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6,704 results for “reports”
Figure 1 in The first report on oribatid mites in tundra belts of the Lovozersky Mountains on the Kola Peninsula, Russia
Figure 1 The location of the study plots in the Lovozersky Mountains (indicated by the arrow).
Excel mapping tools for 2020 zoonoses data reporting
<p>The main objective of the mapping tools is to provide a simple and useable platform for Member states and other reporting countries to map their country-specific standard terminology to that used by EFSA and to enable the production of an XML file for the submission of sample or aggregated-based zoonoses monitoring data via the Data Collection Framework (DCF).</p> <p>The catalogues and the specific hierarchy of each data model (AMR, ESBL, PRV, FBO, AP, DS and SSD2 for <em>Echinococcus multilocularis</em>) are already inserted into each of the specific mapping tool. Specific Excel mapping tools corresponding to each of the seven data models are available.</p> <p>Dynamic or manual version of the tool can be chosen for the first six data models.</p> <p>The Emulti_SSD2_tool can be used to report sample-based results of <em>Echinococcus multilocularis</em> under the Commission Delegated Regulation (EU) 2018/772.</p>
Catalogues for 2020 zoonoses data reporting
<p>The EFSA catalogues applied for the 2020 zoonoses data reporting are made available in Excel format for easier use and reference.</p>
Supporting publication for 'Prevalence sample-based guidance for reporting 2020 data'
<p>These two Excel documents help in mapping terms from the matrix catalogue ZOO_CAT_MATRIX used in the aggregated prevalence data model to FoodEx2 codes and offer examples on how prevalence data can be reported using SSD2.</p>
Data from: Increases and decreases in marine disease reports in an era of global change
<p>Outbreaks of <span class="il">marine</span> infectious diseases have caused widespread mass mortalities, but the lack of baseline data has precluded evaluating whether <span class="il">disease</span> is increasing or decreasing in the ocean. We use an established literature proxy method from Ward and Lafferty (2004) to analyze a 44-year <span class="il">global</span> record of normalized <span class="il">disease</span> reports from 1970 to 2013. Major <span class="il">marine</span> hosts are combined into nine taxonomic groups, from seagrasses to <span class="il">marine</span> mammals, to assess <span class="il">disease</span> swings, defined as positive or negative multi-decadal shifts in <span class="il">disease</span> reports across related hosts. Normalized <span class="il">disease</span> reports increased significantly between 1970 and 2013 in corals and urchins, indicating positive <span class="il">disease</span> swings in these environmentally sensitive ectotherms. Coral <span class="il">disease</span> reports in the Caribbean correlated with increasing temperature anomalies, supporting the hypothesis that warming oceans drive infectious coral diseases. Meanwhile, <span class="il">disease</span> risk may also decrease in a changing ocean. <span class="il">Disease</span> reports decreased significantly in fish and elasmobranchs, which have experienced steep human-induced population declines and diminishing population density that, while concerning, may reduce <span class="il">disease</span>. The <span class="il">increases</span> and decreases in <span class="il">disease</span> reports across the 44-year record transcend short-term fluctuations and regional variation. Our results show that long-term changes in <span class="il">disease</span> reports coincide with recent decades of widespread environmental <span class="il">change</span> in the ocean.</p>
Figures 3A-D in A new species of Trichomyia (Diptera: Psychodidae: Trichomyiinae) and report of antennal sensilla in adult
Figures 3A-D. (A) Ascoid of Trichomyia sp. 1; (B) Flagellomere of Trichomyia sp.3 and three short sensilla (arrows pointing the bristles); (C) Apiculus of Trichomyia sp. 2 with apical sensillum (arrow showing the sensillum); (D) Apiculus of Trichomyia sp. 2 with apical sensillum (arrow showing the sensillum). (Abbreviations: ap = apiculus, asc = ascoid).
Figures 1A-G in A new species of Trichomyia (Diptera: Psychodidae: Trichomyiinae) and report of antennal sensilla in adult
Figures 1A-G. Trichomyia muiraquita sp. nov. (A) Head, anterior view; (B) Palpus; (C) Scape, pedicel and basal flagellomeres; (D) Halter; (E) Right wing; (F) Thorax.
Figures 2A-D in A new species of Trichomyia (Diptera: Psychodidae: Trichomyiinae) and report of antennal sensilla in adult
Figures 2A-D. Trichomyia muiraquita sp. nov. (A) Male terminalia, lateral: gonocoxite, arm of gonocoxite, gonostilus, epandrium, post-hypandrial plate; cerci and aedeagus; (B) Male terminalia, dorsal: arm of gonocoxites, post-hypandrial plate, aedeagus, right gonostylus; (C) Apex of gonostylus, lateral; (D) Cerci, epandrium and hypropoct. (Abbreviations: agx = arm of gonocoxite, aed = aedeagus, gst = gonostylus, hyp = hypoproct, pph = plate pos-hipandrial).
