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Dataset results
35 results for “MEA”
A Multicenter Observational Study to Understand the Clinical Characteristics, Treatment Patterns and Access to Novel Therapies of Patients With Diffuse Large B-Cell Lymphoma in the MEA Region
ClinicalTrials.gov study NCT07065344. IPD Sharing: Not stated. Countries: 0. Publications: 0.
RNA sequencing of LNZ308 glioma cells treated under differential conditions including monotherapies, dual therapy and synergistic triple regimen employing γ-irradiation, temozolomide and oncolytic mea
GEO Series GSE111247. Homo sapiens. 45 samples. Type: Expression profiling by high throughput sequencing.
Mea La Mung Kee (แม่ละมุ้งคี)
Source: Objaverse 1.0 / Sketchfab
Dataset of D.-H. Chae et al., Meas. Sci.Technol 33 (2022) 065012, "Investigation of the stability of graphene devices for quantum resistance metrology at direct and alternating current"
<p>Compilation of the datasets used to generate the figures in the following journal publication:</p> <p>"Investigation of the stability of graphene devices for quantum resistance metrology at direct and alternating current"</p> <p>by Dong-Hun Chae, Mattias Kruskopf, Jan Kucera, Jaesung Park, Ngoc Thanh Mai Tran, Dan Bee Kim, Klaus Pierz, Martin Götz, Yefei Yin, Pavel Svoboda, Petr Chrobok, François Couëdo and Félicien Schopfer,</p> <p>Meas. Sci. Technol. 33 (2022) 065012,</p> <p>DOI:10.1088/1361-6501/ac4a1a</p>
Mea Kasa Hot Spring (น้ำพุร้อนแม่กาษา)
Source: Objaverse 1.0 / Sketchfab
A Cross-sectional Study of COPD and Symptom Variability in MEA Countries
ClinicalTrials.gov study NCT03425760. IPD Sharing: Not stated. Countries: 0. Publications: 0.
Meanook (MEA) Ground-based Vector Magnetic Field (L2) 1.0 s Data
Meanook, AB, Ground-based Vector Magnetic Field Level 2 Data, 1.0 s Time Resolution, Station Code: (MEA), Station Location: (GEO Latitude 54.6, Longitude 246.7), NRCan Network
Datasets for S. Bauer et al., Meas. Sci. Technol. (2020), "A Four-Terminal-Pair Josephson Impedance Bridge Combined with a Graphene Quantized Hall Resistance"
<p>Compilation of the datasets used to generate the figures in the following journal publication;</p> <p>"A Four-Terminal-Pair Josephson Impedance Bridge Combined with a Graphene Quantized Hall Resistance"</p> <p>by Bauer, S., Behr, R., Elmquist, R.E., Götz, M., Herick, J., Kieler, O., Kruskopf, M., Lee, J., Palafox, L., Pimsut, Y., and Schurr, J,</p> <p>Meas. Sci. Technol. (2020),</p> <p>DOI: 10.1088/1361-6501/abcff3</p> <p> </p>
Datasets of Park et al.,Meas. Sci. Technol. 33 115019 (2022), "Glass encapsulation of molecular-doped epitaxial graphene for quantum resistance metrology"
<p>Compilation of the datasets used to generate the figures in the following journal publication:</p> <p>"Glass encapsulation of molecular-doped epitaxial graphene for quantum resistance metrology"</p> <p>by Jaesung Park, Kyung-Geun Lim and Dong-Hun Chae,</p> <p>Meas. Sci. Technol. 33 (2022) 115019,</p> <p>DOI: <a href="https://doi.org/10.1088/1361-6501/ac8222">10.1088/1361-6501/ac8222</a></p>
Neuronal and neuron-astrocyte co-culture MEA recordings.
<p>A dataset consisting of various standard 60-electrode MEA recordings. The recorded cultures are:</p> <p>'NS' - E18 primary rat cortical neurons (A1084001, Thermo Fisher Scientific, Waltham, MA, USA));</p> <p>'8020' - 80 % E18 primary rat cortical neurons (A1084001, Thermo Fisher Scientific, Waltham, MA, USA)) + 20 % rat cortical astrocytes (N7745100, Thermo Fisher Scientific, Waltham, MA,USA);</p> <p>'5050' - 50 % E18 primary rat cortical neurons (A1084001, Thermo Fisher Scientific, Waltham, MA, USA)) + 50 % rat cortical astrocytes (N7745100, Thermo Fisher Scientific, Waltham, MA,USA);</p> <p>The MEAs were recorded at 14, 19, and 28 DIV. The recordings lasted 5 minutes after 5 minutes of settling on the preamplifier.</p> <p>The file name format is: div*XX*-*meaID*-*cultureType*-5min.csv</p> <p>where:</p> <p>*XX* is the recorded day in vitro</p> <p>*meaID* is the ID of the MEA in order to be able to follow the same MEA during the development</p> <p>*cultureType* is either NS, 8020 or 5050 so the user knows which coculture was plated on the recorded MEA </p> <p><br> For all the other plating details please refer to: <em><strong>Preprint version <a href="http://doi.org/10.36227/techrxiv.22692313.v1">10.36227/techrxiv.22692313.v1</a></strong></em></p> <p> </p>
CRRES MEA 1-min electron distribution functions
This data set holds 1-min resolution electron distributions from the Medium Electron Sensor/Analyzer (MEA) on the CRRES spacecraft. The data consists of counting rates from 17 energy channels in the range of 0.1-2 MeV and 19 pitch angle bins at 1 minute time intervals. The average flux, 90 degree flux and N value are included. Also included are the spacecraft geographic coordinates and altitude, L shell, and the local and equatorial magnetic field magnitudes from the 1977 Olson-Pfitzer model of the earth's geomagnetic field. The raw high resolution (0.512 sec) data and documentation of raw data can be obtained via ftp from NSSDC.
