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22 results for “parasite intensity”
Seasonal Effects in Gastrointestinal Parasite Prevalence, Richness and Intensity in Vervet Monkeys Living in a Semi-Arid Environment
<p>Data and R Notebook for Seasonal Effects in Gastrointestinal Parasite Prevalence, Richness and Intensity in Vervet Monkeys Living in a Semi-Arid Environment</p>
Fig. 3 in Behavioral, physiological and morphological correlates of parasite intensity in the wild Cururu toad (Rhinella icterica)
Fig. 3. Association between locomotor performance and pulmonary parasite intensity in Rhinella icterica (N = 20; r = –0.49, P = 0.03). SVL = snout-vent length.
Fig. 2 in Behavioral, physiological and morphological correlates of parasite intensity in the wild Cururu toad (Rhinella icterica)
Fig. 2. Association between standard metabolic rate and total parasite intensity in Rhinella icterica (N = 22; r = –0.45, P = 0.03).
Fig. 1 in Behavioral, physiological and morphological correlates of parasite intensity in the wild Cururu toad (Rhinella icterica)
Fig. 1. Association between the score of the first component of a PCA on morphological variables (large heart, kidney and intestine masses) and parasite intensities in Rhinella icterica (N = 16). Full circles represent total parasite intensity (r = 0.66, P <0.01), open circles represent pulmonary parasite intensity (r = 0.71, P <0.01), and open triangles represent intestinal parasite intensity (r = 0.51, P = 0.04).
Figure 6 in Intensity and prevalence of some crustacean fish parasites in Turkey and their molecular identification
Figure 6. Livoneca punctata on gill of Alosa immaculata (A), manca (B, D), adult female L. punctata and its juvenile manca (C).
Figure 5 in Intensity and prevalence of some crustacean fish parasites in Turkey and their molecular identification
Figure 5. Infestation of Nerocila spp. on Platichthys flesus (A) and infestation of Nerocila bivittata on Pegusa nasuta (B), mechanic injury on caudal peduncle of sole (C), clear lesions on caudal fin of sea bass (D. labrax) (D).
Effects of an alternative host on the prevalence and infection intensity of a bumble bee parasite
<p>Several bee parasites are transmitted through flowers, and some of them can infect multiple host species. Given the shared use of flowers by bee species, parasites can potentially encounter multiple host species, which could affect the evolution of parasite virulence. We used the trypanosomatid parasite <em>Crithidia bombi</em> and its host, the common eastern bumble bee (<em>Bombus impatiens</em>), to explore the effect of infecting an alternative host, the alfalfa leaf-cutter bee (<em>Megachile rotundata</em>), on parasite infectivity and ability to replicate. We conducted a serial passage experiment on primary and alternative hosts, assessing infectivity and intensity of infection during five passes. Parasite cells from each pass through the alternative host were also used to infect a group of primary hosts. We found that serial passes through the alternative host increased infectivity, but there was no effect on intensity of infection. Interestingly, both the probability and intensity of infection on the primary host increased after serial passage through the alternative host. This increase in intensity of infection could be due to maladaptation after selection of new <em>C. bombi</em> strains has occurred in the alternative host. This study suggests that host switching has the potential to affect the adaptation of bee parasites to their hosts.</p>
Data and R code used in the preprint "Parasite intensity is driven by temperature in a wild bird" (doi 10.1101/323311)
<p>Data (as text files) and R code used in the preprint entitled "Parasite intensity is driven by temperature in a wild bird", recommended by <em>Peer Community In Ecology </em>(doi 10.1101/323311). See preprint and supplementary material; some explanations are also included in the R code. </p>
Figure. Location of Beymelek Lagoon in southern Turkey. in Prevalence and intensity of parasitic helminths of thicklip grey mullet Chelon labrosus in hosts in Beymelek Lagoon Lake in Antalya, Turkey, according to season, host size, age, and sex of the host
Figure. Location of Beymelek Lagoon in southern Turkey.
