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Fig. 1 in Effect of entomopathogens on Africanized Apis mellifera L. (Hymenoptera: Apidae)
Fig. 1. Survival curve (hours) of Africanized Apis mellifera workers. (A) Entomopathogens sprayed on A. mellifera, (B) entomopathogens on a smooth surface, (C) entomopathogens on soy leaves and (D) entomopathogens mixed with candy paste. Kaplan–Meier vs Weibull survival test adjusted for time (h). Temperature 27 ± 2 ◦ C, 12-h photophase and relative humidity of 60 ± 10%. Sterilized distilled water.
Fig. 2 in Effect of different diets on biology, reproductive variables and life and fertility tables of Harmonia axyridis (Pallas) (Coleoptera, Coccinellidae)
Fig. 2. Survival probability (lx), expressed in percentage, and specific fertility (mx) expressed as average number of eggs per day of Harmonia axyridis (Pallas, 1773) fed on Brevicoryne brassicae Linnaeus, 1758. Temperature 25 ± 1 ◦C, 70 ± 10% RU and humidity and 12:12 h L:D.
Fig. 1 in Effect of different diets on biology, reproductive variables and life and fertility tables of Harmonia axyridis (Pallas) (Coleoptera, Coccinellidae)
Fig. 1. Survival probability (lx), expressed in percentage, and specific fertility (mx) expressed as average number of eggs per day of Harmonia axyridis (Pallas, 1773) fed on Cinara atlantica (Wilson, 1919). Temperature 25 ± 1 ◦C, 70 ± 10% RU and humidity and 12:12 h L:D.
Fig. 3 in Effect of different diets on biology, reproductive variables and life and fertility tables of Harmonia axyridis (Pallas) (Coleoptera, Coccinellidae)
Fig. 3. Survival probability (lx), expressed in percentage, and specific fertility (mx) expressed as average number of eggs per day of Harmonia axyridis (Pallas, 1773) fed on Anagasta kuehniella (Zeller, 1879). Temperature 25 ± 1 ◦C, 70 ± 10% RU and humidity and 12:12 h L:D.
Fig. 1 in Effect of the presence of brood and fungus on the nest architecture and digging activity of Acromyrmex subterraneus Forel (Hymenoptera, Formicidae)
Fig. 1. Plaster mold of a nest excavated by Acromyrmexsubterraneus workers. (A) Plaster mold of tunnels; (B) a molded and dried nest ready to be removed; (C) labeled and measured structure.
Fig. 3 in Effect of the presence of brood and fungus on the nest architecture and digging activity of Acromyrmex subterraneus Forel (Hymenoptera, Formicidae)
Fig. 3. Boxplot showing the variation in digging activity according to treatment (indicated above each graph) and time.
Figure 3 in Effects of genetic relatedness, spatial distance, and context on intraspecific aggression in the red wood ant Formica pratensis (Hymenoptera: Formicidae)
Figure 3. Correlation between spatial distance and aggression levels in the field. Open circles correspond to monodomous colonies and filled circles correspond to the polydomous one.
Figure 1. Map showing the localities where F in Effects of genetic relatedness, spatial distance, and context on intraspecific aggression in the red wood ant Formica pratensis (Hymenoptera: Formicidae)
Figure 1. Map showing the localities where F. pratensis colonies were sampled for the analysis of genetic relatedness and tested for their aggressive behavior towards each other. The numbers denote the localities. 1: Balaban village (N 41°49ʹ18ʺ, E 27°40ʹ44ʺ) containing three nests; B1, B2, and B3, 2: Asilbeyli village (N 41°39ʹ32ʺ, E 27°13ʹ50ʺ), one nest (As), 3: Ulukonak village (N 41°39ʹ35ʺ, E 27°01ʹ52ʺ) one nest (U), 4: Doğanköy village (N 41°56ʹ12ʺ, E 26°41ʹ20ʺ) one nest (D), and 5: Ahmetler village (N 42°00ʹ37ʺ, E 27°11ʹ12ʺ), three nests; Ah1, Ah2, and Ah3.
Figure 11 in All aspects of the toxic effects of lipopolysaccharide on rat liver and the protective effect of vitamin E and sodium selenite
Figure 11. DNA damage in rat liver exposed to LPS + VE and/or SS. (a) Control group, (b) VE, (c) SS, (d, e) LPS, (f) LPS + VE, (g) LPS + SS, and (h, i) LPS + VE + SS groups.
Figure 10 in All aspects of the toxic effects of lipopolysaccharide on rat liver and the protective effect of vitamin E and sodium selenite
Figure 10. Histological features of the apoptotic areas in the liver of the rats. A, B, C → Control; D, E, F → SS; G, H, I →VE; J, K, L → SS + VE; M, N, O → LPS; P, Q, R → LPS + SS; S, T, U, → LPS + VE; and V, W, X → LPS + SS + VE.
Figure 6 in All aspects of the toxic effects of lipopolysaccharide on rat liver and the protective effect of vitamin E and sodium selenite
Figure 6. Liver sections of the LPS + SS-treated rats. Showing (A) ⇑: dilation of the sinusoids, *: necrosis, and ►: vacuolar degeneration; and (B) ↑↑: vascular congestion and ⇒: hemorrhage at 200×.
