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123 results for “Mildew”
Species‑specific influence of powdery mildew mycelium on the efficiency of PM accumulation by urban greenery - Data
<p>Dataset of article: Przybysz, A., Nawrocki, A., Mirzwa-Mróz, E. <em>et al.</em> Species-specific influence of powdery mildew mycelium on the efficiency of PM accumulation by urban greenery. <em>Environ Sci Pollut Res</em> (2023). https://doi.org/10.1007/s11356-023-28371-6</p>
Supporting data and code for: Demographic and genetic impacts of powdery mildew in a young oak cohort
<p>This is a new release following the submission of the related PCI recommended manuscript to the <em>Annals of Forest Science</em> journal. It contains the necessary scripts to produce most of the analyses and figures of the manuscript. Apart from minor modifications following the recommendation in '<em>PCI Forest and Wood Sciences</em>', the main change is the addition of an extra dataset "Data_S2.txt" to the additional datasets. This dataset was previously included as a table in the 'supplementary material' file.</p>
Trinity assembly of Downy mildew of Grapevine lesion samples DMG-C
<p>De novo Trinity assembly from RNAseq</p>
Supplementary file for the manuscript entitled: Demographic and genetic impacts of powdery mildew in a young oak cohort
<p>Barres et al 2023 Supplementary material-vf.pdf: supplementary material file for the related article</p>
Mildew ratings and yields of winter wheat and spring oat varieties on NIAB and AHDB Recommended Lists, 1972-2022
<p>Data on powdery mildew ratings and yields in fungicide-treated trials relative to controls, for winter wheat and spring oat varieties on UK Recommended Lists from 1972 to 2022. These data are used in the graphs in Figure 1 of Brown & Wulff (2022) 'Diversifying the menu for crop powdery mildew resistance', Cell, DOI https://doi.org/10.1016/j.cell.2022.02.003. Data are compiled from published information. (c) NIAB for data from 1972 to 2001. (c) Agriculture and Horticulture Development Board </p>
Pangenome graph analysis reveals extensive effector copy-number variation in spinach downy mildew
<p>Data produced for the comparison of six <em>Peronospora effusa</em> isolates. For each isolate, we provide the genome assemblies, gene and repeat annotation, effector clustering, and gene variation. Additionally, we provide the repeat library that was used to annotate the transposable elements for each isolate and the pangenome graph.</p> <p>DOI: https://doi.org/10.1101/2024.05.30.596583 </p>
Trinity assembly of Powdery mildew of Grapevine lesion samples from Italy PMG
<p>De novo trinity assembly powdery mildew of grapevine from NGS data (RNAseq)</p>
Trinity assembly of Downy mildew of Grapevine lesion samples DMG-G
<p>De Novo trinity assembly RNAseq NGS-Data</p>
Trinity assembly of Downy mildew of Grapevine lesion samples DMG-A
<p>Tirnity Assembly of NGS reads </p>
Trinity assembly of Downy mildew of Lettuce lesion samples
<p>De novo trinity assembly of RNAseq NGS data</p>
Barley endosomal MONENSIN SENSITIVITY1 is a target of the powdery mildew effector CSEP0162 and plays a role in plant immunity
<p><span>Encasements formed around haustoria and biotrophic hyphae as well as hypersensitive reaction (HR) cell death are essential plant immune responses to filamentous pathogens. In this study we examine the components that may contribute to the absence of these responses in susceptible barley attacked by the powdery mildew fungus. We find that the effector CSEP0162 from this pathogen targets plant MONENSIN SENSITIVITY1 (MON1), which is important for the fusion of multivesicular bodies to their target membranes. Overexpression of CSEP0162 and silencing of barley MON1 both inhibit encasement formation. We find that the Arabidopsis ecotype No-0 has resistance to powdery mildew, and that this is partially dependent on MON1. Surprisingly, we find the MON1-dependent resistance in No-0 not only includes an encasement response, but also an effective HR. Similarly, silencing of MON1 in barley also blocks Mla3-mediated HR-based powdery mildew resistance. Our results indicate that MON1 is a vital plant immunity component, and we speculate that the barley powdery mildew fungus introduces the effector CSEP0162 to target MON1 and hence reduces encasement formation and HR.</span></p>
Barley endosomal MONENSIN SENSITIVITY1 is a target of the powdery mildew effector CSEP0162 and plays a role in plant immunity
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Inhibition mechanism and antibacterial activity of natural antibacterial agent citral on bamboo mold and its anti- mildew effect on bamboo
<p><span><span>Bamboo, a natural material, has been widely used in the fields of decoration, architecture and furniture. However, bamboo is easy to mildew and lose its use value. In this paper, inhibition mechanism and antibacterial activity of natural antibacterial agent citral on bamboo mold and its anti- mildew effect on bamboo were studied. The results showed that citral could change the shape of mycelium, destroy the integrity of mycelium structure, cell wall, and cell membrane structure, thereby causing leakage of nucleic acid, protein, and other substances in the cell, as well as destroy the pH balance of the inside and outside of the cell, to inhibit or kill mold. When the concentration of citral is 100mg/mL, the antibacterial rates of citral against <i>Penicillium citrinum </i>(PC), <i>Trichoderma viride</i> (TV), <i>Aspergillus niger</i> (AN) and a hybrid fungi group comprising PC, TV, and AN (Hun) were more than 100%. However, compared with the direct effect of citral on mold, the antibacterial property of bamboo treated with citral was significantly reduced, the mildew proof effect can be achieved only if the concentration of citral to treat bamboo is increased to more than 2 times of the concentration of citral directly acting on mold.</span></span></p>
Data from: Soft selective sweeps in fungicide resistance evolution: recurrent mutations without fitness costs in grapevine downy mildew
Adaptation produces hard or soft selective sweeps depending on the supply of adaptive genetic polymorphism. The evolution of pesticide resistance in parasites is a striking example of rapid adaptation that can shed light on selection processes. Plasmopara viticola, which causes grapevine downy mildew, forms large populations, in which resistance has rapidly evolved due to excessive fungicide use. We investigated the pathways by which fungicide resistance has evolved in this plant pathogen, to determine whether hard or soft selective sweeps were involved. An analysis of nucleotide polymorphism in 108 field isolates from the Bordeaux region revealed recurrent mutations of cytb and CesA3 conferring resistance to quinone outside inhibiting (QoI) and carboxylic acid amide (CAA) fungicides, respectively. Higher levels of genetic differentiation were observed for nucleotide positions involved in resistance than for neutral microsatellites, consistent with local adaptation of the pathogen to fungicide treatments. No hitchhiking was found between selected sites and neighbouring polymorphisms in cytb and CesA3, confirming multiple origins of resistance alleles. We assessed resistance costs, by evaluating the fitness of the 108 isolates through measurements of multiple quantitative pathogenicity traits under controlled conditions. No significant differences were found between sensitive and resistant isolates, suggesting that fitness costs may be absent or negligible. Our results indicate that the rapid evolution of fungicide resistance in P. viticola has involved a soft sweep.
