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44 results for “mutation spectrum”

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zenodo48/100

Bedrock radioactivity influences the rate and spectrum of mutation - Orthologous genes

<p>Alignments of the 2490 orthologous genes used in the article &quot;Natural Bedrock radioactivity influences the rate and spectrum of mutation&quot; to estimate the mutational spectrum and synonymous substitution rate.</p> <p>To compute accurate synonymous substitution rate, we removed genes with short sequences (&lt;half of the alignment) and genes strongly supporting another phylogeny using ProfileNJ <a href="https://paperpile.com/c/Klqlpb/W5sS">(Noutahi et al. 2016)</a> with a bootstrap threshold of 90%, resulting in a subset of 769 genes listed in the file &quot;List_769_1-to-1_orthologs_EvolutionRate.txt&quot;.</p> <p>Transcriptome paired-end reads used to define these orthologous genes have been deposited to the European Nucleotide Archive and are available under the study ID PRJEB14193.</p> <p>Sequences were aligned with Prank<a href="https://paperpile.com/c/Klqlpb/pilh"> (L&ouml;ytynoja &amp; Goldman 2008)</a> using a codon model and sites ambiguously aligned were removed with Gblocks <a href="https://paperpile.com/c/Klqlpb/c5kb">(Castresana 2000)</a>.</p>

opencc-by-4.0Mar 2020View details →
dryad40/100

Data files associated with: Evolution of the mutation spectrum across a mammalian phylogeny

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publicSep 2023View details →
dryad36/100

Spectrum of mutational signatures in T-cell lymphoma reveals a key role for UV radiation in mycosis fungoides and Sezary syndrome

<p>T-cell non-Hodgkin's lymphomas (NHL) develop following transformation of tissue resident T-cells. We performed a meta-analysis of mutational catalogues derived from whole exome sequencing data from 403 patients with eight subtypes of T-cell NHL to identify mutational signatures and recurrent gene mutations associated with specific causal peaks within these signatures. Signature 1, indicative of age-related deamination, was prevalent across all T-cell NHL subtypes, reflecting the derivation of these malignancies from memory T-cell subsets. Adult T-cell leukemia-lymphoma (ATLL) was specifically associated with signature 17, which was found to strongly correlate with the IRF4 K59R mutation that is exclusive to ATLL. Signature 7, implicating UV exposure as a potential initiating factor was uniquely identified in cutaneous T-cell lymphoma, contributing 52% of the mutational burden in mycosis fungoides and 23% in Sezary syndrome.  Importantly this UV signature was observed in CD4+ T-cells isolated from blood suggesting extensive re-circulation of these T-cells through both skin and blood and strongly implicating a role for UV in the pathogenesis of cutaneous T-cell lymphoma.</p>

opencc-zeroDec 2019View details →
dryad36/100

A modified fluctuation assay reveals a natural mutator phenotype that drives mutation spectrum variation within Saccharomyces cerevisiae

<p>Although studies of <em>Saccharomyces cerevisiae</em> have provided many insights into mutagenesis and DNA repair, most of this work has focused on a few laboratory strains. Much less is known about the phenotypic effects of natural variation within <em>S. cerevisiae</em>'s DNA repair pathways. Here, we use natural polymorphisms to detect historical mutation spectrum differences among several wild and domesticated <em>S. cerevisiae</em> strains. To determine whether these differences are likely caused by genetic mutation rate modifiers, we use a modified fluctuation assay with a <em>CAN1</em> reporter to measure de novo mutation rates and spectra in 16 of the analyzed strains. We measure a 10-fold range of mutation rates and identify two strains with distinctive mutation spectra. These strains, known as AEQ and AAR, come from the panel's 'Mosaic beer' clade and share an enrichment for C &gt; A mutations that is also observed in rare variation segregating throughout the genomes of several Mosaic beer and Mixed origin strains. Both AEQ and AAR are haploid derivatives of the diploid natural isolate CBS 1782, whose rare polymorphisms are enriched for C &gt; A as well, suggesting that the underlying mutator allele is likely active in nature. We use a plasmid complementation test to show that AAR and AEQ share a mutator allele in the DNA repair gene <em>OGG1</em>, which excises 8-oxoguanine lesions that can cause C &gt; A mutations if left unrepaired.</p>

opencc-zeroDec 2021View details →
zenodo36/100

dataset related to article "EXPANDING THE GENETIC SPECTRUM OF PRIMARY FAMILIAL BRAIN CALCIFICATION DUE TO SLC2OA2 MUTATIONS: A CASE SERIES"

