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Figure 5 in Investigation of protective effects of lithium borate on spermatogenesis and testes histopathology against cadmium-induced acute toxicity in rats

Figure 5. Testes tissue, control and LTB groups, negative COX-2 expression (AC), Cd group, severe COX-2 expressions in damaged tubules and intertubular intervals (B), LTB + Cd group, mild COX-2 expression in interstitial tissue (arrowheads) (D), IHC - P, Bar: 20µm.

Figure 4 in Investigation of protective effects of lithium borate on spermatogenesis and testes histopathology against cadmium-induced acute toxicity in rats

Figure 4. Testes tissue, Control and LTB groups, negative 8-OHdG expression (AC), Cd group, severe cytoplasmic 8-OHdG expressions in spermatocytes and spermatogonium in damaged tubules (arrowheads) (B), LTB + Cd group, mild cytoplasmic 8-OHdG expression in spermatocytes (arrowheads) (D), IHC - P, Bar: 20 µm.

Figure 3. Negative Caspase-3 in Investigation of protective effects of lithium borate on spermatogenesis and testes histopathology against cadmium-induced acute toxicity in rats

Figure 3. Negative Caspase-3 expressions in testes tissues of control and LTB groups (A and C), severe Caspase-3 expression in spermatocytes of Cd group (arrowheads) (B), mild Caspase- 3 expression in spermatocytes (arrowheads) of LTB + Cd group (D), IHC - P, Bar: 20 µm.

Figure 1 in Investigation of protective effects of lithium borate on spermatogenesis and testes histopathology against cadmium-induced acute toxicity in rats

Figure 1. Testicular tissue, normal anatomical appearance (A), oedema, hyperaemia, congestion and haemorrhage (B), normal anatomical appearance (C), moderate oedematous and mild hyperaemic (D).

Figure 5 in Determination of oxidative, genotoxic, and histopathologic effects of metal pollution on the fish fauna inhabiting Karasu River, Turkey

Figure 5. Pathological damages in A. mossulensis A) separation from the granular layer (black arrow), hyperemia (orange arrows) and infiltration (red arrow) in the cerebellum, B) separation in pia mater (black arrow), congestion and damage in vein (orange arrow), degeneration in periventricular layer (green arrow) and axonopathy (circle) C) dilatation (orange arrow) and vacuolization (yellow arrows) in the cortex H & E.

Figure 4 in Determination of oxidative, genotoxic, and histopathologic effects of metal pollution on the fish fauna inhabiting Karasu River, Turkey

Figure 4. Pathological damages in brain of C. capoeta A) vascular dilatation (orange arrow), degeneration (red arrow) and dilatation (black arrow), B) vacuolization (yellow arrows) and infiltration (blue arrow) in the Purkinje layer, C) vascular dilatation with congestion (orange arrow) H & E.

Figure 1 in Determination of oxidative, genotoxic, and histopathologic effects of metal pollution on the fish fauna inhabiting Karasu River, Turkey

Figure 1. Sampling stations; Station 1: Dumlu, Station 2: Ilıca, Station 3: Aşkale I, Station 4: Aşkale II.

Figure 2 in Determination of oxidative, genotoxic, and histopathologic effects of metal pollution on the fish fauna inhabiting Karasu River, Turkey

Figure 2. The erythrocytic nuclear abnormalities determined in the three fish species A) micronucleus (arrow) in C. capoeta, B) notched nucleus (arrow) in C. capoeta, C) binucleated nucleus (arrow) in A. mossulensis, D) kidney shaped nucleus (black arrows) and bud nucleus (red arrow) in A. mossulensis, E) lobed nucleus (arrow) in S. cephalus, F) Bud nucleus (arrow) in S. cephalus Giemsa.

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Fig. 2. The PCR products identified within the 18S in Usefulness of PCR-RFLP of 18S rRNA gene for rapid post-mortem diagnostics of highly pathogenic Eimeria spp. (Apicomplexa: Eimeriidae) of European bison, Bison bonasus L. with histopathological correlation

Fig. 2. The PCR products identified within the 18S rRNA of Eimeria bovis following digestion with two restriction endonucleases: AluI recognising AG∧CT and Hin1II recognising CATG∧. M1: GeneRuler 100 bp Plus DNA Ladder (Thermo Fisher Scientific); M2: GeneRuler 50bp DNA Ladder (Thermo Fisher Scientific); lane 1: European bison colon wall tissue; lane 2: European bison colon wall tissue after digestion; lane 3: E. bovis oocysts of European bison; lane 4: E. bovis oocysts of European bison after digestion.

