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68 results for “cause of death”
BeBOD estimates of mortality and years of life lost for 131 causes of death, 2004-2021
<p><strong>Belgian National Burden of Disease Study</strong></p> <p><strong>Estimates of the fatal burden of disease</strong></p> <p><em>Causes of death</em></p> <p>Our estimates are based on the official causes of death database compiled by <a href="https://statbel.fgov.be/en/themes/population/mortality-life-expectancy-and-causes-death/causes-death">Statbel</a>. We first map the ICD-10 codes of the underlying causes of death to the Global Burden of Disease cause list, consisting of 131 unique causes of deaths. Next, we perform a probabilistic redistribution of ill-defined deaths to specific causes, to obtain a specific cause of death for each deceased person.</p> <p><em>Years of Life Lost</em></p> <p>In addition to counting the number of deaths, we also calculate Years of Life Lost (YLLs) as a measure of premature mortality. YLLs correspond to the life expectancy at the age of death, and therefore give a higher weight to deaths occurring at younger ages. We calculate YLLs using the Global Burden of Disease reference life table, which represents the theoretical maximum number of years that people can expect to live.</p> <p><em>More information</em></p> <p>For additional background on BeBOD, please visit <a href="https://www.sciensano.be/en/projects/belgian-national-burden-disease-study">https://www.sciensano.be/en/projects/belgian-national-burden-disease-study</a>.</p> <p>Explore the estimates via <a href="https://burden.sciensano.be/shiny/mortality">https://burden.sciensano.be/shiny/mortality</a>.</p>
BeBOD estimates of mortality and years of life lost for 131 causes of death, 2004-2020
<p><strong>Belgian National Burden of Disease Study</strong></p> <p><strong>Estimates of the fatal burden of disease</strong></p> <p><em>Causes of death</em></p> <p>Our estimates are based on the official causes of death database compiled by <a href="https://statbel.fgov.be/en/themes/population/mortality-life-expectancy-and-causes-death/causes-death">Statbel</a>. We first map the ICD-10 codes of the underlying causes of death to the Global Burden of Disease cause list, consisting of 131 unique causes of deaths. Next, we perform a probabilistic redistribution of ill-defined deaths to specific causes, to obtain a specific cause of death for each deceased person.</p> <p><em>Years of Life Lost</em></p> <p>In addition to counting the number of deaths, we also calculate Years of Life Lost (YLLs) as a measure of premature mortality. YLLs correspond to the life expectancy at the age of death, and therefore give a higher weight to deaths occurring at younger ages. We calculate YLLs using the Global Burden of Disease reference life table, which represents the theoretical maximum number of years that people can expect to live.</p> <p><em>More information</em></p> <p>For additional background on BeBOD, please visit <a href="https://www.sciensano.be/en/projects/belgian-national-burden-disease-study">https://www.sciensano.be/en/projects/belgian-national-burden-disease-study</a>.</p> <p>Explore the estimates via <a href="https://burden.sciensano.be/shiny/mortality">https://burden.sciensano.be/shiny/mortality</a>.</p>
Fig. 1 in Air sac trematodes: Morishitium polonicum as a newly identified cause of death in the common blackbird (Turdus merula)
Fig. 1. Necropsy of Turdus merula, female. Gross lesions are represented by heavy parasite colonization of coelomic cavity. A. Many trematodes are clearly seen on different serosal membranes. Note the presence of parasites on the liver serosa, air sacs and pericardium (arrow). B. After removal of all the organs of the gastroenteric apparatus, an involvement of kidney and lungs serosa is also evident. Note the presence of an inflammatory focus with exudate at the periphery of the left lung (arrowhead).
Fig. 4 in Air sac trematodes: Morishitium polonicum as a newly identified cause of death in the common blackbird (Turdus merula)
Fig. 4. Maximum likelihood analyses of sequences of mitochondrial DNA loci of the newly isolated Morishitium polonicum and previously sequenced Cyclocoelidae. (A) CO1, (B) ND1. The bars indicate the number of substitutions per nucleotide.
Figure 2 in Identification of causes of death of Baikal seal (Pusa sibirica Gmelin, 1788)
Figure 2. Myocardia in the Baikal seal, hematoxylin–Rego. Magnification 200×.