Leptomeningeal metastasis from Adrenocortical carcinoma: a case report
<p>Adrenocortical carcinoma (ACC) is an uncommon endocrine malignancy with limited treatment options. While overall 5-year survival rate in patients with ACC is 35%, the disease is often rapidly progressive with long-term survival in only 5% of patients. Although tumor stage, grade and excess hormonal activity predict unfavorable prognosis, additional biomarkers are needed to identify patients with aggressive disease.</p> <p>A 23-year-old woman presented with rapidly progressing signs and symptoms of Cushing's syndrome, with associated abdominal pain and fullness. Evaluation revealed a large left adrenal mass which had developed over 8 months. En bloc surgical resection was performed by an endocrine surgeon, and pathology revealed adrenocortical carcinoma with Ki67 of 60%. Despite adjuvant treatment with mitotane and etoposide-doxorubicin-carboplatin chemotherapy, the patient had rapid disease progression with metastatic spread to liver, lung, bone, brain and leptomeningies and she died eleven months after the initial diagnosis. Subsequent analysis of patient's tumor revealed mutations in TP53 and MEN1. RNA sequencing was compared against the The Cancer Genome Atlas data set and clustered with the high steroid, proliferative subtype, associated with the worst prognosis. The tumor also demonstrated a low BUB1B/PINK1 ratio as well as G0S2 hypermethylation, both predictive of very aggressive ACC.</p> <p>This case represents a subset of ACC characterized by rapid and fatal progression. Clinically available predictors as well as recently reported molecular signatures and biomarkers correlated with this tumor's aggressiveness, suggesting that development and validation of combinations of biomarkers may be useful in guiding personalized approaches to patients with ACC.</p>
A Bayesian Approach to Detect Pedestrian Destination-Sequences from WiFi Signatures: Data (tech. report 2013)
<p>This dataset contains the data used in:</p> <p>Danalet, A., Farooq, B. and Bierlaire, M. (2013). A Bayesian Approach to Detect Pedestrian Destination-Sequences from WiFi Signatures, Technical report, Transport and Mobility Laboratory, ENAC, Ecole Polytechnique Fédérale de Lausanne, Lausanne. URL: http://infoscience.epfl.ch/record/189759 (full text available)</p> <p>It contains data and a technical report describing</p> <ul> <li>WiFi traces</li> <li>Pedestrian Semantically-Enriched Routing Graph (SERG), and</li> <li>Potential Attractivity measure (PAM).</li> </ul>
Data of quantitative, ratiometric dual fluorescence reporter assay at the single-cell level
<p>The data set shows the evaluation of a new dual fluorescence reporter assay using three test genes, vimentin, lamin A/C and Arp3. This fluorescence protein-based reported assay successfully predicts siRNA efficacy with high fidelity. Data include images of Hela cells transfected with Vim-T4 ad Vim-T5 knockdown contructs; images of dual fluorescence assay; data analysis for knockdown experiments, Western blotting analysis of vimentin-knockdown in Hela cells, knockdown of endogenous lamin A/C in Hela cells; quantification of lamin A/C knockdown in Hela cells; dual fluorescence assay of human vimentin genes using three plasmids; Western blotting of Arp3 knockdown in SCC9 cells.</p> <p>All files can be opened using LibreOffice or OpenOffice. Some of the ODS files have a minor problem when opening them in Excel in the Windows computers. Excel will give warning about some unreadable contents. By choosing the repair option, the file contents will be shown without changes compared to the original data. Detailed explanation is given in the “Explanation file”.</p>
libs-github-api: fix "lines" mistake — GitHub API reports bytes, not lines
<p>Correct mistake in several places, including data file and scripts.</p>
IMP HTML reports (preprint)
<p>ZIP file contains the HTML files which are referred to as the "Additional file 1" in the IMP manuscript.<br> </p>
Supporting Data: A large-scale dataset of solar event reports from automated feature recognition modules.
