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5,794 results for “exposures”
Toxicology and dietary exposure of 3-monochloropropanediol and glycidol: a review
<p>Dataset for the article "Toxicology and dietary exposure of 3-monochloropropanediol, glycidol and their fatty acid esters: a review" to be published in the Journal of Food Quality</p>
Behavioral data associated with "Passive exposure to task-relevant stimuli enhances categorization learning"
<p>Behavioral data associated with Schmid et al. (2023) "<i>Passive exposure to task-relevant stimuli enhances categorization learning</i>", and example code for loading these data. See README.md for details.</p><p> </p>
Supporting Data for "Local exposure misclassification in national models: relationships with urban infrastructure and demographics"
<p><strong>Overview:</strong> This dataset accompanies the recent publication "Local exposure misclassification in national models: relationships with urban infrastructure and demographics" (DOI: 10.1038/s41370-023-00624-z). It provides essential data for replicating and extending the analysis conducted in the study. The script for the analysis is available at https://github.com/SEChambliss-AQ/LD-analysis. The dataset consists of four key files.</p> <p><strong>Files Included:</strong></p> <ol> <li> <p><strong>Gridded OSM and GSV Data (gridded_OSM_GSV.RDS):</strong></p> <ul> <li>This R object offers a 100mx100m grid covering select neighborhoods in the San Francisco Bay Area.</li> <li>Each grid cell includes average air pollution levels (Ultrafine Particle Count in thousand count per cubic meter; Nitrogen Dioxide in ppb) from mobile monitoring.</li> <li>The file also provides normalized z-scores of local density of urban infrastructure related to air pollutants based on OpenStreetMap (OSM) data.</li> <li>Further details can be found in the associated publication.</li> </ul> </li> <li> <p><strong>BartMachine Outputs (bartMachine R objects.zip):</strong></p> <ul> <li>A collection of R object outputs from the bartMachine package, an R-Java Bayesian Additive Regression Trees implementation.</li> <li>These objects, which can be regenerated using the provided script, are included to reduce computational requirements for future analyses.</li> <li>The script for generating these outputs is available at the above <a href="https://github.com/SEChambliss-AQ/LD-analysis">GitHub Repository</a>.</li> </ul> </li> <li> <p><strong>NO2 Predictions (CACES_criteria.csv):</strong></p> <ul> <li>Land Use Regression (LUR) data for Nitrogen Dioxide, provided by the Center for Air, Climate, and Energy Solutions.</li> <li>Methodologies for integrating these data with the gridded dataset are described in the publication and script.</li> </ul> </li> <li> <p><strong>UFP Predictions (CACES_UFP.csv):</strong></p> <ul> <li>Similar to the NO2 dataset, this file contains LUR data for Ultrafine Particle predictions.</li> <li>Methods for data integration are detailed in the publication and available script.</li> </ul> </li> </ol> <p><strong>Usage:</strong> These files are intended for researchers and analysts aiming to replicate or build upon the study's findings. They provide a source of data for exploring the complex interplay between air pollution, urban infrastructure, and demographic factors in urban environments. For detailed methodology and analysis, refer to the original publication and the accompanying GitHub repository.</p>
Dynamics of leaching of POPs and additives from plastic in a Procellariiform gastric model: Diet and polymer dependent effects and implications for long-term exposure
<p>Procellariiform seabirds are known to have high rates of plastic ingestion. We investigated the bioaccessibility of plastic-associated chemicals [plastic additives and sorbed persistent organic pollutants (POPs)] leached from plastic over time using an in vitro Procellariiform gastric model. High-density polyethylene (HDPE) and polyvinyl chloride (PVC), commonly ingested by Procellariiform seabirds, were manufactured with one additive [decabrominated diphenyl ether (PBDE-209) or bisphenol S (BPS)]. HDPE and PVC added with PBDE-209 were additionally incubated in salt water with 2,4,4'-trichloro-1,1'-biphenyl (PCB-28) and 2,2',3,4,4',5'-hexachlorobiphenyl (PCB-138) to simulate sorption of POPs on plastic in the marine environment. Our results indicate that the type of plastic (nature of polymer and additive), presence of food (i.e., lipids and proteins) and gastric secretions (i.e., pepsin) influence the leaching of chemicals in a seabird. In addition, 100% of the sorbed POPs were leached from the plastic within 100 hours, while only 2-5% of the additives were leached from the matrix within 100 hours, suggesting that the remaining 95% of the additives could continue to be leached. Overall, our study illustrates how plastic type, diet and plastic retention time can influence a Procellariform's exposure risk to plastic-associated chemicals.</p>
