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ShareScore release 0.9.0
Dataset results
27 results for “maternal inflammation”
Data for Human milk lactoferrin variation in relation to maternal inflammation and iron deficiency in northern Kenya
<p>This file contains the data utilized for a journal manuscript, "Human milk lactoferrin variation in relation to maternal inflammation and iron deficiency in northern Kenya" by Fujita, Wander, Paredes Ruvalcaba, and Odo, currently under review for publication. Data are found under the Data tab, and the variable coding information under the Code tab.</p> <p>Please contact Fujita (masakof@msu.edu) for questions regarding the data.</p> <p>In using the data, please cite the DOI for this file and the above-mentioned article. Also, please acknowledge the following parties for grant support:</p> <p> National Science Foundation (BCS-0622358, BCS-1638167);</p> <p> Wenner-Gren Foundation (Gr. 7460, Gr. 9278);</p> <p> African Futures Research Leadership Program of the Alliances for African Partnership, Michigan State University; and</p> <p> Provost Undergraduate Research Initiative Grant, Michigan State University </p> <p>The data originates in the 2006 fieldwork among the Ariaal people in northern Kenya under approvals by the institutional review boards of the University of Washington and Kenya Medical Research Institute. The lactoferrin concentration in milk was determined more recently using de-identified milk specimens, and as such required no further approvals.</p> <p> </p>
Making Maternal Post-partum Vitamin A Supplementation Effective: The Role of Timing and Inflammation
ClinicalTrials.gov study NCT00952640. IPD Sharing: Not stated. Countries: 1. Publications: 4.
Data from: Maternal obesity increases insulin resistance, low-grade inflammation and osteochondrosis lesions in foals and yearlings until 18 months of age
Obesity is a growing concern in horses. The effects of maternal obesity on maternal metabolism and low-grade inflammation during pregnancy, as well as offspring growth, metabolism, low-grade inflammation, testicular maturation and osteochondrotic lesions until 18 months of age were investigated. Twenty-four mares were used and separated into two groups at insemination according to body condition score (BCS): Normal (N, n=10, BCS ≤4) and Obese (O, n=14, BCS ≥4.25). BCS and plasma glucose, insulin, triglyceride, urea, non-esterified fatty acid, serum amyloid A (SAA), leptin and adiponectin concentrations were monitored throughout gestation. At 300 days of gestation, a Frequently Sampled Intravenous Glucose Tolerance Test (FSIGT) was performed. After parturition, foals' weight and size were monitored until 18 months of age with plasma SAA, leptin, adiponectin, triiodothyronine (T3), thyroxine (T4) and cortisol concentrations measured at regular intervals. At 6, 12 and 18 months of age, FSIGT and osteoarticular examinations were performed. Males were gelded at one year and expression of genes involved in testicular maturation analysed by RT-qPCR. Throughout the experiment, maternal BCS was higher in O versus N mares. During gestation, plasma urea and adiponectin were decreased and SAA and leptin increased in O versus N mares. O mares were also more insulin resistant than N mares with a higher glucose effectiveness. Postnatally, there was no difference in offspring growth between groups. Nevertheless, SAA plasma concentrations were increased in O versus N foals until 6 months, with O foals being consistently more insulin resistant with a higher glucose effectiveness. At 12 months of age, O foals were significantly more affected by osteochondrosis than N foals. All other parameters were not different between groups. In conclusion, maternal obesity altered metabolism and increased low-grade inflammation in both dams and foals. The risk of developing osteochondrosis at 12 months of age was also higher in foals born to obese dams.
Data from: Maternal obesity increases insulin resistance, low-grade inflammation and osteochondrosis lesions in foals and yearlings until 18 months of age
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TNF-signaling modulates neutrophil-mediated immunity at the feto-maternal interface during LPS-induced intrauterine inflammation
GEO Series GSE145918. Macaca mulatta. 23 samples. Type: Expression profiling by high throughput sequencing.
Fetal brain response to maternal inflammation requires microglia [Developing_Microglia]
GEO Series GSE225464. Mus musculus. 13 samples. Type: Expression profiling by high throughput sequencing.
Fetal brain response to maternal inflammation requires microglia
GEO Series GSE225465. Mus musculus. 39 samples. Type: Expression profiling by high throughput sequencing.
Fetal brain response to maternal inflammation requires microglia [Maternal_Inflammation]
GEO Series GSE225430. Mus musculus. 26 samples. Type: Expression profiling by high throughput sequencing.
A RCT of CenteringPregnancy on Birth Outcomes and Maternal Inflammation
ClinicalTrials.gov study NCT04097548. IPD Sharing: NO. Countries: 1. Publications: 0.
Early Environmental and Maternal Determinants of Airway Inflammation in Wheezing Disorders in Infants
ClinicalTrials.gov study NCT04286464. IPD Sharing: Not stated. Countries: 1. Publications: 0.
Kinetics of Inflammation Markers in Maternal Plasma: Study of the Correlation With the Diagnosis of Chorioamnionitis in Women Hospitalized for Spontaneous Rupture of the Fetal Membranes
ClinicalTrials.gov study NCT01903759. IPD Sharing: Not stated. Countries: 1. Publications: 0.
The Role of Physical Activity During Pregnancy on Metabolic Function, Inflammation, and Maternal and Neonatal Outcomes
ClinicalTrials.gov study NCT03504319. IPD Sharing: UNDECIDED. Countries: 1. Publications: 0.
Maternal antibiotic exposure enhances neonatal ILC2 responses and aggravates allergic airway inflammation in adults
GEO Series GSE231887. Mus musculus. 6 samples. Type: Expression profiling by high throughput sequencing.
Trans-maternal exposure to Helicobacter pylori induces stable and highly suppressive regulatory T-cells and protects against allergic airway inflammation
GEO Series GSE116116. Mus musculus. 5 samples. Type: Expression profiling by high throughput sequencing.
Maternal Western Diet Programs Cardiometabolic Dysfunction and Hypothalamic Inflammation via Epigenetic Mechanisms Predominantly in the Male Offspring [RNA-seq]
GEO Series GSE242187. Mus musculus. 25 samples. Type: Expression profiling by high throughput sequencing.
A maternally inherited polymorphism in mitochondrial genome alters immune cell metabolism and protects mice from skin inflammation
GEO Series GSE126956. Mus musculus. 8 samples. Type: Expression profiling by array.
Maternal antibiotic exposure enhances neonatal ILC2 responses and aggravates allergic airway inflammation in adults [ATAC-seq]
GEO Series GSE242246. Mus musculus. 4 samples. Type: Genome binding/occupancy profiling by high throughput sequencing.
Maternal Obesity is Associated with Ovarian Inflammation and Up-regulation of Early Growth Response Factor (Egr)-1
GEO Series GSE76002. Rattus norvegicus. 6 samples. Type: Expression profiling by array.
Maternal Western Diet Programs Cardiometabolic Dysfunction and Hypothalamic Inflammation via Epigenetic Mechanisms Predominantly in the Male Offspring [miRNA-seq]
GEO Series GSE242188. Mus musculus. 25 samples. Type: Non-coding RNA profiling by high throughput sequencing.
Maternal inflammation promotes autoimmune susceptibility in offspring
GEO Series GSE165720. Mus musculus. 18 samples. Type: Expression profiling by high throughput sequencing; Genome binding/occupancy profiling by high throughput sequencing.
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