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117
datasets available to search
ShareScore release 0.7.1
Dataset results
117 results for “Myodes”
Comparison of Genome-Wide Binding of MyoD in Normal Human Myogenic Cells and Rhabdomyosarcomas Identifies Regional and Local Suppression of Promyogenic Transcription Factors (expression)
GEO Series GSE50411. Homo sapiens. 6 samples. Type: Expression profiling by array.
Distinct activities of Myf5 and MyoD indicate sequential roles in skeletal muscle lineage specification and differentiation (ChIP-Seq)
GEO Series GSE75370. Mus musculus. 10 samples. Type: Genome binding/occupancy profiling by high throughput sequencing.
A KAP1 phosphorylation switch controls MyoD function during skeletal muscle differentiation (ChIP-seq)
GEO Series GSE62660. Mus musculus. 3 samples. Type: Genome binding/occupancy profiling by high throughput sequencing.
MYOD Gene Expression Regulation during Myogenic Conversion of Fibroblasts
GEO Series GSE98198. Homo sapiens. 8 samples. Type: Expression profiling by high throughput sequencing; Genome binding/occupancy profiling by high throughput sequencing.
Myod and Myog ChIP-seq in quiescent and activated muscle stem cells (mouse)
GEO Series GSE185104. Mus musculus. 14 samples. Type: Genome binding/occupancy profiling by high throughput sequencing.
A KAP1 phosphorylation switch controls MyoD function during skeletal muscle differentiation
GEO Series GSE62664. Mus musculus. 9 samples. Type: Expression profiling by high throughput sequencing; Genome binding/occupancy profiling by high throughput sequencing.
Epigenetic analysis reveals the repressive function of MyoD during myogenic differentiation
GEO Series GSE63716. Mus musculus. 8 samples. Type: Genome binding/occupancy profiling by high throughput sequencing.
Conversion of MyoD to a neurogenic factor: binding site specificity determines lineage [RNA-seq]
GEO Series GSE64622. Mus musculus. 8 samples. Type: Expression profiling by high throughput sequencing.
MYOD Remodeling of the Genome Architecture during Myogenic Conversion of Somatic Cells
GEO Series GSE98509. Homo sapiens. 16 samples. Type: Expression profiling by high throughput sequencing; Genome binding/occupancy profiling by high throughput sequencing; Third-party reanalysis; Other.
MyoD induced enhancer RNA interacts with hnRNPL protein via CAAA motif to activate target gene transcription during myogenic differentiation [RNA-Seq]
GEO Series GSE114658. Mus musculus. 4 samples. Type: Expression profiling by high throughput sequencing.
Data from: TBP/TFIID-dependent activation of MyoD target genes in skeletal muscle cells
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Transcription factor Runx1 cooperates with MyoD and c-Jun to regulate the balance of myoblast proliferation/differentiation [ChIP-seq]
GEO Series GSE56077. Mus musculus. 16 samples. Type: Genome binding/occupancy profiling by high throughput sequencing.
MyoD and FoxO3 mediated hotspot interaction orchestrates super-enhancer activity during myogenic differentiation (Foxo3 ChIP-seq)
GEO Series GSE99207. Mus musculus. 2 samples. Type: Genome binding/occupancy profiling by high throughput sequencing.
Snail regulates MyoD binding-site occupancy to direct enhancer switching and differentiation-specific transcription in myogenesis
GEO Series GSE24904. Mus musculus. 27 samples. Type: Expression profiling by array; Genome binding/occupancy profiling by high throughput sequencing.
MyoD and FoxO3 mediated hotspot interaction orchestrates super-enhancer activity during myogenic differentiation
GEO Series GSE93916. Mus musculus. 13 samples. Type: Genome binding/occupancy profiling by high throughput sequencing.
MyoD induced enhancer RNA interacts with hnRNPL protein via CAAA motif to activate target gene transcription during myogenic differentiation [CLIP-Seq]
GEO Series GSE114656. Mus musculus. 1 samples. Type: Other.
Liaison between SNAI2 and MYOD enhances oncogenesis and suppresses differentiation in Fusion-Negative Rhabdomyosarcoma
GEO Series GSE137168. Homo sapiens. 22 samples. Type: Genome binding/occupancy profiling by high throughput sequencing; Expression profiling by high throughput sequencing; Other.
A MYOD-SKP2 axis boosts tumorigenesis in fusion negative rhabdomyosarcoma by preventing differentiation through p57Kip2 targeting [ChIP-seq]
GEO Series GSE241279. Homo sapiens. 4 samples. Type: Genome binding/occupancy profiling by high throughput sequencing.
CRISPR/Cas9/AAV9-sgRNA Mediated In Vivo Genome Editing Reveals the Indispensability of Myc During Muscle Stem Cells Activation by Remodeling the 3D Chromatin [DEEP-seq(MyoD&Myc&Bcl6&Pknox2)]
GEO Series GSE157871. Mus musculus. 6 samples. Type: Other.
Identification of the unique gene expression profile in MyoD-tdTomato Low- and MyoD-tdTomato High-expressing muscle stem cells (MuSCs) populations
GEO Series GSE211755. Mus musculus. 9 samples. Type: Expression profiling by high throughput sequencing.
ScienceDex guides
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These curated guides explain access requirements, typical timelines, costs, and reuse considerations for widely used research datasets.
Allen Brain Atlas
Allen Brain Atlas is an Allen Institute collection of brain map atlases, datasets, APIs, and analysis tools covering mouse, human, and non-human primate brain resources.
Annotated Behaviour and Observability Dataset (ABODe)
ABODe is a University of Edinburgh DataShare dataset for behavior classification in group-housed mice using home-cage video, identities, bounding boxes, ground-plate positions, and annotator labels.
DANDI Archive for NWB datasets
DANDI is a BRAIN Initiative archive for publishing and sharing neurophysiology data, including electrophysiology, optophysiology, and behavioral data packaged as NWB and related standards.
International Brain Laboratory public data
The International Brain Laboratory public data releases expose standardized mouse decision-making experiments, including Neuropixels recordings, widefield calcium imaging, behavior, and session metadata accessed through the ONE API.
OpenNeuro
OpenNeuro is a free, open platform for sharing neuroimaging datasets, with public search, dataset pages, and download paths for web, S3, DataLad, and the OpenNeuro CLI.