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6 results for “Globodera pallida”
Figure 2 in First report of morphological and molecular characterization of Moroccan populations of Globodera pallida
Figure 2: Photomicrographs of morphological characterization of cyst, egg, and second-stage juvenile. A, B: Cyst of GlOBOdeRa pallida, C: Vulva (v), anus (a), and cuticular ridges (r) of cyst, D, E: Stylet knob shape of J2, F: Juvenile tail.
Figure 3 in First report of morphological and molecular characterization of Moroccan populations of Globodera pallida
Figure 3: Amplified PCR products from GlOBOdeRa Spp. digested by three enzymes AluI, MboI, and RsaI. A: Amplified PCR products, B: AluI, C: RsaI, D: MboI, MW: molecular weight markers (1 kb), NC: negative control, PC: undigested DNA, S1-S2: Eastern region samples, S3-S4: Western region samples (Gharb), S5-S6: Doukkala region samples.
Figure 1 in First report of morphological and molecular characterization of Moroccan populations of Globodera pallida
Figure 1: Sampled areas and regions infested with GlOBOdeRa pallida in Morocco.
Data from: Exploring the causes of small effective population sizes in cyst nematodes using artificial Globodera pallida populations
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Data from: Adaptation to resistant hosts increases fitness on susceptible hosts in the plant parasitic nematode Globodera pallida
Trade-offs between virulence (defined as the ability to infect a resistant host) and life-history traits are of particular interest in plant pathogens for durable management of plant resistances. Adaptation to plant resistances (i.e., virulence acquisition) is indeed expected to be associated with a fitness cost on susceptible hosts. Here, we investigated whether life-history traits involved in the fitness of the potato cyst nematode Globodera pallida are affected in a virulent lineage compared to an avirulent one. Both lineages were obtained from the same natural population through experimental evolution on resistant and susceptible hosts, respectively. Unexpectedly, we found that virulent lineages were more fit than avirulent lineages on susceptible hosts: they produced bigger cysts, containing more larvae and hatching faster. We thus discuss possible reasons explaining why virulence did not spread into natural G. pallida populations.
Data from: Adaptation to resistant hosts increases fitness on susceptible hosts in the plant parasitic nematode Globodera pallida
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