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5,794 results for “exposures”
Dataset from: "Global labor loss due to humid heat exposure underestimated for outdoor workers"
<p>Data from Environmental Research Letters manuscript 'Global labor loss due to humid heat exposure underestimated for outdoor workers', DOI: https://doi.org/10.1088/1748-9326/ac3dae.</p> <p>Associated Python (Jupyter Lab) scripts and working environment to load and plot data can be found at: https://github.com/LukeAParsons/erfs_comparison</p> <p>Abstract:</p> <p>'Humid heat impacts a large portion of the world’s population that works outdoors. Previous studies have quantified humid heat impacts on labor productivity by relying on exposure response functions that are based on uncontrolled experiments under a limited range of heat and humidity. Here we use the latest empirical model, based on a wider range of temperatures and humidity, for studying the impact of humid heat and recent climate change on labor productivity. We show that globally, humid heat may currently be associated with over 650 billion hours of annual lost labor (148 million full time equivalent jobs lost), 400 billion hours more than previous estimates. These differences in labor loss estimates are comparable to losses caused by the COVID-19 pandemic. Globally, annual heat-induced labor productivity losses are estimated at 2.1 trillion in 2017 PPP$, and in several countries are equivalent to more than 10% of gross domestic product. Over the last four decades, global heat-related labor losses increased by at least 9% (>60 billion hours annually using the new empirical model) highlighting that relatively small changes in climate (<0.5 ◦C) can have large impacts on global labor and the economy.'</p>
Echolocation call parameters of Daubenton's bats during exposure to masking noise
<p>Echolocating bats hunt prey on the wing under conditions of poor lighting by emission of loud calls and subsequent auditory processing of weak returning echoes. To do so, they need adequate echo-to-noise ratios (ENRs) to detect and distinguish target echoes from masking noise. Early obstacle avoidance experiments report high resilience to masking in free-flying bats, but whether this is due to spectral or spatiotemporal release from masking, advanced auditory signal detection or an increase in call amplitude (Lombard effect) remains unresolved. We hypothesized that bats with no spectral, spatial or temporal release from masking noise, defend a certain ENR via a Lombard effect. We trained four bats (<em>Myotis daubentonii</em>) to approach and land on a target that broadcasted broadband noise at four different levels. An array of seven microphones enabled acoustic localization of the bats and source level estimation of their approach calls. Call duration and peak frequency did not change, but average call source levels (SL<sub>RMS</sub>, at 0.1 m as dB re. 20 μPa, root-mean-square) increased, from 112 dB in the no-noise treatment, to 118 dB (maximum 129 dB) at the maximum noise level of 94 dB. The magnitude of the Lombard effect was small (0.13 dB SL<sub>RMS</sub>/dB of noise), resulting in mean broadband and narrowband ENRs of -11 and 8 dB respectively at the highest noise level. Despite these poor ENRs, the bats still performed echo-guided landings, making us conclude that they are very resilient to masking even when they cannot avoid it spectrally, spatially or temporally.</p>
Validation of Spectral Light Simulation Tools: Dataset of Simulated and Measured Indoor Light Exposure
<p>Since the discovery of a new photoreceptor in our eye, and with the growing awareness about the related ipRGC-influenced light (IIL) responses, design applications related to these responses are flourishing. Optimizing our ocular light exposure in buildings can have beneficial effects on our health, well-being, and performance through the action of this photoreceptor. To compare different design options and optimize the lighting conditions for building occupants, lighting simulations are typically used. However, as our IIL responses depend on various aspects of the light exposure including its spectral characteristics, spectral simulations are required. The dataset shared here was originally collected to validate two spectral simulation tools, <em>ALFA</em> and <em>Lark</em>, for the study of building design in relation to occupants’ IIL responses. The validation was done by comparing the simulation outputs against actual measurements, and assessing how reliable these tools were in predicting spectral irradiance under different indoor light conditions. Data were collected in two different experimental setups, one under daylight conditions only and the other one under electric light conditions only. The experimental protocol and README files contain detailed information on how the data was collected and what data was collected.</p>