Figure 4 in Intensity and prevalence of some crustacean fish parasites in Turkey and their molecular identification
Figure 4. Caligus minimus male (A), caudal rami (B), lunules (C), sternal furca (D).
Figure 2 in Intensity and prevalence of some crustacean fish parasites in Turkey and their molecular identification
Figure 2. Caligus minimus on mouth cavity (A) and tongue (B) of Dicentrarchus labrax.
Figure 1 in Intensity and prevalence of some crustacean fish parasites in Turkey and their molecular identification
Figure 1. Sampling area.
Effects of an alternative host on the prevalence and infection intensity of a bumble bee parasite
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Intense nocturnal warming alters growth strategies, coloration, and parasite load in a diurnal lizard
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Parasite intensity and the evolution of migratory behavior
Migration can allow individuals to escape parasite infection, which can lead to a lower infection probability (prevalence) in a population and/or fewer parasites per individual (intensity). Since individuals with more parasites often have lower survival and/or fecundity, infection intensity shapes the life-history tradeoffs determining when migration is favored as a strategy to escape infection. Yet, most theory relies on susceptible-infected (SI) modeling frameworks, defining individuals as either healthy or infected, ignoring details of infection intensity. Here we develop a novel modeling approach that captures infection intensity as a spectrum, and ask under what conditions migration evolves as function of how infection intensity changes over time. We show that the relative timescales of migration and infection accumulation determine when migration is favored. We also find that population-level heterogeneity in infection intensity can lead to partial migration, where less-infected individuals migrate while more infected individuals remain resident. Our model is one of the first to consider how infection intensity can lead to migration. Our results frame migratory escape in light of infection intensity, rather than prevalence, thus demonstrating that decreased infection intensity should be considered a benefit of migration, alongside other typical drivers of migration.
Parasite intensity and the evolution of migratory behavior
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Data from: Metrics matter: the effect of parasite richness, intensity and prevalence on the evolution of host migration
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Data from: Genetic admixture predicts parasite intensity: evidence for increased hybrid performance in Darwin’s tree finches
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Data from: Low intensity blood parasite infections do not reduce the aerobic performance of migratory birds
Blood parasites (Haemosporidia) are thought to impair the flight performance of infected animals, and therefore, infected birds are expected to differ from their non-infected counterparts in migratory capacity. Since haemosporidians invade host erythrocytes, it is commonly assumed that infected individuals will have compromised aerobic capacity, but this has not been examined in free-living birds. We tested if haemosporidian infections affect aerobic performance by examining metabolic rates and exercise endurance in migratory great reed warblers (Acrocephalus arundinaceus) experimentally treated with Plasmodium relictum pGRW04 and in naturally infected wild birds over consecutive life history stages. We found no effect of acute or chronic infections on resting metabolic rate, maximum metabolic rate or exercise endurance in either experimentally treated or free-living birds. Oxygen consumption rates during rest and while undergoing maximum exercise as well as exercise endurance increased from breeding to migration stages in both infected and non-infected birds. Importantly, phenotypic changes associated with preparation for migration were similarly unaffected by parasitaemia. Consequently, migratory birds experiencing parasitaemia levels typical of chronic infection do not differ in migratory capacity from their uninfected counterparts. Thus, if infected hosts differ from uninfected conspecifics in migration phenology, another mechanism than changes in aerobic capacity should be involved.
Data from: Invest more and die faster: the life history of a parasite on intensive farms
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Allen Brain Atlas
Allen Brain Atlas is an Allen Institute collection of brain map atlases, datasets, APIs, and analysis tools covering mouse, human, and non-human primate brain resources.
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DANDI Archive for NWB datasets
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International Brain Laboratory public data
The International Brain Laboratory public data releases expose standardized mouse decision-making experiments, including Neuropixels recordings, widefield calcium imaging, behavior, and session metadata accessed through the ONE API.
OpenNeuro
OpenNeuro is a free, open platform for sharing neuroimaging datasets, with public search, dataset pages, and download paths for web, S3, DataLad, and the OpenNeuro CLI.