Replication data for global population profile of tropical cyclone exposure during 2002 and 2019
<p>Tropical cyclones have far-reaching impacts on livelihoods and population health that often persist years after the event. Characterizing the demographic and socioeconomic profile and the vulnerabilities of the exposed populations is essential to assess health and other risks associated with future tropical cyclone events. Estimates of exposure to tropical cyclones are often regional rather than global and do not consider population vulnerabilities. Here, we combine spatially resolved annual demographic estimates with tropical cyclone wind fields estimates to construct a global profile of the populations exposed to tropical cyclones between 2002 and 2019. We find that approximately 560 million people are exposed yearly and that the number of people exposed has increased across all cyclone intensities over the study period. The age distribution of those exposed has shifted away from children (under-5) and towards older people (over-60) in recent years compared to the early 2000s. Populations exposed to tropical cyclones are more socioeconomically deprived than those unexposed within the same country, and this relationship is more pronounced for people exposed to higher intensity storms. By characterizing the patterns and vulnerabilities of populations exposed to tropical cyclones, our results can help identify mitigation strategies and assess the global burden and future risks of tropical cyclones.</p>
Data from: Metabolomic profiles of acute and chronic ambient hydrogen sulfide exposure in a mouse model
<p>Hydrogen sulfide (H<sub>2</sub>S) is an environmental toxicant of health concern following acute or chronic human exposures. Male 6-8 week-old C57BL/6J mice were exposed by whole-body inhalation to 1000 ppm H<sub>2</sub>S for 45 min and euthanized at 5 min and 72 h for acute exposure. For subchronic study, mice were exposed to 5 ppm H<sub>2</sub>S 2 h/day, 5 days/week for 5 weeks. The brainstem was removed for metabolomic analysis. The metabolomics analyses consisted of three assays, (1) primary metabolism by GC-TOF MS, (2) biogenic amines (hydrophilic compounds) by HILIC-MS/MS and (3) lipidomics by RPLC-MS/MS. Metabolomics were performed in West Coast Metabolomics Center, University of California at Davis, CA, USA. 348, 311, and 565 known metabolites were detected and analyzed by primary metabolism, biogenic amines, and lipidomic metabolomics assays. 33, 19, and 46 metabolites were increased at 5 min and 72 h post acute H<sub>2</sub>S exposures and subchronic ambient H<sub>2</sub>S exposures, respectively, compared to room air control group. 22, 17, and 32 metabolites were decreased at 5 min and 72 h post acute H<sub>2</sub>S exposures and subchronic ambient H<sub>2</sub>S exposures, respectively, compared to room air control group. Acute H<sub>2</sub>S exposure decreased excitatory neurotransmitters aspartate and glutamate concentrations while the inhibitory neurotransmitter serotonin was increased. Glutamate and serotonin were also decreased after ambient H<sub>2</sub>S exposure. Branched-chain amino acids, fructose, and glucose were increased by acute H<sub>2</sub>S exposure. In ambient H<sub>2</sub>S exposure, glucose was decreased while MUFAs, PUFAs, inosine, and hypoxanthine were increased. Collectively, these results provide important mechanistic clues of acute and subchronic ambient H<sub>2</sub>S poisonings and show that H<sub>2</sub>S alters neurotransmission homeostasis.</p>
Coral community metabolism during year-long exposure to experimental elevated CO2 conditions
<p>The implications of ocean acidification are acute for calcifying organisms, notably tropical reef corals, for which accretion generally is depressed and dissolution enhanced at reduced seawater pH. We describe year-long experiments in which back reef and fore reef (17-m depth) communities from Moorea, French Polynesia, were incubated outdoors under pCO<sub>2</sub> regimes reflecting endpoints of representative concentration pathways (RCPs) expected by the end the century. Incubations were completed in 3–4 flumes (5.0 × 0.3 m, 500 L) in which seawater was refreshed and circulated at 0.1 m s<sup>-1</sup>, and the response of the communities was evaluated monthly by measurements of net community calcification (NCC) and net community photosynthesis (NCP). For both communities, NCC (but not NCP) was affected by treatments and time, with NCC declining with increasing pCO<sub>2</sub>, and for the fore reef, becoming negative (i.e., dissolution was occurring) at the highest pCO<sub>2</sub> (1067–1433 µatm, RCP8.5). There was scant evidence of community adjustment to reduce the negative effects of ocean acidification, and inhibition of NCC intensified in the back reef as the abundance of massive <em>Porites</em> spp. declined. These results highlight the risks of dissolution under ocean acidification for coral reefs and suggest these effects will be most acute in fore reef habitats. Without signs of amelioration of the negative effects of ocean acidification during year-long experiments, it is reasonable to expect that the future of coral reefs in acidic seas can be predicted from their current known susceptibility to ocean acidification.</p>