Differential effects of early or late exposure to prenatal maternal immune activation on mouse embryonic neurodevelopment
<p>Exposure to maternal immune activation (MIA) in utero is a risk factor for neurodevelopmental and psychiatric disorders. MIA-induced deficits in adolescent and adult offspring have been well characterized, however, less is known about the effects of MIA-exposure on embryo development. To address this gap, we collected high-resolution ex vivo magnetic resonance imaging (MRI) of C57BL/6 mouse embryos (gestational day [GD]18) who were prenatally exposed to MIA either early (GD9) or late (GD17) in gestation. We further examined hippocampal neuroanatomy using electron microscopy and identified differential effects due to MIA-timing. </p> <p>The data published here was collected and analyzed for the following publication available as a preprint on BioRxiv (https://www.biorxiv.org/content/10.1101/2021.07.14.452084v2). Briefly, We identify striking neuroanatomical changes in the embryo brain, particularly in the late exposed offspring. An increase in apoptotic cell density was observed in the GD9 exposed offspring, while an increase in the density of dark neurons and glia, putative markers for increased neuroinflammation and oxidative stress, was observed in GD17 exposed offspring, particularly in females. Overall, our findings integrate imaging techniques across different scales to identify differential impact of MIA-timing on the earliest stages of neurodevelopment.</p> <p>In this dataset, you will find a total of <strong>187 preprocessed structural MRIs </strong>(in MINC format) of whole embryos at gestational day 18. These embryos were exposed to poly I:C or vehicle control (0.9% sterile saline) at GD9 or 17. A multi-channel 7.0-T MRI scanner with a 40 cm diameter bore (Varian Inc., Palo Alto, CA), with a custom-built 16-coil solenoid array was used to acquire T2-weighted, gadolinium enhanced structural images at 40 μm3 resolution images from 16 samples concurrently (3D fast spin echo sequence using a cylindrical k-space acquisition; TR/TE=350/12 ms, echo train length=6, two averages, field-of-view 20 mm x 20 mm x 25 mm, matrix size=504 x 504 x 630).</p> <p>An N4 correction for B1 bias field inhomogeneities and denoising using non-local means (minc_anlm) was applied to the T2-weighted images, and the background was set to zero using minc tools. The demographics information for each animal is included in the <strong>demographics.csv</strong> file. </p> <p>The dorsal hippocampus was selected as a region of interest in which the total number and density of total cells, dark neurons, dark glia, apoptotic cells were assessed (as presented in our manuscript). This is available in the <strong>EM_raw_data_per_slice.csv</strong> and <strong>EM_average_per_mouse.csv </strong>files. </p> <p>Included in this data set are the <strong>structural MRIs in MINC format</strong>, corresponding demographics information (<strong>demographics.csv</strong>), electron microscopy data from the dorsal hippocampus (<strong>EM_raw_data_per_slice.csv</strong> and <strong>EM_average_per_mouse.csv)</strong> data, and a <strong>readme.txt</strong> file providing further detail on the data structure and content, and on how to interpret the data column titles.The raw (not-preprocessed) MINC files, as well as MINC files cropped to the head of the embryos available upon request to the authors. </p> <p>Finally, the authors would like to acknowledge the funding bodies that supported the completion of this work including the Canadian Institute for Health Research, the Fonds de Recherche du Québec en Santé, and the Healthy Brains for Healthy Lives at McGill University.</p>
