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5 results for “plant mitochondria”
Data for: A unique C-terminal domain contributes to the molecular function of restorer-of-fertility proteins in plant mitochondria
<p><em><span>Restorer-of-fertility</span></em><span><em> </em>(<em>Rf</em>) genes have practical applications in hybrid seed production as a means to control self-pollination. They encode pentatricopeptide repeat (PPR) proteins that are targeted to mitochondria where they specifically bind to transcripts that induce cytoplasmic male sterility and repress their expression. </span></p> <p>We have identified a unique domain, RfCTD (Restorer-of-fertility C-terminal domain), which discriminates <em>Restorer-of-fertility-like</em> (RFL) proteins from hundreds of PPR proteins encoded in plant genomes. Using the sequence of this domain from hundreds of plant species, we have constructed a sequence profile that can quickly and accurately identify RfCTD sequences in plant genomes or transcriptomes. </p> <p>This data set contains PPR genes identified in 213 plant genomes (as summarised in accompanying table). </p>
Data for: A unique C-terminal domain contributes to the molecular function of restorer-of-fertility proteins in plant mitochondria
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Fig. 5. Compound 7 induced apoptosis through a mitochondria-dependent pathway. A in Bufadienolide glycosides and bufadienolides from the whole plants of Helleborus lividus, and their cytotoxic activity
Fig. 5. Compound 7 induced apoptosis through a mitochondria-dependent pathway. A, Mitochondrial membrane morphology of HL- 60 cells treated with 7. HL-60 cells were stained with MitoCapture™ reagent after treatment with either 0.21 μM of 7 or 33 μM of cisplatin for 6 h and observed under a fluorescence microscope. B, Release of cytochrome c from mitochondria of HL-60 cells treated with 7. HL-60 cells were treated with either 0.21 μM of 7 or 33 μM of cisplatin for 8 h, and the release of cytochrome c into the cytoplasm was evaluated by Western blot analysis.
Fig. 5. Compound 2 induced apoptosis through a mitochondria-dependent pathway. A in Bufadienolides from the whole plants of Helleborus foetidus and their cytotoxicity
Fig. 5. Compound 2 induced apoptosis through a mitochondria-dependent pathway. A, Mitochondrial membrane morphology of HL-60 cells treated with 2. HL-60 cells were stained with MitoCapture™ reagent after treatment with either 0.5 μM of 2 or 33 μM of cisplatin for 10 h and observed under a fluorescence microscope. B, Release of cytochrome c from mitochondria of HL-60 cells treated with 2. HL-60 cells were treated with either 0.5 μM of 2 or 33 μM of cisplatin for 6 h, and the release of cytochrome c into the cytoplasm was evaluated by Western blot analysis.
Protein aggregation in plant mitochondria lacking Lon1 inhibits translation and induces unfolded protein responses
GEO Series GSE216535. Arabidopsis thaliana. 6 samples. Type: Expression profiling by high throughput sequencing.
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