Behavioural ecology meets oncology: quantifying the recovery of animal behaviour to a transient exposure to a cancer risk factor
<p>Wildlife is increasingly exposed to sublethal transient cancer risk factors, including mutagenic substances, which activate their anti-cancer defences, promote tumourigenesis, and may negatively impact populations. Little is known about how exposure to cancer risk factors impacts the behaviour of wildlife. Here, we investigated the effects of a sublethal, short-term exposure to a carcinogen at environmentally relevant concentrations on the activity patterns of wild <em>Girardia tigrina</em> planaria during a two-phase experiment, consisting of a 7-day exposure to cadmium period followed by a 7-day recovery period. To comprehensively explore the effects of the exposure on activity patterns, we employed the double hierarchical generalized linear model framework which explicitly models residual intraindividual variability in addition to the mean and variance of the population. We found that exposed planaria were less active compared to unexposed individuals and were able to recover to pre-exposure activity levels albeit with a reduced variance in activity at the start of the recovery phase. Planaria showing high activity levels were less predictable with larger daily activity variations and higher residual variance. Thus, the shift in behavioural variability induced by an exposure to a cancer risk factor can be quantified using advanced tools from the field of behavioural ecology. This is required to understand how tumourous processes affect the ecology of species.</p>
Quantitative analysis of honey bee blood-ethanol levels following exposure to ethanol vapors
<p>The use of invertebrate models has allowed researchers to examine the mechanisms behind alcoholism and its effects with a cost-effective system. In that respect, the honey bee is an ideal model species to study the effects of ethanol (EtOH) due to the behavioural and physiological similarities of honey bees with humans when alcohol is consumed. Although both ingestion and inhalation methods are used to dose subjects in insect EtOH model systems, there is little literature on the use of the EtOH vapor-exposure method for experiments using honey bees. The experiment presented here provides baseline data for a dose EtOH-haemolymph response curve when using EtOH vapor-inhalation dosing with honey bees (<em>Apis mellifera</em>). Bees were exposed to EtOH vapors for 0, 1, 2.5, or 5 min, and haemolymph was collected 1 min post EtOH exposure. Haemolymph samples were analyzed using gas chromatography (GC) for haemolymph EtOH concentration. The ethanol-haemolymph level of the bees increased linearly with ethanol exposure time. The results provide a dosing guide for haemolymph EtOH level in the honey bee model ethanol-inhalation system, and thus makes the honey bee model more robust.</p>
Annex C - Human dietary exposure assessment
<p><span>Occurrence data on PCNs in food as considered for exposure assesssment, chronic dietary exposure to PCNs and the contribution of different food groups to the dietary exposure.</span></p>
Weather station temperature exposure metadata and exposure bias estimates
<p>Thermometer exposure metadata and monthly estimates of the exposure biases arising from the transition to Stevenson screens at mid-latitude weather stations in an extended version of the CRUTEM5 station database (which starts in 1781; Osborn et al., 2021).</p> <p>This dataset accompanies Wallis et al. (2024). Please see the attached readme file and Wallis et al. (2024) for further information about the dataset format and its creation.</p> <p>---</p> <p><strong>References</strong></p> <p>Osborn, T.J., Jones, P.D., Lister, D.H., Morice, C.P., Simpson, I.R., Winn, J., Hogan, E. & Harris, I.C. (2021) Land surface air temperature variations across the globe updated to 2019: the CRUTEM5 dataset. <em>Journal of Geophysical Research: Atmospheres</em>, <strong>126</strong>, e2019JD032352, https://doi.org/10.1029/2019JD032352</p> <p>Wallis, E.J., Osborn, T.J., Taylor, M., Jones, P.D., Joshi, M. & Hawkins, E. (2024) Quantifying exposure biases in early instrumental land surface air temperature observations. <em>International Journal of Climatology, </em>https://doi.org/10.1002/joc.8401</p>