<p>Sanger sequences (.abi files) of all patients of our Fondazione Besta Cohort.</p>

opencc-by-4.0Mar 2022View details →
dryad36/100

Pedigree-based and phylogenetic methods support surprising patterns of mutation rate and spectrum in the gray mouse lemur

<p>Mutations are the raw material on which evolution acts, and knowledge of their frequency and genomic distribution is crucial for understanding how evolution operates at both long and short timescales. At present, the rate and spectrum of <i>de novo</i> mutations have been directly characterized in relatively few lineages. Our study provides the first direct mutation rate estimate for a strepsirrhine (i.e., the lemurs and lorises), which comprise nearly half of the primate clade. Using high-coverage linked-read sequencing for a focal quartet of gray mouse lemurs (<i>Microcebus</i> <i>murinus</i>), we estimated the mutation rate to be 1.52 × 10<sup>–8</sup> (95% credible interval: 1.28 × 10<sup>−8</sup> to 1.78 × 10<sup>−8</sup>) mutations/site/generation, a rate among the highest calculated for a mammal. Further, we found an unexpectedly low count of paternal mutations, and only a modest overrepresentation of mutations at CpG-sites. Despite the surprising nature of these results, we found both the rate and spectrum to be robust to the manipulation of a wide range of computational filtering criteria. We also sequenced a technical replicate to estimate a false negative and false positive rate for our data and show that any point estimate of a <i>de novo </i>mutation rate should be considered with a large degree of uncertainty. To validate these observations, we conducted an independent analysis of context-dependent substitution types for gray mouse lemur and five additional primate species for which <i>de novo</i> mutation rates have also been estimated. These comparisons revealed general consistency of the mutation spectrum between the pedigree-based and the substitution rate analyses for all species compared.</p>

opencc-zeroJul 2021View details →
dryad36/100

Spectrum of mutational signatures in T-cell lymphoma reveals a key role for UV radiation in mycosis fungoides and Sezary syndrome

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publicFeb 2020View details →
dryad36/100

Pedigree-based and phylogenetic methods support surprising patterns of mutation rate and spectrum in the gray mouse lemur

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publicJul 2021View details →
dryad36/100

A modified fluctuation assay reveals a natural mutator phenotype that drives mutation spectrum variation within Saccharomyces cerevisiae

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publicDec 2021View details →
dryad32/100

Data from: The genetic basis of a rare flower color polymorphism in Mimulus lewisii provides insight to the evolutionary mutation spectrum

A long-standing question in evolutionary biology asks whether the genetic changes contributing to phenotypic evolution are predictable. Here, we identify a genetic change associated with segregating variation in flower color within a population of Mimulus lewisii. To determine whether these types of changes are predictable, we combined this information with data from other species to investigate whether the spectrum of mutations affecting flower color transitions differs based on the evolutionary time-scale since divergence. We used classic genetic techniques, along with gene expression and population genetic approaches, to identify the putative, loss-of-function mutation that generates rare, white flowers instead of the common, pink color in M. lewisii. We found that a frameshift mutation in an anthocyanin pathway gene is responsible for the white-flowered polymorphism found in this population of M. lewisii. Comparison of our results with data from other species reveals a broader spectrum of flower color mutations segregating within populations relative to those that fix between populations. These results suggest that the genetic basis of fixed differences in flower color may be predictable, but that for segregating variation is not.

opencc-zeroDec 2012View details →
ClinicalTrials.gov32/100

Evaluation of Somatic Mutation Spectrum as Biomarker for Survival Outcome in Chinese CRC

ClinicalTrials.gov study NCT04228614. IPD Sharing: Not stated. Countries: 1. Publications: 6.

restrictedIPD-UNDECIDEDFeb 2026View details →
ClinicalTrials.gov32/100

Investigation of Tumour Spectrum of Germline Mutations in Breast and Ovarian Cancer Genes.