Sublethal exposure to deltamethrin stimulates reproduction and has limited effects on post-hatching maternal care in the European earwig
<p>Data set and associated R script used to obtain the statistical results presented in the corresponding study.</p>
Interactions among multiple stressors vary with exposure duration and biological response
<p>Coastal ecosystems are exposed to multiple anthropogenic stressors. Effective management actions would be better informed from generalized predictions of the individual, combined and interactive effects of multiple stressors; however, few generalities are shared across different meta-analyses. Using an experimental study, we present an approach for analysing regression-based designs with generalized additive models that allowed us to capture nonlinear effects of exposure duration and stressor intensity and access interactions among stressors. We tested the approach on a globally distributed marine diatom, using 72 h photosynthesis and growth assays to quantify the individual and combined effects of three common water quality stressors; photosystem II-inhibiting herbicide exposure, dissolved inorganic nitrogen (DIN) enrichment and reduced light (due to excess suspended sediment). Exposure to DIN and reduced light generally resulted in additivity, while exposure to diuron and reduced light resulted in additive, antagonistic or synergistic interactions, depending on the stressor intensity, exposure period and biological response. We thus find the context of experimental studies to be a primary driver of interactions. The experimental and modelling approaches used here bridge the gap between two-way designs and regression-based studies, which provides a way forward to identify generalities in multiple stressor interactions.</p>
Data from: Active regulation of ultraviolet light exposure overrides thermal preference behaviour in eastern fence lizards
<p>1. Over a century of ecophysiological studies on lizards have perpetuated the assumption that basking and shuttling movements between sun and shade function solely for temperature regulation. However, these behaviors also modulate exposure to ultraviolet (UV) wavelengths that are essential for maintaining physiological homeostasis as well as ensuring proper growth and development and enhancing long-term fitness.</p> <p>2. An alternative hypothesis is that lizards also actively regulate their UV exposure. In this scenario, UV needs may even override temperature needs (or vice versa), generating asymmetries in the ability of a lizard to regulate both conditions equally. We test this hypothesis using field and laboratory data collected on adult <em>Sceloporus undulatus</em>.</p> <p>3. We found that <em>S. undulatus</em> actively regulate UV exposure and prioritize UV over temperature, favoring body temperatures much higher than preferred values to sustain preferred UV exposure. In stark contrast, temperature had no reciprocal impact on UV regulation behavior. Our field data support these patterns, suggesting that lizards may even seek out hotter environments despite thermal costs to enhance UV exposure.</p> <p>4. We conclude that <em>S. undulatus</em> actively regulate for UV as well as temperature. Unfortunately, outside of zoos and private hobbyists, appreciation of the importance of UV for ectotherm survival and reproductive success has been minimal. Addressing this deficit will therefore be vital to improve our understanding of the factors shaping the evolution of ectotherm photoregulation behavior in nature.</p>
OMOP2OBO Drug Exposure Ingredient Mappings
<p><strong>OMOP2OBO Drug Exposure Ingredient Mappings V1.0</strong></p> <p>These mappings were created by the OMOP2OBO mapping algorithm (see links below). OMOP2OBO - the first health system-wide, disease-agnostic mappings between standardized clinical terminologies and eight Open Biomedical Ontology (OBO) Foundry ontologies spanning diseases, phenotypes, anatomical entities, cell types, organisms, chemicals, vaccines, and proteins. These mappings are also the first to be explicitly created using standard terminologies in the Observational Medical Outcomes (OMOP) common data model (CDM), ensuring both semantic and clinical interoperability across a space of N conditions [and N relationships curated in these ontologies].