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Figure 4 from: Rutkowski T, Maák I, Vepsäläinen K, Trigos-Peral G, Stephan W, Wojtaszyn G, Czechowski W (2019) Ants trapped for years in an old bunker; survival by cannibalism and eventual escape. Journal of Hymenoptera Research 72: 177-184. https://doi.org/10.3897/jhr.72.38972
Figure 4 On the earth mound in the bunker, the density of ants was high on the day when the boardwalk was put. Photo taken on 18.09.2016 (Wojciech Stephan).
Figure 2 from: Rutkowski T, Maák I, Vepsäläinen K, Trigos-Peral G, Stephan W, Wojtaszyn G, Czechowski W (2019) Ants trapped for years in an old bunker; survival by cannibalism and eventual escape. Journal of Hymenoptera Research 72: 177-184. https://doi.org/10.3897/jhr.72.38972
Figure 2 The upper part of the ant trail in Fig. 1; the ants (on the left) were unable to move along the ceiling and thus could not reach the outlet of the ventilation pipe. Photo taken on 18.09.2016 (Wojciech Stephan).
Figure 1 from: Rutkowski T, Maák I, Vepsäläinen K, Trigos-Peral G, Stephan W, Wojtaszyn G, Czechowski W (2019) Ants trapped for years in an old bunker; survival by cannibalism and eventual escape. Journal of Hymenoptera Research 72: 177-184. https://doi.org/10.3897/jhr.72.38972
Figure 1 A piece of board leaning against the wall initiated an ant trail leading upwards. Photo taken on 18.09.2016 (Wojciech Stephan).
Figure 3 from: Rutkowski T, Maák I, Vepsäläinen K, Trigos-Peral G, Stephan W, Wojtaszyn G, Czechowski W (2019) Ants trapped for years in an old bunker; survival by cannibalism and eventual escape. Journal of Hymenoptera Research 72: 177-184. https://doi.org/10.3897/jhr.72.38972
Figure 3 The boardwalk just assembled, to provide the ants a means to reach the ventilation pipe. Photo taken on 18.09.2016 (Wojciech Stephan).
Figure 5 in Effects of salinity tolerances on survival and life history of 2 cladocerans
Figure 5. Net reproductive rate (R) of S. mucronata under 0 different salt concentrations (with regression equations) in 8-day experiments (with 95% confidence interval).
Figure 3 in Effects of salinity tolerances on survival and life history of 2 cladocerans
Figure 3. Total progeny of S. mucronata under different salt concentrations (with regression equations) in 8-day experiments (with 95% confidence interval).
Figure 4 in The effect of streptomycin on survival, development, and some biochemical aspects of Drosophila melanogaster
Figure 4. Effects of dietary streptomycin on CAT enzyme activities in 3rd instar larvae, pupae, and adults of D. melanogaster. Bars represent the means of four replicates. Means followed by different letters are significantly different (p <0.05, LSD test).
Figure 1 in The effect of streptomycin on survival, development, and some biochemical aspects of Drosophila melanogaster
Figure 1. Effects of dietary streptomycin on MDA content in 3rd instar larvae, pupae, and adults of D. melanogaster. Bars represent the means of four replicates. Means followed by different letters are significantly different (p <0,05, LSD test).
Fig. 2. Plots A-B in Adaptations, life-history traits and ecological mechanisms of parasites to survive extremes and environmental unpredictability in the face of climate change
Fig. 2. Plots A-B. Hypothetical thermal curves of the free-living stages of two parasite populations with different thermal adaptation histories and similar thermal optimum (highest point in the curve). The blue curve represents a population adapted to a highly variable environment and the orange curve a population adapted to a less variable environment. The dashed black line is a hypothetical current mean temperature in the environment and the dashed grey line represents an increased mean temperature as a consequence of climate change. In plot A, the historical temperature sits close to the thermal optimum in both populations, and an increase in temperature results in a decrease in parasite performance, which is greater for the parasite adapted to the less variable environment. In plot B, the historical temperature is well below the thermal optimum of both parasites, and an increase in temperature results in improved performance for both parasites. In both scenarios, an increase in mean temperature causes a much higher relative change in performance in the population from the less variable environment as indicated in the difference in size among the shade areas. Plot C shows the hypothetical temperature and thermal development ranges for the free-living stages of parasites inhabiting three different latitudes. The temperature range increases with latitude but the development range of parasites does not because, although the thermal range in high latitudes is wider, a large portion of this range occurs <0 ◦C. While parasites from high latitudes might be highly resistant to freezing temperatures, they are also more vulnerable to high temperatures. (For interpretation of the references to colour in this figure legend, the reader is referred to the Web version of this article.)