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Fig. 3 in Usefulness of PCR-RFLP of 18S rRNA gene for rapid post-mortem diagnostics of highly pathogenic Eimeria spp. (Apicomplexa: Eimeriidae) of European bison, Bison bonasus L. with histopathological correlation

Fig. 3. The virtual double digestion of the 18S rRNA gene of eimerians infecting the large intestine of the European bison with the restriction enzymes Mval (BstNI) recognising CC∧WGG, and KpnI recognising GGTAC∧C, simulated with SnapGene version 5.0.6 (GSL Biotech LLC); M: GeneRuler 50 bp DNA Ladder (Thermo Fisher Scientific). (A) A three-band pattern for E. bovis (20 bp, 210 bp, 343 bp). (B) A four-band pattern for E, zuernii (20 bp, 100 bp, 210 bp, 242 bp). (C) A two-band pattern for E. alabamensis (212 bp, 362 bp).

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Fig. 1 in Usefulness of PCR-RFLP of 18S rRNA gene for rapid post-mortem diagnostics of highly pathogenic Eimeria spp. (Apicomplexa: Eimeriidae) of European bison, Bison bonasus L. with histopathological correlation

Fig. 1. Histopathological lesions associated with endogenous stages of Eimeria spp. in sections of the ileum and colon of European bison (H-E staining). (A) Shortening and blunting of the intestinal villi of the ileum with diffuse infiltration of mononuclear inflammatory cells within the lamina propria, edematous stroma, dilated crypt containing necrotic debris (arrow), and atrophy of submucosal lymphoid follicles (× 20 magnification). (B) Schizonts and degenerating merozoites in the crypt lumen of the colon (arrows); immature macrogamont with a central nucleus (arrowhead) (× 1000 magnification). (C) Immature microgamonts in the epithelial cells of the colon crypt (arrows) (× 400 magnification). (D) Mature microgamont in the epithelial cells of the colon crypt (arrow) (× 1000 magnification). (E) Gametogonic stages of Eimeria development in the epithelial cells of the colon. Microgamont with peripheral microgames (arrowhead), (a) nearly mature microgamonts, (b) macrogamont with eosinophilic wall-forming bodies, (c) early oocyst (× 400 magnification). (F) Mature macrogamont in the epithelial cells of the cecum (arrow) (× 1000 magnification).

Fig. 2. Macroscopic examination. A in Exo-erythrocytic stages of Haemoproteus sp. in common buzzard (Buteo buteo): A histopathological and molecular study

Fig. 2. Macroscopic examination. A pale enlarged liver (Li); B hyperplastic nodules on the surface of the large spleen (S), inflated kidneys (K), and hypostatic congestion of left lung (Lu).

Fig. 5 in Exo-erythrocytic stages of Haemoproteus sp. in common buzzard (Buteo buteo): A histopathological and molecular study

Fig. 5. Maximum-likelihood tree analysis based on partial cytb gene sequences (433 bp) of Haemoproteus from the studied sequence (linage BUTBUT15) and other sequences obtained from MalAvi database. Numbers on the branches indicate the percent of replicates that reproduced the topology for each clade. The percentages of replicate trees in which the associated taxa clustered together in the bootstrap test (1000 replicates) are shown next to the branches. Black circle indicates sequence obtained from the study.

Fig. 1 in Exo-erythrocytic stages of Haemoproteus sp. in common buzzard (Buteo buteo): A histopathological and molecular study

Fig. 1. Gametocytes of haemosporidians (arrows) from the blood of the common buzzards. A-C mature macrogametocytes of L. buteonis in fusiform host cells; A host cell nucleus is not distorted but displaced laterally; B&C host cell nucleus is lateral and flattened; D young gametocyte of Haemoproteus. Giemsa stained thin blood films. Scale bar = 10 μm.

Fig. 4 in Exo-erythrocytic stages of Haemoproteus sp. in common buzzard (Buteo buteo): A histopathological and molecular study

Fig. 4. Megalomeront of Haemoproteus sp. (linage BUTBUT15) from the kidney of a common buzzard. A&B same megalomeront of different magnifications (arrows). The structure is covered with a semi-thick capsule-like wall and contained highly eosinophilic irregularly-shaped cytomeres which have merozoites. Note that the host cell nucleus is not visible inside or close to megalomeront. Scale bars = 200 μm (A); 80 μm (B).