</p> <p>The mappings in this repository were created between OMOP standard drug exposure concepts at the ingredient-level (i.e., RxNorm) to the Chemical Entities of Biological Interest (ChEBI), the National Center for Biotechnology Information Taxon Ontology (NCBITaxon), the Protein Ontology (PRO), and the Vaccine Ontology (VO). All concepts were aligned to at least one ChEBI concept and the remaining ontologies (NCBITaxon, PR, and VO) were mapped by their drug class and/or type (e.g., biologics versus vaccines). For these OMOP domains, owl:intersectionOf (“and”), and owl:unionOf (“or”) constructors were used to construct semantically expressive mappings.</p> <p><br> <strong>Mapping Details</strong><br> Mappings included in this set were generated automatically using OMOP2OBO or through the use of a Bag-of-words embedding model using TF-IDF. Cosine similarity is used to compute similarity scores between all pairwise combinations of OMOP and OBO concepts and ancestor concepts. To improve the efficiency of this process, the algorithm searches only the top 𝑛 most similar results and keeps the top 75th percentile among all pairs with scores >= 0.25. Manually created mappings are also included.</p> <p><strong><em>Mapping Categories</em></strong></p> <ul> <li><strong>Automatic One-to-One Concept</strong>: Exact label or synonym, dbXRef, or expert validated mapping @ concept-level; 1:1</li> <li><strong>Automatic One-to-One Ancestor:</strong> Exact label or synonym, dbXRef, or expert validated mapping @ concept ancestor-level; 1:1</li> <li><strong>Automatic One-to-Many Concept: </strong>Exact label or synonym, dbXRef, cosine similarity, or expert validated mapping @ concept-level; 1:Many</li> <li><strong>Automatic One-to-Many Ancestor:</strong> Exact label or synonym, dbXRef, cosine similarity, or expert validated mapping @ concept-level; 1:Many</li> <li><strong>Manual One-to-One: </strong>Hand mapping created using expert suggested resources; 1:1</li> <li><strong>Manual One-to-Many:</strong> Hand mapping created using expert suggested resources; 1:Many</li> <li><strong>Cosine Similarity:</strong> score suggested mapping -- manually verified</li> <li><strong>UnMapped:</strong> No suitable mapping or not mapped type</li> </ul> <p><em><strong>Mapping Statistics</strong></em><br> Additional statistics have been provided for the mappings and are shown in the table below. This table presents the counts of OMOP concepts by mapping category and ontology:</p> <table align="center"> <thead> <tr> <th scope="col">Mapping category</th> <th scope="col">ChEBI</th> <th scope="col">NCBITaxon</th> <th scope="col">PRO</th> <th scope="col">VO</th> </tr> </thead> <tbody> <tr> <td>Automatic One-to-One Concept</td> <td>3151</td> <td>155</td> <td>43</td> <td>108</td> </tr> <tr> <td>Automatic One-to-Many Constructor</td> <td>404</td> <td>1</td> <td>1</td> <td>0</td> </tr> <tr> <td>Automatic One-to-One Ancestor</td> <td>147</td> <td>17</td> <td>20</td> <td>4</td> </tr> <tr> <td>Automatic One-to-Many Ancestor</td> <td>210</td> <td>3</td> <td>2</td> <td>2</td> </tr> <tr> <td>Cosine Similarity</td> <td>109</td> <td>4241</td> <td>18</td> <td>17</td> </tr> <tr> <td>Manual</td> <td>322</td> <td>230</td> <td>157</td> <td>21</td> </tr> <tr> <td>Manual One-to-Many</td> <td>72</td> <td>14</td> <td>8</td> <td>2</td> </tr> <tr> <td>UnMapped</td> <td>7392</td> <td>7146</td> <td>11558</td> <td>11653</td> </tr> </tbody> </table> <p><br> <strong>Provenance and Versioning: </strong>The V1.0 deposited mappings were created by OMOP2OBO v1.0.0 on October 2022 using the OMOP Common Data Model V5.0 and OBO Foundry ontologies downloaded on September 14, 2020. </p> <p><strong>Caveats:</strong> Please note that these are the original mappings that were created for the preprint. They have not been updated to current versions of the ontologies. In our experience, this should result in very few errors, but we do suggest that you check the ontology concepts used against current versions of each ontology before using them.</p> <p> </p> <p><strong>Important Resources and Documentation</strong></p> <ul> <li>GitHub: <a href="https://github.com/callahantiff/OMOP2OBO">OMOP2OBO</a></li> <li>Project Wiki: <a href="https://github.com/callahantiff/OMOP2OBO/wiki">OMOP2OBO - wiki</a></li> <li>Zenodo Community: <a href="https://zenodo.org/communities/omop2obo">OMOP2OBO</a></li> <li>Preprint Manuscript: <a href="https://doi.org/10.5281/zenodo.5716421">10.5281/zenodo.5716421</a></li> </ul>