Figure 4 in All aspects of the toxic effects of lipopolysaccharide on rat liver and the protective effect of vitamin E and sodium selenite
Figure 4. Liver sections of the LPS-treated rats. (A) Showing Δ: leukocyte infiltration, *: necrosis, Δ: dilation of the sinusoids at 200×; (B) ►: vacuolar degeneration, →: binucleated hepatocytes at 400×; (C) ⇒: hemorrhage, Δ: leukocyte infiltration, *: necrosis, ↑↑: vascular congestion; and (D) ⇑: dilation of the sinusoids and *: necrosis at 200×.
Figure 3 in All aspects of the toxic effects of lipopolysaccharide on rat liver and the protective effect of vitamin E and sodium selenite
Figure 3. Liver section of the control rats, CV: central vein at 200×. Histological structures (A, B) of the control, VE, SS, and VE + SStreated rats were similar to the control group.
Figure 2 in All aspects of the toxic effects of lipopolysaccharide on rat liver and the protective effect of vitamin E and sodium selenite
Figure 2. TEAC values (μmol of Trolox equiv/gram tissue) in the liver of rats treated with LPS (10 mg/kg bw), VE (200 mg/kg bw), and SS (0.35 mg/kg bw). Significance was accepted as P <0.05.
Figure 1 in All aspects of the toxic effects of lipopolysaccharide on rat liver and the protective effect of vitamin E and sodium selenite
Figure 1. FRAP values (μmol of FeII equiv/gram tissue) in the liver of rats treated with LPS (10 mg/kg bw), VE (200 mg/kg bw), and SS (0.35 mg/kg bw). Significance was accepted as P <0.05.
Figure 4 in Evaluation of nickel-induced brain injuries in rats via oxidative stress and apoptosis: attenuating effects of hyperoside
Figure 4. Morphology and location of PAS granules of brain tissues of control (A), Hyp-treated (B), Ni-treated (C), and Ni + Hyp-treated (D) rats. Homogeneous PAS staining was observed in the control group (A) and the Hyp-treated (B) groups. Clusters of granules shown inside square (C) and by black arrow (D), respectively (200× magnification).
Figure 3 in Evaluation of nickel-induced brain injuries in rats via oxidative stress and apoptosis: attenuating effects of hyperoside
Figure 3. Histological views of brain tissues of control (A), Hyp-treated (B), Ni-treated (C), and Ni + Hyp-treated (D) rats. Small blood vessels (BV) from pia mater penetrate the cerebral cortex substance pyramidal cells (PC) with apical dendrites closely associated with smaller, round glial cells (GC) (A and B). Perivascular inflammation around a blood vessel (inside square) (C) and perivascular inflammatory cell infiltrates in hematoxylin and eosin-stained brain tissue (D) (200× magnification).
Figure 2 in The effects of feeding obese rats with bee bread on leptin and ghrelin expression
Figure 2. Cells stained with ghrelin (A) and leptin (B) in hypothalamus tissue are shown with black arrows. A: Group 1 (fed with standard rat food), B: Group 2 (obese - fed with high-fat diet), C: Group 3 (high-fat diet and 100 mg/kg/day bee bread), D: Group 4 (high-fat diet and 200 mg/kg/day bee bread), E: Group 5 (high-fat diet and 300 mg/kg/day metformin). Image zoom 400×.
Figure 1 in The effects of feeding obese rats with bee bread on leptin and ghrelin expression
Figure 1. Cells, stained with hematoxylin and eosin in hypothalamus tissue are shown with black arrows. Image zoom 400×. A: Group 1 (fed with standard rat food), B: Group 2 (obese - fed with high-fat diet), C: Group 3 (high-fat diet and 100 mg/kg/day bee bread), D: Group 4 (high-fat diet and 200 mg/kg/day bee bread), E: Group 5 (high-fat diet and 300 mg/kg/day metformin).
Figure 5 in Evaluation of nickel-induced brain injuries in rats via oxidative stress and apoptosis: attenuating effects of hyperoside
Figure 5. Brain tissues of control (A) and Hyp (B) groups were negative for Congo red. Amyloid deposits in brain parenchyma and around blood vessels shown inside square (C) and marked by black arrows (D) (200× magnification). Differences in measured parameters among the groups were analyzed with a nonparametric test (Kruskal–Wallis). Dual comparisons between groups exhibiting significant values were evaluated with a Mann–Whitney U test (P <0.05).
ScienceDex guides
Understand access before you commit
These curated guides explain access requirements, typical timelines, costs, and reuse considerations for widely used research datasets.
Allen Brain Atlas
Allen Brain Atlas is an Allen Institute collection of brain map atlases, datasets, APIs, and analysis tools covering mouse, human, and non-human primate brain resources.
Annotated Behaviour and Observability Dataset (ABODe)
ABODe is a University of Edinburgh DataShare dataset for behavior classification in group-housed mice using home-cage video, identities, bounding boxes, ground-plate positions, and annotator labels.
DANDI Archive for NWB datasets
DANDI is a BRAIN Initiative archive for publishing and sharing neurophysiology data, including electrophysiology, optophysiology, and behavioral data packaged as NWB and related standards.
International Brain Laboratory public data
The International Brain Laboratory public data releases expose standardized mouse decision-making experiments, including Neuropixels recordings, widefield calcium imaging, behavior, and session metadata accessed through the ONE API.
OpenNeuro
OpenNeuro is a free, open platform for sharing neuroimaging datasets, with public search, dataset pages, and download paths for web, S3, DataLad, and the OpenNeuro CLI.