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646 results for “Pseudomonas aeruginosa”

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zenodo36/100

Efectos de las mezclas del fármaco cloperidona y los antibióticos gentamicina/azitromicina sobre la formación de biopelícula de Pseudomonas aeruginosa

<p>Pseudomonas aeruginosa es una bacteria oportunista en infecciones nosocomiales, que tiene la capacidad de reducir la actividad de los antibi&oacute;ticos de amplio espectro debido a la formaci&oacute;n de biopel&iacute;cula, llevando a que sea categorizada como una bacteria de prioridad cr&iacute;tica a nivel mundial. La biopel&iacute;cula est&aacute; formada por comunidades de bacterias dentro de una matriz extracelular compuesta de prote&iacute;nas, polisac&aacute;ridos y ADN extracelular que le confieren protecci&oacute;n, por ello se ha considerado como un blanco potencial para el control de P. aeruginosa. En este sentido, el objetivo del presente estudio fue evaluar el efecto de Cloperidona, Gentamicina y Azitromicina, as&iacute; como de las mezclas binarias de Cloperidona/Gentamicina y Cloperidona/Azitromicina sobre la susceptibilidad y la formaci&oacute;n de la biopel&iacute;cula de P. aeruginosa (PAO1) ATCC BAA-47. Para lo cual, se determin&oacute; las concentraciones m&iacute;nimas inhibitorias (CMI) por la t&eacute;cnica de microdiluci&oacute;n para los compuestos y las dos mezclas, as&iacute; como su efecto en la formaci&oacute;n de biopel&iacute;cula de P. aeruginosa por la t&eacute;cnica de tinci&oacute;n con cristal violeta. A partir de los resultados se determin&oacute; que las CMI para inhibir el crecimiento de P aeruginosa de los antibi&oacute;ticos gentamicina y azitromicina fueron de 1 y 31.25 &micro;g/mL, respectivamente, mientras que Cloperidona no mostr&oacute; efectos inhibitorios sobre el crecimiento a la m&aacute;xima concentraci&oacute;n evaluada de 100 &micro;g/mL. Adicionalmente, para los compuestos, el mayor efecto en la formaci&oacute;n de biopel&iacute;cula a la menor concentraci&oacute;n fue para Cloperidona a 0.39 &micro;g/mL, gentamicina a 0.5 &micro;g/mL y azitromicina a 1.95 &micro;g/mL, con porcentajes de inhibici&oacute;n de 56%, 50% y 88%, respectivamente. Para las dos mezclas Cloperidona/Azitromicina y Cloperidona/Gentamicina (concentraciones sub-MIC) ninguna present&oacute; mayores efectos inhibitorios si se compara con el efecto de la gentamicina cuando fue evaluada de manera independiente. Sin embargo, las mezclas Cloperidona/Gentamicina (0.39 &micro;g/mL/ 0.03 &micro;g/mL) y Cloperidona/Azitromicina (0.39 &micro;g/mL/ 0.97 &micro;g/mL) fueron las que mostraron el mayor efecto inhibitorio sobre la biopel&iacute;cula con porcentajes de 77% y 88%, adem&aacute;s de presentar efectos sin&eacute;rgicos ya que fue posible bajar la concentraci&oacute;n de los antibi&oacute;ticos en ambos casos para tener efectos inhibitorios en la formaci&oacute;n de biopel&iacute;cula. Estos resultados permiten concluir que las mezclas de los antibi&oacute;ticos con Cloperidona pueden ser utilizados para la reducci&oacute;n en la formaci&oacute;n de la biopel&iacute;cula, uno de los principales mecanismos que utiliza la bacteria para reducir la acciones de los antibi&oacute;ticos y as&iacute; ser considerado como estrategia para frenar la resistencia microbiana.</p>

opencc-by-4.0Sep 2023View details →
ClinicalTrials.gov36/100

Study to Evaluate Arikayce™ in CF Patients With Chronic Pseudomonas Aeruginosa Infections

ClinicalTrials.gov study NCT01315678. IPD Sharing: Not stated. Countries: 18. Publications: 1.

restrictedIPD-UNDECIDEDFeb 2026View details →
ClinicalTrials.gov36/100

Dose Escalation Study of KB001 in Cystic Fibrosis Patients Infected With Pseudomonas Aeruginosa

ClinicalTrials.gov study NCT00638365. IPD Sharing: Not stated. Countries: 1. Publications: 1.

restrictedIPD-UNDECIDEDFeb 2026View details →
ClinicalTrials.gov36/100

Aztreonam for Inhalation Solution vs Tobramycin Inhalation Solution in Patients With Cystic Fibrosis & Pseudomonas Aeruginosa

ClinicalTrials.gov study NCT00757237. IPD Sharing: Not stated. Countries: 13. Publications: 1.

restrictedIPD-UNDECIDEDFeb 2026View details →
ClinicalTrials.gov36/100

Effort to Prevent Nosocomial Pneumonia Caused by Pseudomonas Aeruginosa in Mechanically Ventilated Subjects

ClinicalTrials.gov study NCT02696902. IPD Sharing: Not stated. Countries: 14. Publications: 1.

