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Figure 6 in Potential Hepatoprotective Effect of Cheatomorpha gracilis extract against High Fat Diet (HFD)-Induced Liver Damage, and its characterization by HPLC

Figure 6. Photomicrographs of hepatocytes in control and experimental treated mice. Liver tissue sections stained with oil red O (×200) (A) and scored (B) by the semi-quantitative percentage of damaged area. (Control) control mice and (AEC) treated with C. gracilis extract (250 mg/kg) alone, showed normal structure. (HFD) high fed diet treated mice, showed a significant increase of lipid content; (AEC + HFD) co-treated with high fed diet and alga extract (250 mg/kg), representative micrograph revealed significantly reduced lipid liver content. Values are expressed as the mean ± SD. **p<0.01; ***p <0.001, indicates a significant difference compared to the control group. ++ p<0.01, indicates a significant difference compared to the HFD alone group.

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Figure 3 in Potential Hepatoprotective Effect of Cheatomorpha gracilis extract against High Fat Diet (HFD)-Induced Liver Damage, and its characterization by HPLC

Figure 3. Effect of ACE supplementation on plasma levels of inflammatory proteins. Data are given as mean ± SD for group of eight mice each. Values are statistically presented as follows: HFD and AEC-treated groups vs. control group: *p <0.05; **p<0.01. HFD + AEC group vs. HFD group: +p <0.05; ++p<0.01.

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Figure 2 in Potential Hepatoprotective Effect of Cheatomorpha gracilis extract against High Fat Diet (HFD)-Induced Liver Damage, and its characterization by HPLC

Figure 2. Antioxidant activity of different extracts of C. gracilis. Values are mean ± SD of three experimental tests, mg GAE/g DR: mg gallic acid equivalents per g dry residue. Table 4. Effect of AEC on body weight, liver and adipose tissues relative weight of control and treated mice groups.

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Figure 5 in Potential Hepatoprotective Effect of Cheatomorpha gracilis extract against High Fat Diet (HFD)-Induced Liver Damage, and its characterization by HPLC

Figure 5. Photographs of liver tissues in control and experimental treated mice. Liver tissue sections stained with hematoxylin and eosin (original magnification × 400). (A), in control mice showing normal histoarchitecture and radiating cell arrangement. (B) In mice treated with aqueous extract of C.gracilis (AEC) (250mg/kg) alone showing the normal liver structure. (C) In High fed diet (HFD) mice, representative photograph shows a significant lipid accumulation. (D) In group co-treated with HFD and AEC, was observed the reduction of fat accumulation.

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Figure 1 in Potential Hepatoprotective Effect of Cheatomorpha gracilis extract against High Fat Diet (HFD)-Induced Liver Damage, and its characterization by HPLC

Figure 1. Phenolic compounds of Cheatomorpha gracilis extract by chemical High Performance Liquid Chromatography (HPLC) analysis (gallic acid, Rutin, quercetin, Apegenin, kaempferol and Naringenin).

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Figure 4 in Potential Hepatoprotective Effect of Cheatomorpha gracilis extract against High Fat Diet (HFD)-Induced Liver Damage, and its characterization by HPLC

Figure 4. Effects of C.gracilis aqueous extract (AEC) on TBARS and protein carbonyls products (PCO) in HFD-treated mice. TBARS: thiobarbituric acid reactive substance. Data are presented as mean ± SD, n =8. *P≤ 0.05, **P≤ 0.01 compared with the control group. +P ≤0.05, ++P ≤0.01 as compared with HFD- treated mice.

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Figure 8 in Pouteria ramiflora leaf extract on emulgel in wound healing activity in diabetic rats

Figure 8. Photomicrograph of histological sections of rats' skin wounds of Control group (Carbopol) (A, B, and C), Ethanolic extract (Ext) (D, E, F) and Gel + Ethanolic extract 2% (Ext+gel) (G, H, I) at 30 days after the skin wound injury (D, E, F). epidermis (ep); granular layer (green arrow); fibroblasts (black arrow). HE.

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Figure 5 in Pouteria ramiflora leaf extract on emulgel in wound healing activity in diabetic rats

Figure 5. Quantification of inflammatory cells, fibroblasts, and angiogenesis (blood vessels) in diabetic animals' skin on groups: Control, Ext and Ext+gel, 14 days. ns (statistically not significant). DUNN's test (P=0.05).

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Figure 4 in Pouteria ramiflora leaf extract on emulgel in wound healing activity in diabetic rats

Figure 4. Photomicrograph of histological sections of diabetic rats' skin wounds. (A, A1), Control, Ext (B, B1) and Ext+gel (C, C1) to 14 days after injury of the skin wound. Epidermis (ep); keratin (k); fibroblast (f); collagen fibers (c); HE. Quantification of inflammatory cells, fibroblasts, and angiogenesis in diabetic animals' skin on groups: Control, Ext and Ext+gel, DUNN's test (P=0.05).

