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331 results for “omega-3 fatty acids”

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zenodo40/100

FIGURE 4 in Effect of dietary omega-3 polyunsaturated fatty acids supplementation of Astyanax lacustris males on semen quality

FIGURE 4 | Results obtained for A. Head lateral displacement amplitude (ALH, µm) and B. Crossbeat frequency (BCF, Hz), after feeding with the inclusion of PUFAs-ô3 in the diet of Astyanax lacustris for a period of 105 days. In-0 – diet without inclusion of PUFAs-ô3; In-3 – feed with inclusion of 3% PUFAs-ô3; In-6 – feed with inclusion of 6% of PUFAs-ô3; In-9 – feed with inclusion of 9% PUFAs-ô3. Different lowercase letters indicate statistical difference between treatments for ALH and BCF (Kruskal-Wallis, p<0.05).

opencc-by-4.0Oct 2023View details →
zenodo40/100

FIGURE 3 in Effect of dietary omega-3 polyunsaturated fatty acids supplementation of Astyanax lacustris males on semen quality

FIGURE 3 | Results obtained for A. Linearity (LIN, %), B. Straightness coefficient (STR, %) and C. Mean oscillation of the spatial trajectory (WOB, %), after feeding with the inclusion of PUFAs-ô3 in the diet of Astyanax lacustris for a period of 105 days. In-0 – diet without inclusion of PUFAs-ô3; In-3 – feed with inclusion of 3% PUFAs-ô3; In-6 – feed with inclusion of 6% of PUFAs-ô3; In-9 – feed with inclusion of 9% PUFAs-ô3. Different lowercase letters indicate statistical difference between treatments for LIN, STR and WOB (Kruskal-Wallis, p<0.05).

opencc-by-4.0Oct 2023View details →
zenodo40/100

FIGURE 2 in Effect of dietary omega-3 polyunsaturated fatty acids supplementation of Astyanax lacustris males on semen quality

FIGURE 2 | Results obtained for A. Percentage of fast (SptzRápido), medium (SptzMedium) and slow (SptzSlow), B. Curvilinear velocity (VCL, µm/s), C. Linear velocity (VSL, µm/ s), D. Mean velocity (VAP, µm/s%) after feeding with the inclusion of PUFAs-ô3 in the diet of Astyanax lacustris for a period of 105 days. In-0 – diet without inclusion of PUFAs-ô3; In-3 – feed with inclusion of 3% PUFAs-ô3; In-6 – feed with inclusion of 6% of PUFAs-ô3; In-9 – feed with inclusion of 9% PUFAs-ô3. Different capital letters indicate statistical difference between treatments for SptzFast. Different lowercase letters indicate statistical difference between treatments for SptzMedium, SptzSlow percentage, VCL, VSL and VAP (Kruskal-Wallis, p<0.05).

opencc-by-4.0Oct 2023View details →
zenodo40/100

FIGURE 1 in Effect of dietary omega-3 polyunsaturated fatty acids supplementation of Astyanax lacustris males on semen quality

FIGURE 1 | Results obtained for total (MOT, %) and progressive (PRG, %) sperm motility after feeding with the inclusion of PUFAs-ô3 in the diet of Astyanax lacustris for a period of 105 days. In-0 – diet without inclusion of PUFAs-ô3; In-3 – feed with inclusion of 3% PUFAs-ô3; In-6 – feed with inclusion of 6% of PUFAs-ô3; In-9 – feed with inclusion of 9% PUFAs-ô3. Different capital letters indicate statistical difference between treatments for MOT. Different lowercase letters indicate statistical difference between treatments for PRG (Kruskal-Wallis, p<0.05).

opencc-by-4.0Oct 2023View details →
ClinicalTrials.gov40/100

Specified Drug-use Survey of the Granular Capsule Formulation of Omega-3 Fatty Acid Ethyl Esters: OCEAN3

ClinicalTrials.gov study NCT02285166. IPD Sharing: YES. Countries: 1. Publications: 1.

controlledIPD-YESFeb 2026View details →
dryad40/100

Data from: Biosynthesis of long-chain omega-3 fatty acids in a generalist seabird

Open the record for dataset details and reuse information.

publicMar 2025View details →
dryad36/100

Data from: Lipidome modulation by dietary omega-3 polyunsaturated fatty acid supplementation or selective soluble epoxide hydrolase inhibition suppresses rough LPS-accelerated glomerulonephritis in lupus-prone mice

