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opencc-by-4.0Oct 2023View details →

Meta-analysis dataset on the temperature sensitivity of monoterpene emissions

The attached file includes the meta-analysis dataset that was used for the study entitled ''High temperature sensitivity of monoterpene emissions from global vegetation ''

opencc-by-4.0Nov 2024View details →

Dataset for "Anthropogenic emission controls reduce summertime ozone-temperature sensitivity in the United States"

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opencc-by-4.0Nov 2021View details →

Elevational variability and controls on temperature sensitivity of soil organic matter decomposition in alpine forests

All data for ECS21-0520 "Elevational variability and controls on temperature sensitivity of soil organic matter decomposition in alpine forests"

opencc-zeroAug 2022View details →

New tree‐level temperature response curves document sensitivity of tree growth to high temperatures across a US‐wide climatic gradient

Temperature is a key climate indicator, whose distribution is expected to shift right in a warming world. However, the high temperature tolerance of trees is less widely understood than their drought tolerance, especially when it comes to sub-lethal impacts of temperature on tree growth. I use a large data set of annual tree ring widths, combined with a flexible degree-day model, to estimate the relationship between temperature and tree radial growth. I find that tree radial growth responds non-linearly to temperature across many ecoregions of the US: across temperate and/or dry ecoregions, spring-summer temperature increases are beneficial or mostly neutral for tree growth up to around 25-30°C in humid climates and 10-15°C in dry climates, beyond which temperature increases suppress growth. Thirty additional degree-days above the optimal temperature breakpoint lead to an average decrease in tree ring width of around 1-5%, depending on ecoregions, seasons, and inclusion or exclusion of temperature-mediated drought impacts. High temperatures have legacy effects across a 5-year horizon in dry ecoregions, but none in the temperate-humid South-East or among temperature-sensitive trees. I find limited evidence that trees acclimatize to high temperatures within their lifetime: local variation in exposure to high temperatures, which stems from local variation in the timing of tree birth, does not significantly impact the response to high temperatures, although temperature-sensitive trees acquire some heightened sensitivity from early exposure. I also find some evidence that trees adapt to high temperatures in the long-run: across humid ecoregions of the US, high temperatures are 40% less harmful to tree growth, where their average incidence is one standard deviation above average. Overall, these results highlight the strength of a new methodology which, applied to representative tree ring data, could contribute to predicting forest carbon uptake potential and composition under global change.

opencc-by-4.0Sep 2024View details →

Tree germination sensitivity to increasing temperatures: a global meta-analysis across biomes, species and populations.

The dataset contains the files used for the meta-analysis on the role of temperature increases on the germination of tree species from different biomes around the world. This meta-analysis is accepted for publication in Global Ecology and Biography (MS reference number: GEB-2024-0273.R1 ; Article DOI: 10.1111/geb.13921). Files S6 and S7 gather data of germination percentage and time, respectively, at population scale. File S5 is a summary of the publications used as data sources for the meta-analysis. The whole dataset comprises 50 papers addressing 63 species and 250 populations, it covers boreal, temperate, Mediterranean and tropical-subtropical biomes, and a time period between 1996 and 2024.

opencc-zeroOct 2021View details →

The mating system affects the temperature sensitivity of male and female fertility

1. To mitigate effects of climate change it is important to understand species' responses to increasing temperatures. This has often been done by studying survival or activity at temperature extremes. Before such extremes are reached, however, effects on fertility may already be apparent. 2. Sex differences in the thermal sensitivity of fertility (TSF) could impact species persistence under climate warming because female fertility is typically more limiting to population growth than male fertility. However, little is known about sex differences in TSF. 3. Here we first demonstrate that the mating system can strongly influence TSF using the seed beetle Callosobruchus maculatus. We exposed populations carrying artificially induced mutations to two generations of short-term experimental evolution under alternative mating systems, manipulating the opportunity for natural and sexual selection on the mutations. We then measured TSF in males and females subjected to juvenile or adult heat stress. 4. Populations kept under natural and sexual selection had higher fitness, but similar TSF, compared to control populations kept under relaxed selection. However, females had higher TSF than males, and strikingly, this sex difference had increased over only two generations in populations evolving under sexual selection. 5. We hypothesized that an increase in male-induced harm to females during mating had played a central role in driving this evolved sex difference, and indeed, remating under conditions limiting male harassment of females reduced both male and female TSF. Moreover, we show that manipulation of mating system parameters in C. maculatus generates intraspecific variation in the sex difference in TSF equal to that found among a diverse set of studies on insects. 6. Our study provides a causal link between the mating system and TSF. Sexual conflict, (re)mating rates, and genetic responses to sexual selection differ among ecological settings, mating systems and species. Our study therefore also provides mechanistic understanding for the variability in previously reported TSFs which can inform future experimental assays and predictions of species responses to climate warming.

ClinicalTrials.gov32/100

Temperature Sensitive Release of PGE2 and Diminished Energy Requirements in Synovial Tissue With Postoperative Cryotherapy - A Prospective Randomised Study After Knee Arthroscopy

ClinicalTrials.gov study NCT01247376. IPD Sharing: Not stated. Countries: 1. Publications: 4.

restrictedIPD-UNDECIDEDFeb 2026View details →

ClinicalTrials.gov32/100

Temperature-Sensitive Liposomal Doxorubicin and Hyperthermia in Treating Women With Locally Recurrent Breast Cancer

ClinicalTrials.gov study NCT00346229. IPD Sharing: Not stated. Countries: 1. Publications: 2.

restrictedIPD-UNDECIDEDFeb 2026View details →

publicJul 2019View details →

Data from: The influence of soil communities on the temperature sensitivity of soil respiration