Death from causes other than Uveal Melanoma-Related in Patients with Uveal Melanoma and its associated cancers
<p>Patients with uveal melanoma die from cancer or other causes. Here, we list the other causes based on SEER 9.</p> <p>The record will be updated to refer to include the full article URL once published. There you can read the full methods.</p>
Data from: Early-life disease exposure and associations with adult survival, cause of death, and reproductive success in preindustrial humans
A leading hypothesis proposes that increased human life span since 1850 has resulted from decreased exposure to childhood infections, which has reduced chronic inflammation and later-life mortality rates, particularly from cardiovascular disease, stroke, and cancer. Early-life cohort mortality rate often predicts later-life survival in humans, but such associations could arise from factors other than disease exposure. Additionally, the impact of early-life disease exposure on reproduction remains unknown, and thus previous work ignores a major component of fitness through which selection acts upon life-history strategy. We collected data from seven 18th- and 19th-century Finnish populations experiencing naturally varying mortality and fertility levels. We quantified early-life disease exposure as the detrended child mortality rate from infectious diseases during an individual's first 5 y, controlling for important social factors. We found no support for an association between early-life disease exposure and all-cause mortality risk after age 15 or 50. We also found no link between early-life disease exposure and probability of death specifically from cardiovascular disease, stroke, or cancer. Independent of survival, there was no evidence to support associations between early-life disease exposure and any of several aspects of reproductive performance, including lifetime reproductive success and age at first birth, in either males or females. Our results do not support the prevailing assertion that exposure to infectious diseases in early life has long-lasting associations with later-life all-cause mortality risk or mortality putatively linked to chronic inflammation. Variation in adulthood conditions could therefore be the most likely source of recent increases in adult life span.
FIGURE 3 in A new species of Podosphaera sect. Sphaerotheca subsect. Sphaerotheca from India-first report of powdery mildew causing wilting and ultimately death of leaves of Filipendula vestita
FIGURE 3. Microphotographs of fruiting bodies of Podosphaera filipendulensis (AMH 9934, holotype) a. Initial stage of developing cleistothecium (homogenious cells). b. Mature cleistothecium. c. Pseudoparenchymatous nature of wall of cleistothecium. d–g. Ruptured cleistothecia each releasing an ascus. h, i. Appendages on the surface of cleistothecia. Scale bars: a = 25 μm, b = 40 μm, c = 20 μm, d = 50 μm, e = 40 μm, f, g = 30 μm, h, i = 20 μm.
FIGURE 4 in A new species of Podosphaera sect. Sphaerotheca subsect. Sphaerotheca from India-first report of powdery mildew causing wilting and ultimately death of leaves of Filipendula vestita
FIGURE 4. Microphotographs of asci of Podosphaera filipendulensis (AMH 9934, holotype) a, b. Fully mature ascus. c, d. Ruptured asci releasing ascospores (arrows for ruptured oculi). e. Ascospores. Scale bars: a = 25 μm, b = 30 μm, c, d = 25 μm, e = 15 μm.
FIGURE 2 in A new species of Podosphaera sect. Sphaerotheca subsect. Sphaerotheca from India-first report of powdery mildew causing wilting and ultimately death of leaves of Filipendula vestita
FIGURE 2. Photographs of fruiting body of Podosphaera filipendulensis on the surface of infected leaf (AMH 9934, holotype) a. Cleistothecia (arrows) in between leaf trichomes. b–f. Cleistothecia with appendages. Scale bars: b, c = 60 μm, d = 30 μm, e = 25 μm, f = 30 μm.
FIGURE 1 in A new species of Podosphaera sect. Sphaerotheca subsect. Sphaerotheca from India-first report of powdery mildew causing wilting and ultimately death of leaves of Filipendula vestita
FIGURE 1. Symptoms on Filipendula vestita a. Host plants in natural habitat. b. Wilting and drying symptom in upper younger leaves. c. Symptom on lower surface of leaf. d. Black cleistothecia on the lower surface of leaf. e. Symptom on upper surface of leaf. f. Heavily infected lower leaf surface. Scale bars: c, d = 5 mm, e = 10 mm, f = 5 mm.
Fig. 1 in Reflex Bleeding in Tonically Immobilized Larvae Causes Debris-Based Camouflage in the Blue Death-Feigning Beetle, Asbolus verrucosus LeConte (Coleoptera: Tenebrionidae)
Fig. 1. Selected developmental measurements of Asbolus verrucosus. A) Histogram of the number of eggs hatching at ambient temperature based on number of days after collection (n = 199), B) Head capsule widths for larvae, C) Time to pupation (circles, n = 12) or eclosion (diamonds, n = 11) for mature larvae transferred to 88 °F at different ages, D) Time required to induce pupation (circles, n = 12) or eclosion (diamonds, n = 11) after mature larvae were transferred to 88 °F at different ages.