Data from: Adaptation of a plant pathogen to partial host resistance: selection for greater aggressiveness in grapevine downy mildew
An understanding of the evolution of pathogen quantitative traits in response to host selective pressures is essential for the development of durable management strategies for resistant crops. However, we still lack experimental data on the effects of partial host resistance on multiple phenotypic traits (aggressiveness) and evolutionary strategies in pathogens. We performed a cross-inoculation experiment with four grapevine hosts and 103 isolates of grapevine downy mildew (Plasmopara viticola) sampled from susceptible and partially resistant grapevine varieties. We analysed the neutral and adaptive genetic differentiation of five quantitative traits relating to pathogen transmission. Isolates from resistant hosts were more aggressive than isolates from susceptible hosts, as they had a shorter latency period and higher levels of spore production. This pattern of adaptation contrasted with the lack of neutral genetic differentiation, providing evidence for directional selection. No specificity for a particular host variety was detected. Adapted isolates had traits that were advantageous on all resistant varieties. There was no fitness cost associated with this genetic adaptation, but several trade-offs between pathogen traits were observed. These results should improve the accuracy of prediction of fitness trajectories for this biotrophic pathogen, an essential element for the modelling of durable deployment strategies for resistant varieties.
Data from: Daytime solar heating controls downy mildew Peronospora belbahrii in sweet basil
The biotrophic oomycete Peronospora belbahrii causes a devastating downy mildew disease in sweet basil. Due to the lack of resistant cultivars current control measures rely heavily on fungicides. However, resistance to fungicides and strict regulation on their deployment greatly restrict their use. Here we report on a 'green' method to control this disease. Growth chamber studies showed that P. belbahrii could hardly withstand exposure to high temperatures; exposure of spores, infected leaves, or infected plants to 35-45°C for 6-9 hours suppressed its survival. Therefore, daytime solar heating was employed in the field to control the downy mildew disease it causes in basil. Covering growth houses of sweet basil already infected with downy mildew with transparent infra-red-impermeable, transparent polyethylene sheets raised the daily maximal temperature during sunny hours by 11-22°C reaching 40-58°C (greenhouse effect). Such coverage, applied for a few hours during 1-3 consecutive days, had a detrimental effect on the survival of P. belbahrii: killing the pathogen and/or suppressing disease progress while enhancing growth of the host basil plants.
The genome of Vitis vinifera cv. Mgaloblishvili reveals resistance and susceptibility factors to downy mildew in the Rpv29 and Rpv31 loci
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FIGURE 3 in A new species of Podosphaera sect. Sphaerotheca subsect. Sphaerotheca from India-first report of powdery mildew causing wilting and ultimately death of leaves of Filipendula vestita
FIGURE 3. Microphotographs of fruiting bodies of Podosphaera filipendulensis (AMH 9934, holotype) a. Initial stage of developing cleistothecium (homogenious cells). b. Mature cleistothecium. c. Pseudoparenchymatous nature of wall of cleistothecium. d–g. Ruptured cleistothecia each releasing an ascus. h, i. Appendages on the surface of cleistothecia. Scale bars: a = 25 μm, b = 40 μm, c = 20 μm, d = 50 μm, e = 40 μm, f, g = 30 μm, h, i = 20 μm.
FIGURE 4 in A new species of Podosphaera sect. Sphaerotheca subsect. Sphaerotheca from India-first report of powdery mildew causing wilting and ultimately death of leaves of Filipendula vestita
FIGURE 4. Microphotographs of asci of Podosphaera filipendulensis (AMH 9934, holotype) a, b. Fully mature ascus. c, d. Ruptured asci releasing ascospores (arrows for ruptured oculi). e. Ascospores. Scale bars: a = 25 μm, b = 30 μm, c, d = 25 μm, e = 15 μm.
FIGURE 2 in A new species of Podosphaera sect. Sphaerotheca subsect. Sphaerotheca from India-first report of powdery mildew causing wilting and ultimately death of leaves of Filipendula vestita
FIGURE 2. Photographs of fruiting body of Podosphaera filipendulensis on the surface of infected leaf (AMH 9934, holotype) a. Cleistothecia (arrows) in between leaf trichomes. b–f. Cleistothecia with appendages. Scale bars: b, c = 60 μm, d = 30 μm, e = 25 μm, f = 30 μm.
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Allen Brain Atlas
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International Brain Laboratory public data
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OpenNeuro
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