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publicMar 2019View details →

Data from: Long-term exposure to higher temperature increases the thermal sensitivity of grazer metabolism and movement

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publicOct 2021View details →

The mating system affects the temperature sensitivity of male and female fertility

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publicMar 2016View details →

Data from: Flowering time of butterfly nectar food plants is more sensitive to temperature than the timing of butterfly adult flight

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publicAug 2022View details →

New tree‐level temperature response curves document sensitivity of tree growth to high temperatures across a US‐wide climatic gradient

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opencc-zeroDec 2018View details →

Data from: Rapid shifts in the thermal sensitivity of growth but not development rate causes temperature-size response variability during ontogeny in arthropods

Size at maturity in ectotherms commonly declines with warming. This near-universal phenomenon, formalised as the temperature-size rule, has been observed in over 80% of tested species, from bacteria to fish. The proximate cause has been attributed to the greater temperature dependence of development rate than growth rate, causing individuals to develop earlier but mature smaller in the warm. However, few studies have examined the ontogenetic progression of the temperature-size response at high resolution. Using marine planktonic copepods, we experimentally determined the progression of the temperature-size response over ontogeny. Temperature-size responses were not generated gradually from egg to adult, contrary to the predictions of a naïve model in which development rate was assumed to be more temperature-dependent than growth rate, and the difference in the temperature dependence of these two rates remained constant over ontogeny. Instead, the ontogenetic progression of the temperature-size response in experimental animals was highly episodic, indicating rapid changes in the extent to which growth and development rates are thermally decoupled. The strongest temperature-size responses occurred temporally mid-way through ontogeny, corresponding with the point at which individuals reached between ~5- 25% of their adult mass. Using the copepod Oithona nana, we show that the temperature-dependence of growth rate varied substantially throughout ontogeny, whereas the temperature dependence of development rate remained constant. The temperature-dependence of growth rate even exceeded that of development rate in some life stages, leading to a weakening of the temperature-size response. Our analyses of arthropod temperature-size responses from the literature, including crustaceans and insects, support these conclusions more broadly. Overall, our findings provide a better understanding of how the temperature-size rule is produced over ontogeny. Whereas we find support for the generality of developmental rate isomorphy in arthropods (shared temperature dependence of development rate across life stages), this concept should not apply to growth rates.

opencc-zeroDec 2014View details →

Data from: Temperature-sensitive fitness cost of insecticide resistance in Chinese populations of the diamondback moth Plutella xylostella

Alleles conferring a higher adaptive value in one environment may have a detrimental impact on fitness in another environment. Alleles conferring resistance to pesticides and drugs provide textbook examples of this trade-off as, in addition to conferring resistance to these molecules, they frequently decrease fitness in pesticide/drug-free environments. We show here that resistance to chlorpyrifos, an organophosphate (OP), in Chinese populations of the diamondback moth, Plutella xylostella, is conferred by two mutations of ace1 – the gene encoding the acetylcholinesterase enzyme targeted by OPs – affecting the amino acid sequence of the corresponding protein. These mutations were always linked, consistent with the segregation of a single resistance allele, ace1R, carrying both mutations, in the populations studied. We monitored the frequency of ace1R (by genotyping more than 20 000 adults) and the level of resistance (through bioassays on more than 50 000 individuals) over several generations. We found that the ace1R resistance allele was costly in the absence of insecticide and that this cost was likely recessive. This fitness costs involved a decrease in fecundity: females from resistant strains laid 20% fewer eggs, on average, than females from susceptible strains. Finally, we found that the fitness costs associated with the ace1R allele were greater at high temperatures. At least two life history traits were involved: longevity and fecundity. The relative longevity of resistant individuals was affected only at high temperatures and the relative fecundity of resistant females – which was already affected at temperatures optimal for development – decreased further at high temperatures. The implications of these findings for resistance management are discussed.

opencc-zeroDec 2016View details →

Data from: Differential temperature sensitivity of synaptic and firing processes in a neural mass model of epileptic discharges explains heterogeneous response of experimental epilepsy to focal brain cooling

Experiments with drug-induced epilepsy in rat brains and epileptic human brain region reveal that focal cooling can suppress epileptic discharges without affecting the brain's normal neurological function. Findings suggest a viable treatment for intractable epilepsy cases via an implantable cooling device. However, precise mechanisms by which cooling suppresses epileptic discharges are still not clearly understood. Cooling experiments in vitro presented evidence of reduction in neurotransmitter release from presynaptic terminals and loss of dendritic spines at post-synaptic terminals offering a possible synaptic mechanism. We show that termination of epileptic discharges is possible by introducing a homogeneous temperature factor in a neural mass model which attenuates the post-synaptic impulse responses of the neuronal populations. This result however may be expected since such attenuation leads to reduced post-synaptic potential and when the effect on inhibitory interneurons is less than on excitatory interneurons, frequency of firing of pyramidal cells is consequently reduced. While this is observed in cooling experiments in vitro, experiments in vivo exhibit persistent discharges during cooling but suppressed in magnitude. This leads us to conjecture that reduction in the frequency of discharges may be compensated through intrinsic excitability mechanisms. Such compensatory mechanism is modelled using a reciprocal temperature factor in the firing response function in the neural mass model. We demonstrate that the complete model can reproduce attenuation of both magnitude and frequency of epileptic discharges during cooling. The compensatory mechanism suggests that cooling lowers the average and the variance of the distribution of threshold potential of firing across the population. Bifurcation study with respect to the temperature parameters of the model reveals how heterogeneous response of epileptic discharges to cooling (termination or suppression only) is exhibited. Possibility of differential temperature effects on post-synaptic potential generation of different populations is also explored.