Fig. 3 in Reflex Bleeding in Tonically Immobilized Larvae Causes Debris-Based Camouflage in the Blue Death-Feigning Beetle, Asbolus verrucosus LeConte (Coleoptera: Tenebrionidae)
Fig. 3. Death feigning in Asbolus verrucosus. A) Adult beetle feigning death, B) Survival analysis of adult death feigning (n = 24), C) Larva feigning death, D) Survival analysis of larval death feigning (n = 32). Scale bars: 1 cm.
Fig. 2 in Reflex Bleeding in Tonically Immobilized Larvae Causes Debris-Based Camouflage in the Blue Death-Feigning Beetle, Asbolus verrucosus LeConte (Coleoptera: Tenebrionidae)
Fig. 2. Captive-bred Asbolus verrucosus pupae and adults. A) Ventral view of a pupa, B) Lateral view of a pupa, C) Dorsal view of a newly eclosed adult, D) Rugose elytra inside elytral sheath from a preserved specimen, E) Tergite with setae from a preserved specimen, F) Setae near urogomphi, G–I) Teneral adults at increasing ages. Scales for top and bottom rows are 5 mm and 0.5 mm for the middle row.
Fig. 4 in Reflex Bleeding in Tonically Immobilized Larvae Causes Debris-Based Camouflage in the Blue Death-Feigning Beetle, Asbolus verrucosus LeConte (Coleoptera: Tenebrionidae)
Fig. 4. Reflex bleeding in Asbolus verrucosus. A) Two recently exhumed larvae feigning death. The larva on the top (arrow) bled and was obscured by debris, B) Exudate (arrows) originating from regions proximal to the second abdominal sternite, C) Micrograph of the exudate (differential interference contrast image merged with DAPI stained image) revealed hemocytes were present, D) Abdominal pleuron near second sternite in a larva prior to reflex bleeding (arrow indicates site where larva bled), E) Same larva as in (D), showing melanization at the site about 10 minutes after bleeding. Scales in A and B are 1 cm, C is 5 mm, D and E are 0.1 cm.
Once Daily Long-Acting Muscarinic Antagonists Administered in the Evening for Prevention of Chronic Obstructive Pulmonary Disease Exacerbations Requiring Hospitalization or Death from Any Cause
ClinicalTrials.gov study NCT05563675. IPD Sharing: NO. Countries: 1. Publications: 2.
PReventive Effect Of Left Bundle Branch Area Pacing Versus righT vEntricular paCing on All Cause deaTh, Heart Failure Progression, and Ventricular dysSYNChrony in Patients With Substantial Ventricular
ClinicalTrials.gov study NCT05585411. IPD Sharing: NO. Countries: 1. Publications: 11.
Complicated Grief Among Children After a Death From Cancer or Another Cause: A Longitudinal and Prospective Study (CAPS)
ClinicalTrials.gov study NCT03330145. IPD Sharing: NO. Countries: 1. Publications: 1.
Data from: Early-life disease exposure and associations with adult survival, cause of death, and reproductive success in preindustrial humans
Open the record for dataset details and reuse information.
Data from: Evolution of male age-specific reproduction under differential risks and causes of death: males pay the cost of high female fitness
Classic theories of ageing evolution predict that increased extrinsic mortality due to an environmental hazard selects for increased early reproduction, rapid ageing and short intrinsic lifespan. Conversely, emerging theory maintains that when ageing increases susceptibility to an environmental hazard, increased mortality due to this hazard can select against ageing in physiological condition and prolong intrinsic lifespan. However, evolution of slow ageing under high-condition-dependent mortality is expected to result from reallocation of resources to different traits and such reallocation may be hampered by sex-specific trade-offs. Because same life-history trait values often have different fitness consequences in males and females, sexually antagonistic selection can preserve genetic variance for lifespan and ageing. We previously showed that increased condition-dependent mortality caused by heat shock leads to evolution of long-life, decelerated late-life mortality in both sexes and increased female fecundity in the nematode, Caenorhabditis remanei. Here, we used these cryopreserved lines to show that males evolving under heat shock suffered from reduced early-life and net reproduction, while mortality rate had no effect. Our results suggest that heat-shock resistance and associated long-life trade-off with male, but not female, reproduction and therefore sexually antagonistic selection contributes to maintenance of genetic variation for lifespan and fitness in this population.
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