Fig. 1 in Adaptations, life-history traits and ecological mechanisms of parasites to survive extremes and environmental unpredictability in the face of climate change
Fig. 1. Schematic of two types of life cycles of parasitic nematodes highlighting stage-specific interactions with the environment and hosts, and adaptations to cope with extreme environmental conditions: A) direct life cycle and B) specific indirect life cycle of protostrongylid parasites. In red are indicated the developmental stages of the parasite. The performance (e.g., survival rate, development rate) of developmental stages in the orange area is directly influenced by changes in environmental conditions. Developmental stages in light blue area are indirectly influenced by environmental conditions experienced by the definitive or intermediate hosts. The effect of the environment on the L3 of protostrongylids can be direct or indirect depending if the L3 migrates out of the intermediate host (direct) or if the L3 remains in the intermediate host (indirect). In the inner triangles, examples of stage-specific adaptations to cope with extremes are indicated. (For interpretation of the references to colour in this figure legend, the reader is referred to the Web version of this article.)
Dataset for: Heat domes increase vulnerability of native stingless bees by simultaneously weakening key survival traits
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ESMO-GROW checklist for the publication PD-1/PD-L1 blockade plus abnobaViscum® therapy is associated with improved survival in advanced or metastasized NSCLC patients, a real-world data registry study according to the ESMO-GROW criteria
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Dataset and Tables - DOCTORAL THESIS - EXTREMOPHILES IN ASTROBIOLOGY: CONTRIBUTIONS TO THE UNDERSTANDING OF BIOSIGNATURES PRODUCTION AND SURVIVAL MOLECULAR STRATEGIES
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Multi-Path Direct Current Spinal Stimulation Extended Survival in The SOD1-G93A Model of Amyotrophic Lateral Sclerosis
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Table 1 in Effect Of Parental Care On The Duration Of Larval Development And Offspring Survival In Nicrophorus Mexicanus Matthews (Coleoptera: Silphidae)
<p><b>Table 1.</b> Percentage of offspring survival in <i>N. mexicanus</i> in the three experimental groups. ni = number initial of larvae hatching for group. L1 = first instar larvae, L2 = second instar larvae, L3 = third instar larvae, pp = prepupae, p = pupae.</p><table><tbody><tr><th></th><th>Offspring Survival (%)</th><th>)</th></tr></tbody><tbody><tr><th>Treatment groups</th><td>ni</td><td>L1­L2 L3­PP PP­P</td></tr><tr><th>1) Control: parental care</th><td>390</td><td>97</td><td>90 70</td><td>70</td></tr><tr><th>2) With Brood­mass. Parents removed pre­hatching</th><td>468</td><td>95</td><td>89 60</td><td>60</td></tr><tr><th>3) Without care parental: with fresh meat, brood­mass</th><td>351</td><td>90</td><td>77 28</td><td>28</td></tr><tr><th>no present and parents removed pre­hatching</th><td></td><td></td><td></td><td></td></tr></tbody></table>