Honeybee optomotor behaviour is impaired by chronic exposure to insecticides
<p>Honeybees use wide&[ndash]field visual motion information to calculate the distance they have flown from the hive, and this information is communicated to conspecifics during the waggle dance. Seed treatment insecticides, including neonicotinoids and novel insecticides like sulfoxaflor, display detrimental effects on wild and managed bees, even when present at sublethal quantities. These effects include deficits in flight navigation and homing ability, resulting in decreased survival of exposed worker bees. Neonicotinoid insecticides disrupt visual motion detection in the locust, resulting in impaired escape behaviours, but it had not previously been shown whether seed treatment insecticides disrupt wide&[ndash]field motion detection in the honeybee. Here, we show that sublethal exposure to two commonly used insecticides, imidacloprid (a neonicotinoid) and sulfoxaflor, results in impaired optomotor behaviour in the honeybee. This behavioural effect correlates with altered stress and detoxification gene expression in the brain. Exposure to sulfoxaflor led to sparse increases in neuronal apoptosis, localized primarily in the optic lobes, however there was no effect of imidacloprid. We propose that exposure to cholinergic insecticides disrupts the honeybee&[nprime]s ability to accurately encode wide&[ndash]field visual motion, resulting in impaired optomotor behaviours. These findings provide a novel explanation for previously described effects of neonicotinoid insecticides on navigation and link these effects to sulfoxaflor for which there is a gap in scientific knowledge. --</p>
The interplay between prior selection, mild intermittent exposure, and acute severe exposure in phenotypic and transcriptional response to hypoxia
<p>Hypoxia has profound and diverse effects on aerobic organisms, disrupting oxidative phosphorylation and activating several protective pathways. Predictions have been made that exposure to mild intermittent hypoxia may be protective against more severe exposure and may extend lifespan. Both effects are likely to depend on prior selection on phenotypic and transcriptional plasticity in response to hypoxia, and may therefore show signs of local adaptation. Here we report the lifespan effects of chronic, mild, intermittent hypoxia (CMIH) and short-term survival in acute severe hypoxia (ASH) in four clones of <em>Daphnia magna</em> originating from either permanent or intermittent habitats, the latter regularly drying up with frequent hypoxic conditions. We show that CMIH extended the lifespan in the two clones originating from intermittent habitats but had the opposite effect in the two clones from permanent habitats, which also showed lower tolerance to ASH. Exposure to CMIH did not protect against ASH; to the contrary, <em>Daphnia</em> from the CMIH treatment had lower ASH tolerance than normoxic controls. Few transcripts changed their abundance in response to the CMIH treatment in any of the clones. After 12 hours of ASH treatment, the transcriptional response was more pronounced, with numerous protein-coding genes with functionality in mitochondrial and respiratory metabolism, oxygen transport, and, unexpectedly, gluconeogenesis showing up-regulation. While clones from intermittent habitats showed somewhat stronger differential expression in response to ASH than those from permanent habitats, there were no significant hypoxia-by-habitat of origin or CMIH-by-ASH interactions. GO enrichment analysis revealed a possible hypoxia tolerance role by accelerating the molting cycle and regulating neuron survival through up-regulation of cuticular proteins and neurotrophins, respectively.</p>
News data for studying media exposure to the Boston Marathon bombings