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Figure 3 in Pouteria ramiflora leaf extract on emulgel in wound healing activity in diabetic rats

Figure 3. Quantification of inflammatory cells, fibroblasts, and angiogenesis (blood vessels) in diabetic animals' skin on groups: Control; Ext and Ext+gel, 7 days. ns (statistically not significant). DUNN's test (P=0.05).

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Figure 2 in Pouteria ramiflora leaf extract on emulgel in wound healing activity in diabetic rats

Figure 2. Photomicrograph of histological sections of diabetic rats' skin wounds of groups: Control, Ext and Ext+gel (A, B, C) at 7 days after injury of the skin wound (A1, B1, C1), crust, epidermis (ep); dermis (d) deep dermis with tissue granulation, fibroblasts (f); blood vessels (angiogenesis) (bv); (HE, 10 and 40x).

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Figure 1 in Pouteria ramiflora leaf extract on emulgel in wound healing activity in diabetic rats

Figure 1. Chart of analysis phytochemical of Ethanolic extract of Pouteria ramiflora leaves, Campo Grande, 2016. Frequency = low (+ = 25%), moderately moderate (++ = 50%) and high intensity (+++ = 100%), besides negative (- = 0%).

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Figure 7 in Phytotoxicity of plant extracts of Vismia japurensis cultivated in vivo and in vitro

Figure 7. Expansion of the 10.0 to 13.0 ppm region of hexanic extract 1H-NMR spectra (top to bottom): (a) Branch hexanic extract; (b) Leaf hexanic extract; (c) Hexanic extract from in vitro seedlings.

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Figure 6 in Phytotoxicity of plant extracts of Vismia japurensis cultivated in vivo and in vitro

Figure 6. Expansion of the region from 5.0 to 5.5 and 6.0 to 8.0 ppm of the hexanic extracts 1H-NMR spectra (top to bottom): (a) Branch hexanic extract; (b) Leaf hexanic extract; (c) Hexanic extract from in vitro seedlings.

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Figure 5. 1H in Phytotoxicity of plant extracts of Vismia japurensis cultivated in vivo and in vitro

Figure 5. 1H-RMN spectra for hexanic extracts (top to bottom): (a) Branch hexanic extract; (b) Leaf hexanic extract; (c) Hexanic extract from in vitro seedlings.

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Figure 3 in Phytotoxicity of plant extracts of Vismia japurensis cultivated in vivo and in vitro

Figure 3. Growth of Lactuca sativa seedlings under the influence of dry and fresh Vismia japurensis leaves (sandwich tests). Significant results are followed by: **p <0.01; ***p <0.001; ****p <0.0001.

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Figure 4 in Phytotoxicity of plant extracts of Vismia japurensis cultivated in vivo and in vitro

Figure 4. Seedlings of Lactuca sativa showing the influence of hexanic extracts: (A) L. sativa in contact to leaf hexanic extract and compared to control; (B) General view of L. sativa in contact with hexanic extract from in vitro seedlings and compared to control; (C) Several L. sativa plants in contact with hexanic extract from in vitro seedlings and compared to control; (D) One L. sativa plant in contact with hexanic extract from in vitro seedlings and compared to control; (E) Plant showed in (D) magnified view (2.5x).

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Figure 2 in Phytotoxicity of plant extracts of Vismia japurensis cultivated in vivo and in vitro

Figure 2. Lactuca sativa seedling growth when in contact to different methanolic extracts. Significant results are followed by: ****p <0.0001.

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Figure 1 in Phytotoxicity of plant extracts of Vismia japurensis cultivated in vivo and in vitro

Figure 1. Lactuca sativa seedling growth when in contact to different hexanic extracts. Significant results are followed by: ****p <0.0001.

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Figure 2 in Determination of the ursolic and oleanolic acids content with the antioxidant capacity in apple peel extract of various cultivars

Figure 2. HPLC chromatogram of ursolic acid and oleanolic acid: Standards (500 µg/ml) (A), Apple peel methanolic extract for Red Delicious (B), Granny Smith (C) and, Royal Gala (D) cultivars. UA: ursolic acid; OA: oleanolic acid.

opencc-by-4.0Dec 2022View details →

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Allen Brain Atlas

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behavioral-neuroscienceopenThe DataShare record exposes download links for annotations, documentation, license text, and the zipped per-snippet data directory.
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DANDI Archive for NWB datasets

DANDI is a BRAIN Initiative archive for publishing and sharing neurophysiology data, including electrophysiology, optophysiology, and behavioral data packaged as NWB and related standards.

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Last verified 2026-04-30Open record

International Brain Laboratory public data

The International Brain Laboratory public data releases expose standardized mouse decision-making experiments, including Neuropixels recordings, widefield calcium imaging, behavior, and session metadata accessed through the ONE API.

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Last verified 2026-04-29Open record

OpenNeuro

OpenNeuro is a free, open platform for sharing neuroimaging datasets, with public search, dataset pages, and download paths for web, S3, DataLad, and the OpenNeuro CLI.

openneuro
neuroscienceopenPublished datasets are available on demand over the internet.
Last verified 2026-04-29Open record

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