Table 2 in Effect Of Parental Care On The Duration Of Larval Development And Offspring Survival In Nicrophorus Mexicanus Matthews (Coleoptera: Silphidae)
<p><b>Table 2.</b> Significant differences in the variation the number of offspring surviving to adult emergence between three experimental groups.</p><table><tbody><tr><th>Compared groups</th><th></th><th></th><th></th><th></th><th></th><th></th></tr></tbody><tbody><tr><td>Difference</td><td>SE</td><td>Q</td><td>Q0.05 P <0­05</td></tr><tr><th>1 vs 3 1 vs 2</th><td>28.13–12.78 = 15.35 28.13–25.93 = 2.2</td><td>4.77 4.68</td><td>3.218 0.47</td><td>2.394 2.394</td><td>*</td></tr><tr><th>2 vs 3</th><td>25.93–12.78 = 13.15</td><td>4.77</td><td>2.75</td><td>2.394</td><td>*</td></tr><tr><th>Experimental groups</th><td>1</td><td></td><td>2</td><td>3</td><td></td></tr><tr><th>average ranges sample size</th><td>28.13 15</td><td></td><td>25.93 14</td><td>12.78 15</td><td></td></tr></tbody></table><p>* indicate a significant difference.</p>
Heterotrophy in parental coral colonies enhances larval survival independently of heat stress
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Data from: Can recombinant human thrombomodulin increase survival among patients with severe septic-induced disseminated intravascular coagulation: a single-centre, open-label, randomised controlled trial
Objective: To determine whether treatment with recombinant human thrombomodulin (rhTM) increases survival among severe septic patients with sepsis-induced disseminated intravascular coagulation (DIC) Design: Single-center, open-label, randomized controlled trial Setting: Single tertiary hospital Participant: 92 severe septic patients with sepsis-induced DIC Interventions: Patients with DIC scores ≥4, as defined by the Japanese Association of Acute Medicine, were diagnosed with DIC. Randomization was performed by the envelope method. The treatment group (rhTM group, n = 47) was intravenously treated with rhTM within 24 h of admission (day 0), and the control group (n = 45) did not receive any anti-coagulants, except in cases of deep venous thrombosis and pulmonary embolism. Primary and secondary measurements: Data were collected on days 0 (admission), 1, 2, 3, 5, 7, and 10. The primary outcome was survival at 28 and 90 days. The secondary endpoints comprised changes in DIC scores, platelet counts, D-dimer, antithrombin III (ATIII), and C-reactive protein (CRP) levels, and Sequential Organ Failure Assessment (SOFA) scores. All analyses were conducted on an intent-to-treat basis. Main Results: The 28-day survival rates were 84 and 83% in the control and rhTM groups, respectively (p = 0.745, log rank test). The 90-day survival rates were 73% and 72% in the control and rhTM groups, respectively (p = 0.94, log rank test). Meanwhile, the rates of recovery from DIC (<4) were significantly higher in the rhTM group than in the control group (p = 0.001, log rank test). Relative change from baseline of D-dimer levels were significantly lower in the rhTM group than in the control group, on day 3 and 5. Conclusion: rhTM treatment decreased D-dimer levels and facilitated DIC recovery in severe septic patients with sepsis-induced DIC. However, the treatment did not improve survival in this cohort.