restrictedIPD-UNDECIDEDFeb 2026View details →
ClinicalTrials.gov36/100

Extension Study of Liposomal Amikacin for Inhalation in Cystic Fibrosis (CF) Patients With Chronic Pseudomonas Aeruginosa (Pa) Infection

ClinicalTrials.gov study NCT01316276. IPD Sharing: Not stated. Countries: 17. Publications: 1.

restrictedIPD-UNDECIDEDFeb 2026View details →
ClinicalTrials.gov36/100

Phase 3 Study of Aztreonam for Inhalation Solution (AZLI) in a Continuous Alternating Therapy Regimen for the Treatment of Chronic Pseudomonas Aeruginosa Infection in Patients With CF

ClinicalTrials.gov study NCT01641822. IPD Sharing: Not stated. Countries: 1. Publications: 1.

restrictedIPD-UNDECIDEDFeb 2026View details →
ClinicalTrials.gov36/100

OPTIMIZing Treatment for Early Pseudomonas Aeruginosa Infection in Cystic Fibrosis

ClinicalTrials.gov study NCT02054156. IPD Sharing: Not stated. Countries: 1. Publications: 1.

restrictedIPD-UNDECIDEDFeb 2026View details →
ClinicalTrials.gov36/100

Safety/Tolerability Study of Arikayce™ in Cystic Fibrosis Patients With Chronic Infection Due to Pseudomonas Aeruginosa

ClinicalTrials.gov study NCT00558844. IPD Sharing: Not stated. Countries: 1. Publications: 2.

restrictedIPD-UNDECIDEDFeb 2026View details →
ClinicalTrials.gov36/100

Ceftolozane-Tazobactam for Directed Treatment of Pseudomonas Aeruginosa Bacteremia and Pneumonia in Patients With Hematological Malignancies and Hematopoietic Stem Cell Transplantation

ClinicalTrials.gov study NCT04673175. IPD Sharing: NO. Countries: 1. Publications: 7.

closedIPD-NOFeb 2026View details →
ClinicalTrials.gov36/100

Study of Aztreonam for Inhalation in Children With Cystic Fibrosis and New Infection of the Airways by Pseudomonas Aeruginosa Bacteria

ClinicalTrials.gov study NCT03219164. IPD Sharing: YES. Countries: 12. Publications: 1.

controlledIPD-YESFeb 2026View details →
dryad36/100

Data from: Life in the cystic fibrosis upper respiratory tract influences competitive ability of the opportunistic pathogen Pseudomonas aeruginosa

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publicAug 2018View details →
dryad36/100

Aztreonam-induced filamentation in Pseudomonas aeruginosa

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publicJul 2024View details →
dryad36/100

Ecology drives the evolution of diverse social strategies in Pseudomonas aeruginosa

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publicAug 2021View details →
dryad36/100

Heterogenous susceptibility to R-pyocins in populations of Pseudomonas aeruginosa sourced from cystic fibrosis lungs

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publicApr 2021View details →
dryad36/100

Experimental data for Pseudomonas aeruginosa from experimental evolution under different bottleneck sizes and antibiotic selection pressures

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publicJul 2021View details →
dryad36/100

DNA Methyltransferase regulates nitric oxide homeostasis and virulence in a chronically adapted Pseudomonas aeruginosa strain

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publicAug 2022View details →
dryad36/100

Personalized inhaled bacteriophage therapy for treatment of multidrug-resistant Pseudomonas aeruginosa in cystic fibrosis

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publicFeb 2025View details →
dryad36/100

High prevalence of lipopolysaccharide mutants and R2-Pyocin susceptible variants in Pseudomonas aeruginosa populations sourced from cystic fibrosis lung infections

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publicSep 2023View details →
dryad32/100

The evolution of virulence in Pseudomonas aeruginosa during chronic wound infection

<p>Opportunistic pathogens are associated with a number of chronic human infections, yet the evolution of virulence in these organisms during chronic infection remains poorly understood. Here, we tested the evolution of virulence in the human opportunistic pathogen Pseudomonas aeruginosa in a murine chronic wound model using a two-part serial passage and sepsis experiment, and found that virulence evolved in different directions in each line of evolution. We also assessed P. aeruginosa adaptation to a chronic wound after 42 days of evolution and found that morphological diversity in our evolved populations was limited compared to that previously described in cystic fibrosis (CF) infections. Using whole-genome sequencing, we found that genes previously implicated in P. aeruginosa pathogenesis (lasR, pilR, fleQ, rpoN and pvcA), contained mutations during the course of evolution in wounds, with selection occurring in parallel across all lines of evolution. Our findings highlight that (i) P. aeruginosa heterogeneity may be less extensive in chronic wounds than in CF lungs; (ii) genes involved in P. aeruginosa pathogenesis acquire mutations during chronic wound infection; (iii) similar genetic adaptations are employed by P. aeruginosa across multiple infection environments and (iv) current models of virulence may not adequately explain the diverging evolutionary trajectories observed in an opportunistic pathogen during chronic wound infection.</p>

opencc-zeroAug 2020View details →

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International Brain Laboratory public data

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Last verified 2026-04-29Open record