ClinicalTrials.gov study NCT03246841. IPD Sharing: YES. Countries: 1. Publications: 1.

controlledIPD-YESFeb 2026View details →
dryad32/100

Data from: Disentangling the influence of mutation and migration in clonal seagrasses using the Genetic Distance Spectrum for microsatellites

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publicFeb 2014View details →
dryad32/100

Data from: The genetic basis of a rare flower color polymorphism in Mimulus lewisii provides insight to the evolutionary mutation spectrum

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publicOct 2014View details →
dryad32/100

Data from: Limited role of generation time changes in driving the evolution of the mutation spectrum in humans

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publicMar 2023View details →
zenodo28/100

Data used in the article "Bedrock radioactivity influences the rate and spectrum of mutation"

<p>Data used in the article entitled &lsquo;Bedrock radioactivity influences the rate and spectrum of mutation&rsquo; by Nathana&euml;lle Saclier, Patrick Chardon, Florian Malard, Lara Konecny-Dupr&eacute;, David Eme, Arnaud Bellec, Vincent Breton, Laurent Duret, Tristan Lef&eacute;bure and Christophe J. Douady.</p> <p>Table 1: Description of sampling sites (species found and measurements of radioactivity).</p> <p>Table 2: Raw activity for each radionuclide in the selected sites</p> <p>Table 3: Computational methods to model the effective dose of radioactivity.</p> <p>Table 4: Corrected activity for each radionuclides.</p> <p>Table 5: Number of reads for each sequenced transcriptome</p> <p>Table 6: Mutation counts for each type of mutation, computed on all positions.</p> <p>Table 7: Mutation counts for each type of mutation, computed on third positions.</p> <p>Table 8: Relative frequency for each type of mutation, computed on all positions.</p> <p>Table 9: Relative frequency for each type of mutation, computed on third positions.</p> <p>&nbsp;</p> <p>&nbsp;</p> <p>&nbsp;</p>

opencc-by-4.0Jul 2019View details →
ClinicalTrials.gov28/100

Homologous Recombination Repair Pathway Gene Mutation Spectrum in Chinese Breast Cancer Patients

ClinicalTrials.gov study NCT05901025. IPD Sharing: NO. Countries: 0. Publications: 9.

closedIPD-NOFeb 2026View details →
dryad28/100

Data from: Neurologic phenotypes associated with COL4A1/2 mutations: expanding the spectrum of disease

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publicAug 2019View details →
geo24/100

Biallelic PAX5 mutations cause hypogammaglobulinemia,sensorimotor deficits and autism spectrum disorder

GEO Series GSE182463. Mus musculus. 10 samples. Type: Expression profiling by high throughput sequencing; Genome binding/occupancy profiling by high throughput sequencing.

openGEO-OpenJul 2022View details →
geo24/100

SHANK2 mutations associated with autism spectrum disorder cause hyperconnectivity of human neurons

GEO Series GSE122550. Homo sapiens. 16 samples. Type: Expression profiling by high throughput sequencing.

openGEO-OpenFeb 2019View details →

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Allen Brain Atlas

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allen-brain-atlas
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abode-home-cage
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DANDI Archive for NWB datasets

DANDI is a BRAIN Initiative archive for publishing and sharing neurophysiology data, including electrophysiology, optophysiology, and behavioral data packaged as NWB and related standards.

dandi-nwb
electrophysiologyopenPublished Dandiset metadata and archive endpoints are available through the production DANDI API.
Last verified 2026-04-30Open record

International Brain Laboratory public data

The International Brain Laboratory public data releases expose standardized mouse decision-making experiments, including Neuropixels recordings, widefield calcium imaging, behavior, and session metadata accessed through the ONE API.

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behavioral-neuroscienceopenPublic sessions can be searched and loaded from the IBL public data server through ONE.
Last verified 2026-04-29Open record

OpenNeuro

OpenNeuro is a free, open platform for sharing neuroimaging datasets, with public search, dataset pages, and download paths for web, S3, DataLad, and the OpenNeuro CLI.

openneuro
neuroscienceopenPublished datasets are available on demand over the internet.
Last verified 2026-04-29Open record