Gene alterations, clinical informations and survival data of gliomas with/without corpus callosum involvement
<p>The raw dataset of this study contained the gene alterations, clinical informations and survival data of glioma patients. Gene alterations data were obtained by Next-generation DNA sequencing of glioma samples of patients. Clinical informations were obtained from Electronic medical records and follow-up. The survival data were obtained by follow-up of patients. In order to explore gene alterations and their correlation with the survival of glioblastoma (GBM) of corpus callosum (CC) involvement (ccGBM), this dataset was analyzed in detail. A total of<b> </b>30 ccGBM and 88 non-ccGBM were finally included. The ccGBM had higher incidence of <i>PDGFRA </i>(33.3% versus 9.1%, P=0.004) alterations than non-ccGBM. <i>PDGFRA </i>amplification (<i>PDGFRAamp</i>, 33.3% versus 9.1%, P=0.004) and missense mutation (<i>PDGFRAmut</i>, 20.0% versus 3.4%, P=0.011) both had higher incidence in ccGBM than in non-ccGBM. <i>PDGFRA </i>alteration was significantly associated with the occurrence of ccGBM (OR=4.91 [95%CI: 1.55-15.52], P=0.007). The ccGBM of <i>PDGFRAamp </i>achieved shorter median PFS (8.6 versus 13.5 months, P=0.025) and OS (12.4 versus 17.9 months, P=0.022) than non-ccGBM of <i>PDGFRAnon-amp</i>. The ccGBM of <i>PDGFRAamp</i> combined with <i>PDGFRAmut</i> (<i>PDGFRAamp-mut</i>)<i> </i>had shorter median PFS (7.6 versus 8.9 months, P=0.022) and OS (9.6 versus 17.8 months, P=0.006) than non-ccGBM of <i>PDGFRA</i> wild type and non-amplification (<i>PDGFRA-w, non-amp</i>). Compared to ccGBM of <i>PDGFRA-w, non-amp</i>, the ccGBM of <i>PDGFRAamp</i> and <i>PDGFRAamp-mut </i>both had shorter median PFS and OS (P<0.05). Hazard ratios (HRs) of <i>PDGFRAamp</i> for PFS and OS of ccGBM were 3.08 (95%CI: 1.02-9.35, P=0.047) and 5.07 (95%CI: 1.52-16.89, P=0.008) respectively, HRs of <i>PDGFRAamp-mut </i>for PFS and OS were 13.16 (95%CI: 3.19-54.40, P<0.001) and 16.36 (95%CI: 2.66-100.70, P=0.003) respectively. The <i>PDGFRA </i>alterations is significantly associated with the occurrence and poor prognosis of ccGBM.</p>
Data from: Delayed transmission selects for increased survival of vesicular stomatitis virus
Life-history theory predicts that traits for survival and reproduction cannot be simultaneous maximized in evolving populations. For this reason, in obligate parasites such as infectious viruses, selection for improved between-host survival during transmission may lead to evolution of decreased within-host reproduction. We tested this idea using experimental evolution of RNA virus populations, passaged under differing transmission times in the laboratory. A single ancestral genotype of Vesicular Stomatitis Virus (VSV), a negative-sense RNA Rhabdovirus, was used to found multiple virus lineages evolved in either ordinary 24-hour cell-culture passage, or in delayed passages of 48 hours. After 30 passages (120 generations of viral evolution), we observed that delayed transmission selected for improved extracellular survival, which traded-off with lowered viral fecundity (slower exponential population growth, and smaller mean plaque size). To further examine the confirmed evolutionary trade-off, we obtained consensus whole-genome sequences of evolved virus populations, to infer phenotype-genotype associations. Results implied that increased virus survival did not occur via convergence; rather, improved virion stability was gained via independent mutations in various VSV structural proteins. Our study suggests that RNA viruses can evolve different molecular solutions for enhanced survival despite their limited genetic architecture, but suffer generalized reproductive trade-offs that limit overall fitness gains.
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Allen Brain Atlas
Allen Brain Atlas is an Allen Institute collection of brain map atlases, datasets, APIs, and analysis tools covering mouse, human, and non-human primate brain resources.
Annotated Behaviour and Observability Dataset (ABODe)
ABODe is a University of Edinburgh DataShare dataset for behavior classification in group-housed mice using home-cage video, identities, bounding boxes, ground-plate positions, and annotator labels.
DANDI Archive for NWB datasets
DANDI is a BRAIN Initiative archive for publishing and sharing neurophysiology data, including electrophysiology, optophysiology, and behavioral data packaged as NWB and related standards.
International Brain Laboratory public data
The International Brain Laboratory public data releases expose standardized mouse decision-making experiments, including Neuropixels recordings, widefield calcium imaging, behavior, and session metadata accessed through the ONE API.
OpenNeuro
OpenNeuro is a free, open platform for sharing neuroimaging datasets, with public search, dataset pages, and download paths for web, S3, DataLad, and the OpenNeuro CLI.