<p>The news data sets released here have been used to study the relationship between media exposure and individuals' threat perception. Media exposure to mass violence has been shown to have a detrimental impact on people's threat perception and mental wellness, but little has been done to explore how exposure to different news content may impact mental health in people's everyday lives. In our study, we empirically test how emotionally potent media coverage of a real-world threat, namely, the Boston Marathon bombings occurred in 2013, alters threat perception of the community members over the first and the third anniversaries (in 2014 and 2016).</p> <p>The data were collected using a wave-based longitudinal design. There are two data sets, and each covers three waves:</p> <ul> <li>Dataset (I) -- news coverage before (Wave 1), during (Wave 2), and after (Wave 3) 2014 anniversary</li> <li>Dataset (II) -- news coverage before (Wave 1), during (Wave 2), and after (Wave 3) 2016 anniversary</li> </ul> <p>The collection procedure was informed by our survey study. Based on the survey completed by our subjects, we identified the four most frequent news outlets in the response: Metro (MT), New York Times (NY), Boston Globe (BG), and Boston Herald (BH). Other outlets, such as USA Today and Wall Street Journal, were reported by less than ten respondents. Therefore, our data collection focused on the news published by the four most frequent outlets.</p> <p>The data sets include the metadata of the news coverage over the aforementioned six waves. The raw content of the news stories was removed to respect the copyright owners.</p> <p><strong>Summary of the data collection procedure </strong></p> <p>We used news aggregators including Google and Yahoo news, to retrieve news articles published by the four outlets on a daily basis. We first collected the URLs of the news articles from the news aggregators and retrieved and parsed the news content using an HTML parser. In total, we collected over 38.5K and 54.1K news articles in dataset I and II, respectively.</p> <p>There are six files; each correspond to news coverage from the outlets in each wave. In these files, each line contains four columns: outlet, time, title, url which indicate the outlet of each news article, the time of publishing, the title of the article, and the URL to the article.</p> <p>We are making the data sets available for academic researchers and public use, to enable the discovery of new insights and development of better techniques to improve crisis communication and mental wellness.</p>
Crop exposure Switzerland (2021)
"Crop exposure for Switzerland in 2021 obtained from <a href="https://geodienste.ch/services/lwb_nutzungsflaechen">geoddienste.ch</a>. Including a crop mask, the number of fields and the total crop area at 1, 2, 4, and 8 km resolution for the following crop types: winter wheat, maize (incl. silage and forage maize), winter barley, rapeseed, and grapevine as well as an aggregate crop class field crops (incl. wheat, maize, barley, and rapeseed). The dataset can be used for any kind of risk assessment studies for these crops in Switzerland.
Fig. 1 in Long-term exposure of Aedes aegypti to Bacillus thuringiensis svar. israelensis did not involve altered susceptibility to this microbial larvicide or to other control agents
Fig. 1 Resistance ratios (RR) betseen the lethal concentrations of Bti and its toxins (Cru11Aa, Cru4Ba), temephos (Tem) and diflubenzuron (Dif) for third-instar Ae. aegypti larvae from the RecBti strain compared to that of the reference strain. a RR at LC50. b RR at LC90
# Replication code and data for: Global projections of heat exposure of older adults
<p># Replication code and data for: Global projections of heat exposure of older adults<br>By Giacomo Falchetta, Enrica De Cian, Ian Sue Wing and Deborah Carr<br>Nature Communications. DOI: 10.1038/s41467-024-47197-5</p> <p>An output data file, containing grid-cell level counts of people by age group, of climate hazard indicators, and of heat exposure metrics for both the historical climate and current demography and for future scenarios and projections is contained in the repository ("aging_climate_output_data.csv"). </p> <p>Software requirements:<br>- R v4.3+: https://cran.r-project.org/bin/windows/base/<br>- RStudio: v2023.06.0+: https://posit.co/download/rstudio-desktop/<br>- Package dependencies: raster, sf, tidyverse, rasterVis, rgdal, maptools, pbapply, terra, knitr, kableExtra, modelsummary, openxlsx, xtable, ggforce, maptools, weights, spatstat, rworldmap, scales, patchwork, stars, viridis, devtools, stargazer, readxl, nominatimlite, urbnmapr</p> <p>To