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Fig. 2. Unsporulated T. gondii oocysts, with a in Exploring the epidemiological role of the Eurasian lynx (Lynx lynx) in the life cycle of Toxoplasma gondii

Fig. 2. Unsporulated T. gondii oocysts, with a diameter of 10–12 μm, after flotation from a faecal sample of a juvenile lynx (left) (ID W20_8385). T. gondii development stage (meront, arrow) in a histological section of small intestine of a lynx (right) (ID W21_4446).

opencc-by-4.0Apr 2014View details →

Fig. 1 in Use of filter papers to determine seroprevalence of Toxoplasma gondii among hunted ungulates in remote Peruvian Amazon

Fig. 1. Animals were hunted in the area surrounding the community of Nueva Esperanza, the only permanent settlement located along the Yavarí-Mirín River. The study site's geographic remoteness reflects extremely limited contact with domestic animals and humans, which eliminates the effect of spillover from domestic animals as a potential source of infection for wildlife. The data from this study are likely to shed light on the maintenance of T. gondii in its natural environment.

Fig. 8 Urine miRNA profile analysis among different groups. a in A combined miRNA-piRNA signature in the serum and urine of rabbits infected with ToxoplaSMa gondii oocysts

Fig. 8 Urine miRNA profile analysis among different groups. a The volcano plot shows the individual statistically significant miRNA between acutely infected rabbits and control rabbits. In this plot, the x-axis is log2 fold-change, which shows the direction of the change (negative scale is decrease and positive scale is increase) in the levels of miRNA expression, while the y-axis is the –log10 FDR, which shows the significance of the change. b The volcano plot shows the individual statistically significant miRNA between chronically infected rabbits and control rabbits. c The volcano plot shows the individual statistically significant miRNA between acutely infected rabbits and chronically infected rabbits. d Venn diagram shows number of differentially expressed miRNA among different comparison pairs

Fig. 10 in A combined miRNA-piRNA signature in the serum and urine of rabbits infected with ToxoplaSMa gondii oocysts

Fig. 10 Venn diagrams showing the common and unique DE miRNAs (a) and DE piRNAs (b) in both serum and urine between the acutely and chronically infected rabbits versus uninfected rabbits

Fig. 5 in A combined miRNA-piRNA signature in the serum and urine of rabbits infected with ToxoplaSMa gondii oocysts

Fig. 5 Global view of temporal sRNA expression profiles in rabbit urine during T. gondii infection. a The sRNA correlation heatmap of sample clustering. b Principal component analysis of all identified urine sRNAs. c Unsupervised hierarchical clustering of sRNA profiling data. sRNA intensity is normalized so that blue represents low intensity and yellow represents high intensity. Columns were hierarchically clustered based on a complete linkage using Pearson correlation coefficients as the distance measure. Sample groups are acutely infected rabbits, chronically infected rabbits and uninfected control rabbits, which are labeled as AI, CI and Con, respectively

Fig. 4 in A combined miRNA-piRNA signature in the serum and urine of rabbits infected with ToxoplaSMa gondii oocysts

Fig. 4 First nucleotide bias of obtained small RNA in urine samples of rabbits. a First nucleotide bias of known miRNAs in rabbit urine. b First nucleotide bias of predicted miRNAs in rabbit urine. c First nucleotide bias of predicted piRNAs in rabbit urine

Fig. 1 in A combined miRNA-piRNA signature in the serum and urine of rabbits infected with ToxoplaSMa gondii oocysts

Fig. 1 Histological features of spleen section from healthy control rabbits and rabbits experimentally infected with Toxoplasma gondii. Images showing the H&E-stained spleen section at 100× (a, b). a Spleen section from healthy, uninfected rabbit. The structures of white pulp (WP) and red pulp (RP) were clearly identified with normal cell density. b Spleen section from a rabbit with acute T. gondii infection. The number and dimension of splenic nodule are increased, and more plasma cells are observed in the splenic cord (black triangle) of red pulp. Note that granulomas are present (big black arrow). Hemosiderin deposition (small black arrow) indicated red blood cell destruction. CA central arteriole. Scale bar = 100 μm

Fig. 6 Serum miRNA profile analysis among different groups. a in A combined miRNA-piRNA signature in the serum and urine of rabbits infected with ToxoplaSMa gondii oocysts

Fig. 6 Serum miRNA profile analysis among different groups. a The volcano plot shows the individual statistically significant miRNA between acutely infected group and control group. In this plot, the x-axis is log2 fold-change, which shows the direction of the change (negative scale is decrease and positive scale is increase) in the levels of miRNA expression, while the y-axis is the –log10 FDR, which shows the significance of the change. b The volcano plot shows the individual statistically significant miRNA between chronically infected rabbits and control rabbits. c The volcano plot shows the individual statistically significant miRNA between acutely infected rabbits and chronically infected rabbits. d Venn diagram shows number of differentially expressed miRNA among different comparison pairs. FDR represents false discovery rate

Fig. 7 Serum piRNA profile analysis among different groups. a in A combined miRNA-piRNA signature in the serum and urine of rabbits infected with ToxoplaSMa gondii oocysts