Human health impact based on adult European consumers' dietary exposure to chemical contaminants and consumption of unprocessed red meat, processed meat, and legumes
<p>This dataset was generated within the PRISMA project (Marie Skłodowska-Curie Grant Agreement No. 101110615) for assessing the health impact for adult consumers from 25 European countries due to dietary exposure to chemical contaminants and consumption of food based on different dietary patterns.</p><p>The risk coefficients used to generate data are presented in Table S1.</p><p>File S2 presents the health impact based on the chronic dietary exposure to chemical contaminants for each country.</p><p>File S3 presents figures regarding the health impact for each country for adult consumers due to dietary exposure to chemical contaminants and consumption of food based on different dietary patterns.</p><p>File S4 presents the health impact based on the consumption of unprocessed red meat, processed meat, and legumes for each country.</p><p> </p>
Nitrogen dioxide exposure, health outcomes, and associated demographic disparities due to gas and propane combustion by US stoves
<p>Gas and propane stoves emit nitrogen dioxide (NO<sub>2</sub>) pollution indoors, but the exposures of different U.S. demographic groups are unknown. We estimate NO<sub>2 </sub>exposure and health consequences using emissions and concentration measurements from >100 homes, a room-specific indoor air quality model, epidemiological risk parameters, and statistical sampling of published housing characteristics and occupant behavior. Gas and propane stoves increase long-term NO<sub>2</sub> exposure by an estimated 4.0 ppbv on average across the United States, 75% of the World Health Organization (WHO)'s exposure guideline. This increased exposure may cause approximately 50 thousand cases of current pediatric asthma from long-term NO<sub>2</sub> exposure alone. Short-term NO<sub>2</sub> exposure from typical gas stove use frequently exceeds both World Health Organization and U.S. EPA benchmarks. Residents of residences <800 ft<sup>2</sup> in size incur 4 times more long-term NO<sub>2</sub> exposure than residents of residences >3000 ft<sup>2</sup> in size; American Indian/Alaska Native and Black and Hispanic/Latino households incur 60% and 20% more NO<sub>2</sub> exposure, respectively, than the national average.</p>
Social experiences shape song preference learning independent of developmental exposure to song
<p>Communication governs the formation and maintenance of social relationships. The interpretation of communication signals depends not only on the signal's content, but also on a receiver's individual experience. Experiences throughout life may interact to affect behavioral plasticity, such that a lack of developmental sensory exposure could constrain adult learning, while salient adult social experiences could remedy developmental deficits. We investigated how experiences impact the formation and direction of female auditory preferences in the zebra finch. Zebra finches form long-lasting pair bonds and females learn preferences for their mate's vocalizations. We found that after two weeks of cohabitation with a male, females formed pair bonds and learned to prefer their partner's song regardless of whether they were reared with ("normally-reared") or without ("song-naïve") developmental exposure to song. In contrast, females that heard but did not physically interact with a male did not prefer his song. In addition, previous work has found that song-naive females do not show species-typical preferences for courtship song. We found that cohabitation with a male ameliorated this difference in preference. Thus, courtship and pair bonding, but not acoustic-only interactions, strongly influence preference learning regardless of rearing experience, and may dynamically drive auditory plasticity for recognition and preference.</p>
Supplementary data: Bayesian multi-exposure image fusion for robust high dynamic range ptychography
<p>Accompanying supplementary data for the paper. To download this data automatically and use the software, please refer to the details in the README of the linked github repository. </p> <p><strong>Github URL: </strong><a href="https://github.com/microscopic-image-analysis/bayes-mef"><strong>https://github.com/microscopic-image-analysis/bayes-mef</strong></a></p>
Two mosquito salivary antigens demonstrate promise as biomarkers of recent exposure to P. falciparum-infected mosquito bites