replicate the analysis:<br>- Clone the replication code repository from https://github.com/giacfalk/aging_climate<br>- Download input data from this Zenodo data repository<br>- Download all the 1km age and gender-stratified global population counts rasters from the following WorldPop page https://hub.worldpop.org/geodata/summary?id=24798 and put them in a subdirectory of the working directory called "AGEPOP"<br>- Run the "project_pop.R" script to generate gridded age-stratified population data for each SSP scenario<br>- Run the "compare_pop_projections.R" file to compare the generated gridded age-stratified population data with an array of pre-existing sources from different countries and produce a summary comparison table (NOTE: before running the script, decompress the "new_comparison_data.zip" folder into the working directory)<br>- Run "projections_exposure_m.R" to quantify heat exposure and generate the figures and tables reported in the paper</p> <p>To process the data and run succesfully, the script requires a computer with at least 32GB RAM. The running time varies based on CPU characteristics, but a runtime of at least 2 hours should be expected to generate all the output data, figures, and tables. All output files are saved in the working directory.</p> <p>___</p> <p>This work is licensed under a Creative Commons Attribution-NonCommercial-ShareAlike 4.0 International License.</p> <div> <div> <div> </div> <div> <div> <div> </div> <div> <p> </p> <p> </p> </div> </div> </div> </div> </div>
Data from : Damage to tropical forests caused by tropical cyclones is driven by wind speed but mediated by topographical exposure and tree characteristics
<p>These datasets have been used in the following paper:</p> <p>Ibanez, T., Bauman, B., Aiba, S.-i., Arsouze, T. Bellingham, P.J., Birkinshaw, C., Birnbaum, P., Curran, T.J., DeWalt, S.J., Dwyer, J., Fourcaud, T., Franklin, J., Kohyama, T.S., Menkes, C. Metcalfe, D.J., Murphy, H., Muscarella, R., Plunkett, G.M., Sam, C., Tanner, E., Taylor, B.N., Thompson, J., Ticktin, T., Tuiwawa, M.V., Uriarte, U., Webb, E.L., Zimmerman, J.K., Keppel, G. Damage to tropical forests caused by tropical cyclones is driven by wind speed but mediated by topographical exposure and tree characteristics. Accepted for publication in <em>Global Change Biology</em>.</p> <p>Data users are invited to cite this paper and the original paper(s) corresponding to the data they use (see "Reference" column in each dataset). We also encourage potential users to contact the data owners for collaboration.</p> <p>These datasets are compiled empirical data on the damage caused by 11 cyclones occurring over the past 40 years, from 74 forest plots representing tropical regions worldwide. Damage are given at the tree (whether or not each tree has been uprooted or snapped) and at the plot level (number of uprooted or snapped trees in each plot).</p> <p>MSW: Maximum sustained wind speed (m.s-1)</p> <p>EXP: Topographical exposure to wind</p> <p>DBH: Diameter at breast height (cm)</p> <p>WD: Wood density (g.cm-3)</p>
Number of fishers adaptations with increasing single hazard exposure
<p>Using a systematic review approach, we identified a global dataset of 301 reported adaptation responses of small-scale fishers to climate change. Here we calculated the number of the different types of fishers’ responses inside each of the corresponding climate change hazard exposure levels (i.e. percentiles) from four selected hazards: sea surface temperature rate of change, accumulated intensity of marine heatwaves, sea level rise of sea surges, and frequency of tropical storms. The magnitude of exposure was calculated using the following percentiles: 25th percentile (low exposure), 50th (medium), 75th (high), and 90th (hotspot)</p>
Responsiveness to cold snaps by turtle embryos depends on exposure timing and duration