Fig. 7 Serum piRNA profile analysis among different groups. a The volcano plot shows the individual statistically significant piRNA between acutely infected rabbits and control rabbits. In this plot, the x-axis is log2 fold-change, which shows the direction of the change (negative scale is decrease and positive scale is increase) in the levels of piRNA expression, while the y-axis is the –log10 FDR, which shows the significance of the change. b The volcano plot shows the individual statistically significant piRNA between chronically infected rabbits and control rabbits. c The volcano plot shows the individual statistically significant piRNA between acutely infected rabbits and chronically infected rabbits. d Venn diagram shows number of differentially expressed piRNA among different comparison pairs. FDR represents false discovery rate

Fig. 3 in A combined miRNA-piRNA signature in the serum and urine of rabbits infected with ToxoplaSMa gondii oocysts

Fig. 3 Global view of temporal sRNA expression profiles in rabbit serum during T. gondii infection. a The sRNA correlation heatmap of sample clustering. b Principal component analysis of all identified serum sRNA. c Unsupervised hierarchical clustering of sRNA profiling data. sRNA intensity is normalized so that blue represents low intensity and yellow represents high intensity. Columns are hierarchically clustered based on a complete linkage using Pearson correlation coefficients as the distance measure. Sample groups including acutely infected rabbits, chronically infected rabbits and uninfected control rabbits are labeled as AI, CI and Con, respectively

Fig. 2 in A combined miRNA-piRNA signature in the serum and urine of rabbits infected with ToxoplaSMa gondii oocysts

Fig. 2 First nucleotide bias of obtained small RNA in serum samples of rabbits. a First nucleotide bias of known miRNAs in rabbit serum. b First nucleotide bias of predicted miRNAs in rabbit serum. c First nucleotide bias of predicted piRNAs in rabbit serum

Fig. 9 Urine piRNA profile analysis among different groups. a in A combined miRNA-piRNA signature in the serum and urine of rabbits infected with ToxoplaSMa gondii oocysts

Fig. 9 Urine piRNA profile analysis among different groups. a The volcano plot shows the individual statistically significant piRNA between acutely infected group and control group. In this plot, the x-axis is log2 fold-change, which shows the direction of the change (negative scale is decrease and positive scale is increase) in the levels of piRNA expression, while the y-axis is the –log10 FDR, which shows the significance of the change. b The volcano plot shows the individual statistically significant piRNA between chronically infected group and control group. c The volcano plot shows the individual statistically significant piRNA between acutely infected group and chronically infected group. d Venn diagram shows number of differentially expressed piRNA among different comparison pairs

opencc-by-4.0Jul 2017View details →

Fig. 1 in First Toxoplasma gondii isolate from an aborted foetus of European bison (Bison bonasus bonasus L.)

Fig. 1 Toxoplasma gondii tachyzoites successfully propagated after several passages in Vero cell cultures

opencc-by-4.0May 2009View details →

Fig. 4 in Changing climate-changing pathogens: Toxoplasma gondii in North-Western Europe

Fig. 4 Expected increases in T. gondii prevalence in NorthWestern Europe towards 2069 based upon the combination of climatic conditions from Figs. 2 and 3. The dotted bright green areas indicate a small increase in T. gondii prevalence as a result of climatic change, pink areas a limited increase, and red areas a substantial increase

opencc-by-4.0May 2009View details →

Fig. 2 in Changing climate-changing pathogens: Toxoplasma gondii in North-Western Europe

Fig. 2 Total precipitation in North-Western Europe as calculated by the CCSR (Center for Climate System Research, University of Tokyo) and NIES (National Institute for Environmental Studies) model under a SRES A1 scenario. Presented is the total mean precipitation in period from 1970 to 1999 (a), and the projected total mean precipitation from 2010 to 2039 (b) and 2040–2069 (c). Figures obtained from www.ipcc-data.org

Figure 5 in Toxoplasma gondii in beef consumed in France: regional variation in seroprevalence and parasite isolation

Figure 5. Comparison of observed values versus predicted values by the final model according to age. The observed values are in green bars, while for the predicted values the red point represents the mean prediction and the blue segment the 95% confidence interval of the prediction. The number above the blue segment is the number of observations for this particular class of age.

Figure 2 in Toxoplasma gondii in beef consumed in France: regional variation in seroprevalence and parasite isolation

Figure 2. Geographical variation of Toxoplasma gondii seroprevalence of French bovine samples according to the area of slaughtering and to age categories: (A) calves; (B) adults; (C) bovines overall (calves and adults). The numbers represent the number of samples collected for each region.

Figure 1 in Toxoplasma gondii in beef consumed in France: regional variation in seroprevalence and parasite isolation

Figure 1. (A) Map of French beef production according to the Ministry of Agriculture database. The colour gradient represents the number of cattle slaughtered in 2007. (B) The numbers represent the number of slaughterhouses per region that were included in the cross-sectional survey of Toxoplasma gondii presence in beef produced in France.

Figure 4 in Toxoplasma gondii in beef consumed in France: regional variation in seroprevalence and parasite isolation

Figure 4. Seroprevalence of Toxoplasma gondii infection in bovines of French origin (adults + calves) accordingly to the age and the titer (6; 10; 25; 50; 100; 200).