<p>Measuring malaria transmission intensity using the traditional entomological inoculation rate is difficult. Antibody responses to mosquito salivary proteins such as SG6 have previously been used as biomarkers of exposure to <em>Anopheles</em> mosquito bites. Here, we investigate four mosquito salivary proteins as potential biomarkers of human exposure to mosquitoes infected with <em>P. falciparum</em>: mosGILT, SAMPSP1, AgSAP, and AgTRIO. We tested population-level human immune responses in longitudinal and cross-sectional plasma samples from subjects with known <em>P. falciparum</em> infection from low and moderate transmission areas in Senegal using a multiplexed magnetic bead-based assay. AgSAP and AgTRIO were the best indicators of recent exposure to infected mosquitoes, with antibody responses to AgSAP in a moderate endemic area, and to AgTRIO in both low and moderate endemic areas, significantly higher than healthy non-endemic control cohort (p-values = 0.0245, 0.0064, and <0.0001 respectively). No antibody responses significantly differed between the low and moderate transmission area, or between equivalent groups during and outside the malaria transmission seasons. For AgSAP and AgTRIO, reactivity peaked 2-4 weeks after clinical <em>P. falciparum</em> infection and declined 3 months after infection. Reactivity to both AgSAP and AgTRIO peaked after infection and did not differ seasonally nor between areas of low and moderate transmission, suggesting reactivity is due to exposure to infectious mosquitos or recent biting rather than general mosquito exposure. Kinetics suggest reactivity is relatively short-lived. AgSAP and AgTRIO are promising candidates to incorporate into multiplexed assays for serosurveillance of population-level changes in <em>P. falciparum</em>-infected mosquito exposure.</p>
Data for: Exposure and sensitivity of terrestrial vertebrates to biological invasions worldwide
<div> <p><em><strong>All details on the dataset are provided in the README. Details on the methods are in the Supplementary Material of the associated paper.</strong></em></p> <p>This dataset is the product of data complilation for the article <strong>Exposure and sensitivity of terrestrial vertebrates to biological invasions worldwide</strong>. It comprises, at a global scale for 110km grid cells:</p> <ul> <li>the exposure to the threat of biological invasions for 304 invasive alien species (3 exposure metrics with 3 alternative normalization methods)</li> <li>the sensitivity of birds, mammals, and reptiles to this threat (1 sensitivity metric with 3 alternative normalization methods)</li> <li>the data completeness associated with exposure and sensitivity, calculated with 2 components for each metric</li> </ul> <p>All the column details are provided in the "Metadata" sheet of each excel file. We also provide the shapefile of land cell units to allow for a full reproduction of the analyses and the maps.</p> <p>Additionally, we provide the sources to access the raw data used to derive the metrics for invasive alien species. </p> </div>
Developmental pyrethroid exposure disrupts molecular pathways for MAP kinase and circadian rhythms in mouse brain
<p><span>Neurodevelopmental disorders (NDDs) are a category of pervasive disorders of the developing nervous system with few or no recognized biomarkers. A significant portion of the risk for NDDs, including attention deficit hyperactivity disorder (ADHD), is contributed by the environment, and exposure to pyrethroid pesticides during pregnancy has been identified as a potential risk factor for NDD in the unborn child. We recently showed that low-dose developmental exposure to the pyrethroid pesticide deltamethrin in mice causes male-biased changes to ADHD- and NDD-relevant behaviors as well as the striatal dopamine system. Here, we used an integrated multiomics approach to determine the broadest possible set of biological changes in the mouse brain caused by developmental pyrethroid exposure (DPE). Using a litter-based, split-sample design, we exposed mouse dams during pregnancy and lactation to deltamethrin (3 mg/kg or vehicle every 3 days) at a concentration well below the EPA-determined benchmark dose used for regulatory guidance. We raised male offspring to adulthood, euthanized them, and pulverized and divided whole brain samples for split-sample transcriptomics, kinomics and multiomics integration. Transcriptome analysis revealed alterations to multiple canonical clock genes, and kinome analysis revealed changes in the activity of multiple kinases involved in synaptic plasticity, including the mitogen-activated protein (MAP) kinase ERK. Multiomics integration revealed a dysregulated protein-protein interaction network containing primary clusters for MAP kinase cascades, regulation of apoptosis, and synaptic function. These results demonstrate that DPE causes a multi-modal biophenotype in the brain relevant to ADHD and identifies new potential mechanisms of action.</span></p>
