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Fig. 3 in Genetic diversity and population genetics of large lungworms (Dictyocaulus, Nematoda) in wild deer in Hungary

Fig. 3 Observed and simulated (expected) mismatch frequency distributions under a model of population expansion for D. eckerti overall (a), D. capreolus overall (b) and the eastern population of

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Fig. 1 in Genetic diversity and population genetics of large lungworms (Dictyocaulus, Nematoda) in wild deer in Hungary

Fig. 1 Map of collecting sites of Dictyocaulus in Hungary. Host species are indicated using different symbols (triangle: fallow deer; square: red deer; circle: roe deer), as are lungworm species (filled symbol: D. eckerti; empty symbol: D. capreolus; leaky symbol: D. sp. S-HU)

Fig. 5 in Uncovering Trypanosoma spp. diversity of wild mammals by the use of DNA from blood clots

Fig. 5. Map of the distribution of the Trypanosoma spp. identified in this study. Thirteen different trypanosomes species/genotypes/MOTUs were identified, in single and mixed infection, in the blood clot of bats, carnivores and marsupials. The trypanosomes are distributed in five Brazilian biomes (Amazon Forest, Atlantic Forest, Cerrado, Pampa, and Pantanal). Each colored circle indicates different trypanosome species/genotypes/MOTUs. Abbreviations: Brazilian states: AC, Acre; ES, Espírito Santo; GO, Goiás; MS, Mato Grosso do Sul; PB, Paraíba; RJ, Rio de Janeiro; RS, Rio Grande do Sul.

Fig. 3. T in Uncovering Trypanosoma spp. diversity of wild mammals by the use of DNA from blood clots

Fig. 3. T. cruzi clade phylogenetic tree based on 18S (SSU) gene. The tree shows ten different species and genotypes identified in the blood clot of Carnivora, Chiroptera, and Didelphimorphia: T. cruzi (DTUs TcI, TcII and TcIII), T. dionisii, T. rangeli, T. sp. Neobats 2 and 3, T. janseni, and two novel MOTUs (T. sp. DID and T. sp. Neobat 4). The tree was inferred with neighbor-joining. The numbers at the nodes correspond, respectively, to NJ, ML and BI support values for the main branches. The scale-bar shows the number of nucleotide substitutions per site. Trypanosoma lewisi and Trypanosoma microti were used as outgroups.

Fig. 2 in Uncovering Trypanosoma spp. diversity of wild mammals by the use of DNA from blood clots

Fig. 2. Trypanosoma spp. identified in the blood clot of Carnivora, Chiroptera and Didelphimorphia. Each color indicates a different trypanosome species, genotype or mixed infections. (For interpretation of the references to color in this figure legend, the reader is referred to the Web version of this article.)

Fig. 4 in Uncovering Trypanosoma spp. diversity of wild mammals by the use of DNA from blood clots

Fig. 4. Lizard/snake/rodent/marsupial clade phylogenetic tree based on 18S (SSU) gene. The tree shows the three different species from the lizard/snake/rodent/ marsupial clade identified in the blood clot of Chiroptera and Didelphimorphia: T. cascavelli, T. gennari, and T. lainsoni. Tree inferred with neighbor-joining. The numbers at the nodes correspond, respectively, to NJ, ML and BI support values for the main branches. The scale-bar shows the number of nucleotide substitutions per site. Trypanosoma serpentis was used as outgroup.

Fig. 1 in Uncovering Trypanosoma spp. diversity of wild mammals by the use of DNA from blood clots

Fig. 1. Methodological algorithm employed for the identification of trypanosomes in blood clot samples.

Fig. 3. A in Wild horse populations in south-east Australia have a high prevalence of Strongylus vulgaris and may act as a reservoir of infection for domestic horses

Fig. 3. A box and whisker plot (with individual data points) of the total strongyle egg counts across the different populations, showing the highest FECs were from samples from Bogong High Plains and Tin Mines, both alpine heathland habitats. Overall 89% of samples had FECs> 500 EPG, classed as 'high level shedders'.

Fig. 3. Predicted relationship between pre-treatment H in Age affects antibody levels and anthelmintic treatment efficacy in a wild rodent

Fig. 3. Predicted relationship between pre-treatment H. polygyrus burden and post-treatment H. polygyrus-specific IgG1 titre for A) juvenile mice, B) young adult mice and C) adult mice. ELW was split into equal thirds to group mice into juvenile (9.2 μg-15.8 μg), young adult (15.8 μg-22.4 μg) or adult mice (22.4 μg- 29.0 μg).