<p>Characterizing how organisms respond to transient temperatures may further our understanding of their susceptibility to climate change. In animals with temperature-dependent sex determination (TSD), unusual transient temperatures during incubation result in sex bias and may therefore impair population breeding capacity. Past studies in the red-eared slider turtle (<em>Trachemys scripta</em>) have demonstrated that the timing and duration of heat exposure ("heat waves") can have major implications for the response of genes involved in gonadal development and the production of female hatchlings. Yet, no study has considered how the response of these genes to transient cold exposure ("cold snaps") may affect gene expression and influence the resulting production of males.</p> <p>We investigated how cold snap timing and duration affect gonadal gene expression in <em>T. scripta</em> embryos. Additionally, we explored the effect of early cold snap exposure duration on resulting hatchling sex ratios. Results show that responsiveness to cool temperatures changes rapidly across development, such that genes that responded to cold snaps when exposure began on incubation day 14 responded differently when cold exposure occurred just 4 to 8 days later. The sex ratio experiment revealed that embryos experiencing an early cold snap also require a long exposure (> 20 days) before most commit to testis development, further suggesting that early development under warm temperatures may lower their sensitivity to later cold snaps. These results highlight how individual responses to incubation temperature can change rapidly across development in turtles and have important effects on sex ratios. We discuss how variation in responsiveness to transient temperatures might help maintain mixed-sex ratios under variable thermal conditions in nature and may permit adaptive population responses to climate change.</p>
Exposure and fragility of a virtual oil refinery testbed for seismic risk assessment
<p><span>Α</span><span> </span><span>virtual mid-size oil refinery, located in a high-seismicity region of Greece, is offered as a testbed for developing and testing system-level assessment methods. The dataset includes (a) a full geolocated exposure model with all pertinent critical assets, namely tanks, pressure vessels, process towers, chimneys, equipment-supporting buildings, and a flare; (b) the corresponding record-wise asset demands and summarized fragilities derived via nonlinear dynamic analyses on reduced-order numerical models.</span></p>
Figure 4 in Perinatal exposure to a high-fat diet alters proopiomelanocortin, neuropeptide Y and dopaminergic receptors gene expression and the food preference in offspring adult rats
Figure 4. Body weight on the 120th offspring from mothers submitted the control diet or the high-fat diet. Values are presented as mean ± SEM using two-way ANOVA followed by the Bonferroni multiple-comparison test. *p<0,05; **p<0,005.
Figure 3 in Perinatal exposure to a high-fat diet alters proopiomelanocortin, neuropeptide Y and dopaminergic receptors gene expression and the food preference in offspring adult rats
Figure 3. Body weight on the second day life's of offspring from mothers submitted the control diet (C) or the high-fat diet (H). Values are presented as mean + SEM using Student t-test. C: n = 23; H: n = 23.
ScienceDex guides
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These curated guides explain access requirements, typical timelines, costs, and reuse considerations for widely used research datasets.
Allen Brain Atlas
Allen Brain Atlas is an Allen Institute collection of brain map atlases, datasets, APIs, and analysis tools covering mouse, human, and non-human primate brain resources.
Annotated Behaviour and Observability Dataset (ABODe)
ABODe is a University of Edinburgh DataShare dataset for behavior classification in group-housed mice using home-cage video, identities, bounding boxes, ground-plate positions, and annotator labels.
DANDI Archive for NWB datasets
DANDI is a BRAIN Initiative archive for publishing and sharing neurophysiology data, including electrophysiology, optophysiology, and behavioral data packaged as NWB and related standards.
International Brain Laboratory public data
The International Brain Laboratory public data releases expose standardized mouse decision-making experiments, including Neuropixels recordings, widefield calcium imaging, behavior, and session metadata accessed through the ONE API.
OpenNeuro
OpenNeuro is a free, open platform for sharing neuroimaging datasets, with public search, dataset pages, and download paths for web, S3, DataLad, and the OpenNeuro CLI.