<p>Lipopolysaccharide (LPS)-accelerated autoimmune glomerulonephritis (GN) in lupus-prone NZBWF1 mice is a preclinical model that is potentially applicable for investigating lipidome-modulating interventions. LPS can be expressed as one of two chemotypes: smooth LPS (S-LPS) and rough LPS (R-LPS) which is devoid of O-antigen polysaccharide sidechain. Since these chemotypes differentially affect TLR4-mediated immune cell responses, these differences may influence GN induction. Therefore, we initially compared the effects of subchronic i.p. injection for 5 wk with 1) <em>Salmonella</em> S-LPS, 2) <em>Salmonella</em> R-LPS, or 3) saline vehicle (VEH) (Study 1) in female NZBWF1 mice. R-LPS induced robust elevations in blood urea nitrogen, proteinuria, and hematuria that were not evident in VEH- or S-LPS-treated mice. Histopathologic examination of R-LPS-treated mice one week after final injection further revealed more robust hypertrophy, hyperplasia, thickened membranes, lymphocytic accumulation containing B and T cells, and glomerular IgG deposition consistent with GN but not in VEH- or S-LPS-treated groups. R-LPS but not S-LPS induced spleen enlargement with lymphoid hyperplasia as well as modest inflammatory cell recruitment in the liver. We next employed our optimized R-LPS model to discern the impact of two lipidome-modulating interventions, omega-3 polyunsaturated fatty acid (PUFA) supplementation and soluble epoxide hydrolase (sEH) inhibition, on GN (Study 2). Specifically, the effects of consuming the omega-3 PUFA docosahexaenoic acid (DHA) (10 g/kg diet) and/or the sEH inhibitor TPPU (22.5 mg/kg diet) on R-LPS triggering were compared. Resultant blood fatty acid profiles and epoxy fatty acid concentrations reflected the anticipated DHA- and TPPU-mediated lipidome changes. The relative rank order of R-LPS-induced GN severity among groups fed experimental diets based on proteinuria, hematuria, histopathologic scoring, and glomerular IgG deposition was: VEH/CON &lt; R-LPS/DHA ≈ R-LPS/TPPU &lt;&lt;&lt; R-LPS/ TPPU+DHA ≈ R-LPS/CON. These interventions had modest to negligible effects on R-LPS-induced splenomegaly, plasma antibody responses, liver inflammation, and inflammation-associated kidney gene expression. Collectively, our results show for the first time that absence of O-antigenic polysaccharide in R-LPS is critical to accelerated GN in lupus-prone mice. Furthermore, intervention by lipidome modulation through DHA feeding or sEH inhibition suppressed R-LPS-induced GN; however, these ameliorative effects were greatly diminished upon combining the treatments.</p>

opencc-zeroDec 2022View details →
zenodo36/100

Evolutionary Mismatch and depression: the correlation between omega-3 fatty acid intake from fish in diet and the prevalence of depression among university students

<p>This compressed file contain the data used for the Bachelor project &quot;Evolutionary Mismatch and depression: the correlation between omega-3 fatty acid intake from fish in diet and the prevalence of depression among university students&quot; by Ziyao Zhang from the University of Amsterdam.</p>

opencc-by-4.0Jul 2023View details →
ClinicalTrials.gov36/100

Obesity and Asthma: Nutrigenetic Response to Omega-3 Fatty Acids

ClinicalTrials.gov study NCT01027143. IPD Sharing: Not stated. Countries: 1. Publications: 1.

restrictedIPD-UNDECIDEDFeb 2026View details →
ClinicalTrials.gov36/100

Omega-3 Fatty Acids for Major Depressive Disorder With High Inflammation: A Personalized Approach

ClinicalTrials.gov study NCT02553915. IPD Sharing: NO. Countries: 1. Publications: 4.

closedIPD-NOFeb 2026View details →
ClinicalTrials.gov36/100

Efficacy Study Regarding the Beneficial Effects of Omega-3 Fatty Acids on Cardiometabolic Health

ClinicalTrials.gov study NCT02042274. IPD Sharing: NO. Countries: 1. Publications: 1.

closedIPD-NOFeb 2026View details →
ClinicalTrials.gov36/100

Dietary Essential Fatty Acid Regulation of Omega-3 HUFA Metabolism; Satiety and Body Composition

ClinicalTrials.gov study NCT01251887. IPD Sharing: Not stated. Countries: 1. Publications: 3.

restrictedIPD-UNDECIDEDFeb 2026View details →
ClinicalTrials.gov36/100

Omega-3 Fatty Acids for Autism Treatment

ClinicalTrials.gov study NCT00786799. IPD Sharing: Not stated. Countries: 1. Publications: 2.

restrictedIPD-UNDECIDEDFeb 2026View details →
ClinicalTrials.gov36/100

Omega-3 Fatty Acids and Insulin Sensitivity

ClinicalTrials.gov study NCT01686568. IPD Sharing: Not stated. Countries: 1. Publications: 2.

restrictedIPD-UNDECIDEDFeb 2026View details →
ClinicalTrials.gov36/100

Omega-3 Fatty Acids for High Triglycerides in HIV-infected Patients

ClinicalTrials.gov study NCT00346697. IPD Sharing: Not stated. Countries: 1. Publications: 1.

restrictedIPD-UNDECIDEDFeb 2026View details →
ClinicalTrials.gov36/100

Omega-3 Fatty Acids, the Omega-3 Index, and Atherosclerosis

ClinicalTrials.gov study NCT00886704. IPD Sharing: Not stated. Countries: 1. Publications: 1.

restrictedIPD-UNDECIDEDFeb 2026View details →
ClinicalTrials.gov36/100

Lycopene or Omega-3 Fatty Acid Nutritional Supplements in Treating Patients With Stage I or Stage II Prostate Cancer

ClinicalTrials.gov study NCT00402285. IPD Sharing: Not stated. Countries: 1. Publications: 2.

restrictedIPD-UNDECIDEDFeb 2026View details →
ClinicalTrials.gov36/100

Impact of Omega-3 Fatty Acid Oral Therapy on Healing of Chronic Venous Leg Ulcers in Older Adults

ClinicalTrials.gov study NCT03576989. IPD Sharing: NO. Countries: 1. Publications: 1.

closedIPD-NOFeb 2026View details →
ClinicalTrials.gov36/100

Omega-3 Fatty Acids For Treatment Of Young Children With Autism (OMG)

ClinicalTrials.gov study NCT01248728. IPD Sharing: Not stated. Countries: 1. Publications: 1.

restrictedIPD-UNDECIDEDFeb 2026View details →
ClinicalTrials.gov36/100

Decreasing Risk of Coronary Artery Disease in Schizophrenia by Omega-3 Fatty Acid Supplementation

ClinicalTrials.gov study NCT00167310. IPD Sharing: Not stated. Countries: 1. Publications: 1.

restrictedIPD-UNDECIDEDFeb 2026View details →

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International Brain Laboratory public data

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OpenNeuro

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