Fig. 1 in Age affects antibody levels and anthelmintic treatment efficacy in a wild rodent

Fig. 1. Dynamics of A) H. polygyrus burden, B) E. hungaryensis burden, C) total faecal IgA and D) H. polygyrus-specific IgG1 over the experimental period as predicted by minimal GAMM models. Red lines represent model estimates for animals treated with anthelmintic, black lines represent model estimates for control animals. Shaded areas represent estimated standard errors.

Fig. 2 in Age affects antibody levels and anthelmintic treatment efficacy in a wild rodent

Fig. 2. Relationship between mouse age (scaled ELW), treatment and post-treatment H. polygyrus burden as predicted by GLM model. The red line represents model estimates for treated animals, black lines represent model estimates for control animals. Shaded areas represent estimated standard errors. Points represent raw data.

Fig. 2 in Wild horse populations in south-east Australia have a high prevalence of Strongylus vulgaris and may act as a reservoir of infection for domestic horses

Fig. 2. Microscopic view of the different eggs. A = Anoplocephala spp. eggs, S <90 = strongyle eggs <90 μm length, S> 90 = strongyle eggs ≥90 μm length, P = Parascaris spp. eggs.

Fig. 6 in Molecular identification of two Eimeria species, E. uekii and E. raichoi as type B, in wild Japanese rock ptarmigans, Lagopus muta japonica

Fig. 6. Histopathologic sections of ceca which were obtained from dead chicks infected with Eimeria spp. Arrowhead indicates macrogametocytes with a prominent wall-forming body (A), and arrows indicate zygotes or early oocysts, which are surrounded by an oocyst wall (A and B). Pathological lesions could not be observed because of severe degradation after death. Scale bars indicate 20 μm.

Fig. 3 in Molecular identification of two Eimeria species, E. uekii and E. raichoi as type B, in wild Japanese rock ptarmigans, Lagopus muta japonica

Fig. 3. Phylogram of E. uekii, type B, other Eimeria spp., and related parasites (Cyclospora spp.) inferred by the neighbor-joining method using partial 18S rRNA gene sequences. Accession numbers and derived hosts are shown in parentheses. Scale bar represents substitutions per nucleotide, and bootstrap values are indicated (> 1000). Cystoisospora spp. are used as an outgroup taxon.

Fig. 4 in Molecular identification of two Eimeria species, E. uekii and E. raichoi as type B, in wild Japanese rock ptarmigans, Lagopus muta japonica

Fig. 4. Phylogram of E. uekii, type B, and other related Eimeria spp. inferred by the neighbor-joining method using partial mitochondrial cytochrome c oxidase subunit I gene sequences. Accession numbers and derived hosts are shown in parentheses. Scale bar represents substitutions per nucleotide, and bootstrap values are indicated (> 1000). Toxoplasma gondii is used as an outgroup taxon.

Fig. 2 in Molecular identification of two Eimeria species, E. uekii and E. raichoi as type B, in wild Japanese rock ptarmigans, Lagopus muta japonica

Fig. 2. Eimeria oocysts detected in the feces of Japanese rock ptarmigans. (A) E. uekii and (B) type B. Scale bars indicate 10 μm.

Fig. 1 in Molecular identification of two Eimeria species, E. uekii and E. raichoi as type B, in wild Japanese rock ptarmigans, Lagopus muta japonica

Fig. 1. Location of three sampled areas in Japan, Mt. Tateyama (36̊35′N, 137̊36′E), Norikuradake (36̊6′N, 137̊33′E), and Kitadake (35̊40′N, 138̊14′E) (triangle boxes 1–3).

Fig. 5 in Molecular identification of two Eimeria species, E. uekii and E. raichoi as type B, in wild Japanese rock ptarmigans, Lagopus muta japonica

Fig. 5. Composite line drawing of oocyst of Eimeria raichoi n. sp (previously referred as type B). Scale bars indicate 10 μm.

Fig. 2 in The wild world of Guinea Worms: A review of the genus Dracunculus in wildlife

Fig. 2. Dracunculus insignis infection in definitive host raccoon (Procyon lotor) and intermediate host (Cyclopoid copepod): (A) lesion from emergence of gravid female D. insignis (marked by arrow); (B) in situ photograph of patent D. insignis infection in forelimb of raccoon; (C) first-stage larvae (L1) of D. insignis (0.596–0.857 mm); (D) thirdstage larvae (L3) of D. insignis (0.434–0.605 mm) with trilobed tail, a distinguishing morphologic characteristic; (E) a copepod infected with D. insignis (L3s marked with arrow).

Fig. 4 in Maintenance of Trypanosoma cruzi, T. evansi and Leishmania spp. by domestic dogs and wild mammals in a rural settlement in Brazil-Bolivian border

Fig. 4. Path analysis on the influences of contact and feeding on wild mammals in relation to infections of dogs surveyed at Urucum settlement, Corumbá, Mato Grosso do Sul, Brazil in 2015.