
<p><strong>database using hypobaric hypoxia, normobaric hypoxia and hypobaric normoxia on pilot trainee</strong></p>
Preoperative Heart Rate Variability and Baroreflex Sensitivity in ASO Patients During Various Sleep Stages
ClinicalTrials.gov study NCT00712946. IPD Sharing: Not stated. Countries: 1. Publications: 3.
Baroreflex Sensitivity Response to Exercise
ClinicalTrials.gov study NCT06709560. IPD Sharing: YES. Countries: 1. Publications: 5.
Heart Rate, Baroreflex Sensitivity and Cardiovascular Morbidity and Mortality in the Population
ClinicalTrials.gov study NCT00741728. IPD Sharing: Not stated. Countries: 1. Publications: 10.
Baroreflex Sensitivity in Patients Undergoing Ablation of Atrial Fibrillation
ClinicalTrials.gov study NCT04503122. IPD Sharing: UNDECIDED. Countries: 1. Publications: 0.
Perioperative Baroreflex Sensitivity
ClinicalTrials.gov study NCT02435875. IPD Sharing: Not stated. Countries: 1. Publications: 0.
Assessment of Spontaneous Baroreflex Sensitivity in Carriers of Implantable Cardioverter Defibrillators: Association With Disorders of the Ventricular Heart Rate. Case-control Study
ClinicalTrials.gov study NCT02930382. IPD Sharing: Not stated. Countries: 1. Publications: 0.
Effects of Electrical Baroreflex Stimulation on Sympathetic Activity, Renal Hemodynamics, and Insulin Sensitivity
ClinicalTrials.gov study NCT01355510. IPD Sharing: Not stated. Countries: 1. Publications: 0.
Prognostic Significance of the Baroreflex Sensitivity Changes After Acute Ischemic Stroke
ClinicalTrials.gov study NCT00422474. IPD Sharing: Not stated. Countries: 1. Publications: 0.
Pilot Study to Evaluate the Somnotouch Device to Quantify Spontaneous Baroreflex Sensitivity
ClinicalTrials.gov study NCT02650232. IPD Sharing: Not stated. Countries: 1. Publications: 0.
Improvement in Baroreflex Sensitivity in OSAS
ClinicalTrials.gov study NCT00284037. IPD Sharing: Not stated. Countries: 1. Publications: 0.
Change of Heart Rate Variability and Baroreflex Sensitivity After Ventral Cardiac Denervation
ClinicalTrials.gov study NCT00190112. IPD Sharing: Not stated. Countries: 0. Publications: 0.
Data set from the article Bari V, Vaini E, Pistuddi V, Fantinato A, Cairo B, De Maria B, Dalla Vecchia LA, Ranucci M, Porta A. Comparison of Causal and Non-causal Strategies for the Assessment of Baroreflex Sensitivity in Predicting Acute Kidney Dysfunction After Coronary Artery Bypass Grafting. Front Physiol. 2019 Oct 18;10:1319. doi: 10.3389/fphys.2019.01319. PMID: 31681021; PMCID: PMC6813722.
<p>Data set from the article Bari V, Vaini E, Pistuddi V, Fantinato A, Cairo B, De Maria B, Dalla Vecchia LA, Ranucci M, Porta A. Comparison of Causal and Non-causal Strategies for the Assessment of Baroreflex Sensitivity in Predicting Acute Kidney Dysfunction After Coronary Artery Bypass Grafting. Front Physiol. 2019 Oct 18;10:1319. doi: 10.3389/fphys.2019.01319. PMID: 31681021; PMCID: PMC6813722.</p> <p>This is the abstract:</p> <p>Coronary artery bypass graft (CABG) surgery may lead to postoperative complications such as the acute kidney dysfunction (AKD), identified as any post-intervention increase of serum creatinine level. Cardiovascular control reflexes like the baroreflex can play a role in the AKD development. The aim of this study is to test whether baroreflex sensitivity (BRS) estimates derived from non-causal and causal approaches applied to spontaneous systolic arterial pressure (SAP) and heart period (HP) fluctuations can help in identifying subjects at risk of developing AKD after CABG and which BRS estimates provide the best performance. Electrocardiogram and invasive arterial pressure were acquired from 129 subjects (67 ± 10 years, 112 males) before (PRE) and after (POST) general anesthesia induction with propofol and remifentanil. Subjects were divided into AKDs (<em>n</em> = 29) or no AKDs (noAKDs, <em>n</em> = 100) according to the AKD development after CABG. The non-causal approach assesses the transfer function from the HP-SAP cross-spectrum in the low frequency (LF, 0.04-0.15 Hz) band. BRS was estimated according to three strategies: (i) sampling of the transfer function gain at the maximum of the HP-SAP squared coherence in the LF band; (ii) averaging of the transfer function gain in the LF band; (iii) sampling of the transfer function gain at the weighted central frequency of the spectral components of the SAP series dropping in the LF band. The causal approach separated the two arms of cardiovascular control (i.e., from SAP to HP and <em>vice versa</em>) and accounted for the confounding influences of respiration via system identification and modeling techniques. The causal approach provided a direct estimate of the gain from SAP to HP by observing the HP response to a simulated SAP rise from the identified model structure. Results show that BRS was significantly lower in AKDs than noAKDs during POST regardless of the strategy adopted for its computation. Moreover, all the BRS estimates during POST remained associated with AKD even after correction for demographic and clinical factors. Non-causal and causal BRS estimates exhibited similar performances. Baroreflex impairment is associated with post-CABG AKD and both non-causal and causal methods can be exploited to improve risk stratification of AKD after CABG.</p>
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