TG 91: Radiation Risk Inference at Low-Dose and Low-Dose Rate Exposure for Radiological Protection Purposes
<p>The detriment-adjusted nominal risk coefficients recommended by ICRP have been based, to a large extent, on data obtained from the atomic bomb survivors in Japan. ICRP introduced a Dose and Dose-Rate Effectiveness Factor (DDREF) of 2 to include in calculations of these risk coefficients for application at the low doses and low-dose rate exposures typical in radiological protection. The DDREF was included to account for the survivors’ single acute exposure, and because a linear quadratic instead of a linear risk to dose response was thought to be the most plausible biological model.</p> <p>This paper summarizes the recent work of ICRP Task Group 91 on this topic. Task Group activities have included reviews of the history of low-dose and low-dose-rate effects, various exposure scenarios and their doses and dose rates, radiation-induced effects at the molecular, cellular, animal, and human level, and biologically-based mechanistic models of carcinogenesis. The Task Group performed a meta-analysis of low-dose-rate epidemiological studies, and re-analyses of published results on radiation-induced effects among animal models and among Japanese atomic bomb survivors.</p>
Data: Responsiveness and habituation to repeated sound exposures and pulse trains in blue mussels
<p>Data abstract:</p> <p>Time series data on the valve gape behaviour of blue mussels (<em>Mytilus edulis</em>) that were exposured to sound treatments. Here, we provide the valve gape (time series data expressed in proportion open) of all mussels over the course of their trial and the timing of the sound exposures.</p> <p> </p> <p>Paper abstract:</p> <p>Anthropogenic sound has been shown to affect marine animals across taxa. However, bivalves and other invertebrates received limited attention and most studies across taxa focussed on immediate, rather than long-term, effects of sound. Most bivalves adopt a sessile or sedentary lifestyle and are therefore expected to be exposed to the same sounds for long periods or repeatedly. For this reason, bivalves are an especially relevant taxonomic group to study long-term effects of sound. In the current study, we examined whether blue mussels (<em>Mytilus edulis</em>) habituate to repeated sound exposures and whether they recover quicker from a single pulse exposure than from a pulse train. We equipped individual mussels with sensors to monitor valve gape and exposed them to repeated sound playback. We found that mussels responded to sound by partially closing their valves. This response was consistent and repeatable, but decayed over sequential exposures to the same sound stimulus, and was stronger again with exposure to a different sound. This pattern is clear evidence for acoustic habituation in a bivalve. Additionally, we found no differences in the initial response and recovery (time to return to baseline levels) between mussels that were exposed to single pulses and pulse trains. Our results therefore show that mussels are able to habituate to sound and suggest that mussels mostly respond to the onset of a pulse train. Future research is needed to determine whether mussels also habituate in situ to actual anthropogenic sound and whether a lack of a behavioural response also implies that other negative effects are also absent.</p> <p> </p> <p>Paper reference:</p> <p>Hubert, J., Booms, E., Witbaard, R., Slabbekoorn, H. (2022). Responsiveness and habituation to repeated sound exposures and pulse trains in blue mussels. <em>Journal of Experimental Marine Biology and Ecology</em>. 547, 151668. DOI: 10.1016/j.jembe.2021.151668</p>
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Allen Brain Atlas
Allen Brain Atlas is an Allen Institute collection of brain map atlases, datasets, APIs, and analysis tools covering mouse, human, and non-human primate brain resources.
Annotated Behaviour and Observability Dataset (ABODe)
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DANDI Archive for NWB datasets
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International Brain Laboratory public data
The International Brain Laboratory public data releases expose standardized mouse decision-making experiments, including Neuropixels recordings, widefield calcium imaging, behavior, and session metadata accessed through the ONE API.
OpenNeuro
OpenNeuro is a free, open platform for sharing neuroimaging datasets, with public search, dataset pages, and download paths for web, S3, DataLad, and the OpenNeuro CLI.