<p>This is the supporting dataset for the paper:</p> <p>A large-scale dataset of solar event reports from automated feature recognition modules. Michael A. Schuh, Rafal A. Angryk, Petrus C. Martens. Journal of Space Weather and Space Climate, 2016.</p>
Nivolumab-associated acute glomerulonephritis: Case report and literature review
<p><strong>Background:</strong> Immune checkpoint inhibitors are changing the landscape of oncologic treatment as they are significantly improving treatment of multiple malignancies. Nivolumab, anti PD-1 antibody, is FDA-approved for treatment of melanoma, non-small cell lung cancer and kidney cancer and can cause a spectrum of autoimmune reaction. The adverse effect can occur at any organ system in the body, but most commonly affects colon, lung, liver or endocrine system. Although rare, kidneys also can be involved.</p> <p><strong>Case presentation: </strong>A 70 year old male with clear cell kidney cancer was admitted with acute kidney injury while he was receiving nivolumab treatment. A kidney biopsy revealed diffuse tubular injury and immune complex mediated glomerulonephritis. Electron microscopic exam of the specimen demonstrated hump-like subepithelial deposits. Nivolumab was discontinued and the patient was started on high dose steroid. After 5 months of systemic corticosteroid and hemodialysis, patient’s kidney function improved to his baseline. In spite of the prolonged treatment interruption, immunosuppressive therapy did not compromise the anticancer effect of nivolumab.</p> <p><strong>Conclusion: </strong>Immune-related adverse effect in kidney can cause autoimmune glomerulonephritis as well as tubulo-interstitial injury. In previous literature, immune-related nephritis generally responded well to systemic corticosteroid. Based our experience, prolonged course of high dose of steroid and hemodialysis may be required to achieve adequate treatment effect. </p>
IMP HTML reports
<p>This file contains all the HTML reports generated by IMP for the analysis of datasets reported in the article.</p>
Figure 1. from: Defining the Scholarly Commons - Reimagining Research Communication. Report of Force11 SCWG Workshop, Madrid, Spain, February 25-27, 2016 - Research Ideas and Outcomes 2: e9340 (26 May 2016) https://doi.org/10.3897/rio.2.e9340
Figure 1. - Demographic breakdown of workshop attendees
Figure 4. from: Defining the Scholarly Commons - Reimagining Research Communication. Report of Force11 SCWG Workshop, Madrid, Spain, February 25-27, 2016 - Research Ideas and Outcomes 2: e9340 (26 May 2016) https://doi.org/10.3897/rio.2.e9340
Figure 4. - Perspective Engine
Figure 2. from: Defining the Scholarly Commons - Reimagining Research Communication. Report of Force11 SCWG Workshop, Madrid, Spain, February 25-27, 2016 - Research Ideas and Outcomes 2: e9340 (26 May 2016) https://doi.org/10.3897/rio.2.e9340
Figure 2. - Opening night presentation by Maryann Martone (available as Google Slides)
OAPEN usage report for open access books funded by the Austrian Science Fund (FWF) 2014 - 2016
<p>For several years now, the Stand-Alone Publications Programme by the Austrian Science Fund (FWF) has been funding the production and simultaneous open-access publication of academic books, see: <a href="http://www.fwf.ac.at/en/research-funding/fwf-programmes/stand-alone-publications/">http://www.fwf.ac.at/en/research-funding/fwf-programmes/stand-alone-publications/</a></p> <p>All FWF-funded books are accessible free of charge in the FWF E-Book Library <a href="https://e-book.fwf.ac.at/">https://e-book.fwf.ac.at/</a> and in the OAPEN Library <a href="http://www.oapen.org/search">http://www.oapen.org/search</a>. </p> <p>The OAPEN Library’s annual data on book usage for 2014 to 2016 shows that the number of archived open-access books is rising steadily, as is the number of downloads per book.</p>
ScienceDex guides
Understand access before you commit
These curated guides explain access requirements, typical timelines, costs, and reuse considerations for widely used research datasets.
Allen Brain Atlas
Allen Brain Atlas is an Allen Institute collection of brain map atlases, datasets, APIs, and analysis tools covering mouse, human, and non-human primate brain resources.
Annotated Behaviour and Observability Dataset (ABODe)
ABODe is a University of Edinburgh DataShare dataset for behavior classification in group-housed mice using home-cage video, identities, bounding boxes, ground-plate positions, and annotator labels.
DANDI Archive for NWB datasets
DANDI is a BRAIN Initiative archive for publishing and sharing neurophysiology data, including electrophysiology, optophysiology, and behavioral data packaged as NWB and related standards.
International Brain Laboratory public data
The International Brain Laboratory public data releases expose standardized mouse decision-making experiments, including Neuropixels recordings, widefield calcium imaging, behavior, and session metadata accessed through the ONE API.
OpenNeuro
OpenNeuro is a free, open platform for sharing neuroimaging datasets, with public search, dataset pages, and download paths for web, S3, DataLad, and the OpenNeuro CLI.