Fig. 3 in Exo-erythrocytic stages of Haemoproteus sp. in common buzzard (Buteo buteo): A histopathological and molecular study

Fig. 3. Meronts of Haemoproteus sp. (linage BUTBUT15) from the lung of a common buzzard. A-D different shapes of pulmonary meronts (arrows). Each meront is surrounded by a thin wall and contained numerous basophilic round to oval merozoites. Note that the host cell nucleus is not visible in meronts. Scale bar = 40 μm.

Fig. 5 in The natural interaction between Myotis nigricans (Schinz, 1821) and its trematodes: A histopathological analysis

Fig. 5. The parasitized intestine of Myotis nigricans. (A) and (B) Trematodes between the intestinal villi do not invade the intestinal glands. (C) Trematode attached to the villus adjacent to its ventral surface, both by the oral sucker and by the ventral sucker. (D) Oral sucker and (E) ventral sucker attached to the epithelium of the intestine, with the view of goblet cells. (F) Peyer patches with visible lymphoid alteration and intestinal glands. (G) Detail of lymphoid cells with nuclear fragmentation and hyperplasia. Abbreviations: (Ig), intestinal glands; (Os), Oral sucker; (Pp), Payer patches; (T), Trematodes; (Vi), Villi; (Vs), ventral sucker.

Fig. 4 in The natural interaction between Myotis nigricans (Schinz, 1821) and its trematodes: A histopathological analysis

Fig. 4. The liver of Myotis nigricans with a parasitized gallbladder. (A) Hepatic parenchyma with significant hepatocyte impairment and visible centrilobular veins and portal triad. (B) Hepatic lobule with the view of the centrilobular vein surrounded by sinusoids capillaries, bile duct, branches of the portal vein, and hepatic artery. (C) Portal triad: bile duct, branches of the portal vein, and hepatic artery. (D), (E) and (F) Hepatocytes with variation in cytoplasmic granular deposits, from clear vacuoles in basophilic cells to dense basophilic granules or chromophobic voluminous granules in cells of low basophilia. Abbreviations and symbols: (arrow), sinusoid capillaries; (arrowhead), granular deposits; (Bd), bile duct; (Cv), centrilobular vein; (Ha), Hepatic artery; (Pv), Portal vein; (Tp) Portal triad; (Va), vacuoles.

Fig. 3 in The natural interaction between Myotis nigricans (Schinz, 1821) and its trematodes: A histopathological analysis

Fig. 3. The non-parasitized intestine of Myotis nigricans. (A) and (B) Longitudinal sections of the jejunum. Details of voluminous, long, and sinuous villi and intestinal glands. (C) Intestinal glands and crypts open at the base of the villi, involved by the periglandular capillary plexus. (D) Intestinal glands and submucosa with circular muscular layers and longitudinal muscular layer. (E) Detail of villus epithelium, showing cylindrical simple, goblet cells and quiliferous vessel. (F) and (G) The ileum segment with numerous Peyer patches located between the submucosa and the intestinal glands. (H) A segment of the ilium with the view of vessels and circular and longitudinal muscle layers. Abbreviations and symbols: (Cml) Circular muscular layer; (Cr) cortical region; (Gc) Globet cells; (Ig) intestinal glands; (Lml), longitudinal muscle layer; (Pp) Payer patches; (Qv) quiliferous vessel; (Vi) Villi; (*) opening of intestinal glands.

Fig. 1 in The natural interaction between Myotis nigricans (Schinz, 1821) and its trematodes: A histopathological analysis

Fig. 1. Histological features of the non-parasitized liver of Myotis nigricans. (A) Detail of hepatic lobes, with a view to the centrilobular vein, a segment of the portal triad, and portal vein. (B) Centrilobular vein surrounded by sinusoids capillaries and rows of hepatocytes. (C) Hepatic capsule formed by cubic cells. (D) Hepatocytes with basophilic cytoplasm, few granulations vary in appearance from translucent to basophilic. Abbreviations: (Cv), Centrilobular vein, (Pv), Portal vein, (arrowhead), hepatocytes.