Data set from the article Cairo B, De Maria B, Bari V, Vaini E, Heusser K, Tank J, Jordan J, Barbic F, Furlan R, Marinou K, Dalla Vecchia L, Porta A. Information-domain method for the quantification of the complexity of the sympathetic baroreflex regulation in healthy subjects and amyotrophic lateral sclerosis patients. Physiol Meas. 2019 Apr 4;40(3):034004. doi: 10.1088/1361-6579/ab0d4b. PMID: 30840931.
<p>Data set from the article Cairo B, De Maria B, Bari V, Vaini E, Heusser K, Tank J, Jordan J, Barbic F, Furlan R, Marinou K, Dalla Vecchia L, Porta A. Information-domain method for the quantification of the complexity of the sympathetic baroreflex regulation in healthy subjects and amyotrophic lateral sclerosis patients. Physiol Meas. 2019 Apr 4;40(3):034004. doi: 10.1088/1361-6579/ab0d4b. PMID: 30840931.</p> <p>this is the abstract:</p> <p><strong>Background: </strong>The sympathetic baroreflex (sBR) adjusts muscle sympathetic nerve activity (MSNA) in response to arterial pressure changes but the relevance of assessing sBR control complexity is unclear.</p> <p><strong>Objective: </strong>We propose a method for the evaluation of sBR control complexity.</p> <p><strong>Approach: </strong>The approach comprises the quantification of complexity of the sBR latency regulation and the assessment of complexity of the relationship linking MSNA burst to R-wave peak regardless of the variability of the sBR latency. The Shannon entropy (SE) of the sBR latency distribution is taken as an estimate of complexity of the sBR latency regulation. The conditional entropy (CE) of the beat-to-beat binary series obtained by coding the presence/absence of the MSNA burst after an R-wave peak is taken as an estimate of complexity of the sBR control regardless of the sBR latency variability. Surrogate analysis was utilized to set the level of inactive or impaired sBR. The approach was applied to 10 young healthy subjects undergoing head-up tilt (HUT) followed by lower body negative pressure to evoke presyncope (preSYNC) before and after 21 d head-down bed rest (HDBR), and to five amyotrophic lateral sclerosis (ALS) patients undergoing HUT.</p> <p><strong>Main results: </strong>In healthy subjects the surrogate analysis suggested that HUT and preSYNC significantly activated the sBR control but its response was weakened after 21 d HDBR. During preSYNC sBR latency increased significantly only after 21 d HDBR. In ALS patients the complexity of the sBR latency regulation was close to the level set by surrogate analysis and HUT did not trigger any sBR control response.</p> <p><strong>Significance: </strong>The proposed method for sBR control complexity quantification was useful in detecting the impairment of the sBR control after 21 d HDBR in healthy subjects and the dysfunction of the sBR regulation in ALS patients.</p>
Data set from the article Bari V, Vaini E, Pistuddi V, Fantinato A, Cairo B, De Maria B, Ranucci M, Porta A. Short-term multiscale complexity analysis of cardiovascular variability improves low cardiac output syndrome risk stratification after coronary artery bypass grafting. Physiol Meas. 2019 Apr 26;40(4):044001. doi: 10.1088/1361-6579/ab12f0. PMID: 30909175.
<p>Data set from the article Bari V, Vaini E, Pistuddi V, et al. Short-term multiscale complexity analysis of cardiovascular variability improves low cardiac output syndrome risk stratification after coronary artery bypass grafting. <em>Physiol Meas</em>. 2019;40(4):044001. Published 2019 Apr 26. doi:10.1088/1361-6579/ab12f0</p> <p>This is the abstract:</p> <p><strong>Background: </strong>Low cardiac output syndrome (LCOS) is a myocardial dysfunction leading to systemic hypoperfusion, favored by particular conditions of the autonomic nervous system. LCOS is one of the adverse events that might occur after cardiac surgery.</p> <p><strong>Objective: </strong>The aim is to test the hypothesis that short-term multiscale complexity (MSC) analysis of heart period (HP) and systolic arterial pressure (SAP) variability series in the frequency bands typical of cardiovascular control could be fruitfully exploited in identifying subjects at risk of developing LCOS after coronary artery bypass graft (CABG).</p> <p><strong>Approach: </strong>HP and SAP beat-to-beat series were derived from electrocardiogram (ECG) and invasive arterial pressure (AP) signal acquired in 128 patients scheduled for CABG before (PRE) and after (POST) the induction of general anesthesia with propofol and remifentanil. Subjects were labeled as LCOS (n = 14) and noLCOS (n = 114) according to the LCOS development. MSC markers were calculated as the complement to 1 of the modulus of the average position of the poles dropping in the low-frequency (LF, 0.04-0.15 Hz) and high-frequency (HF, 0.15-0.5 Hz) bands as derived from the autoregressive model of HP and SAP series. Traditional time and frequency domain indexes were also calculated.</p> <p><strong>Main results: </strong>Traditional parameters were able to assess the depression of the cardiovascular regulation induced by general anesthesia, but showed weak performances in differentiating LCOS and noLCOS groups. Conversely, HP complexity in LF band and SAP complexity in HF band assessed during POST remained associated with LCOS even after entering a multivariate logistic regression model adjusted for clinical and demographic factors.</p> <p><strong>Significance: </strong>The MSC approach can be fruitfully applied to improve risk stratification for LCOS after CABG likely because MSC markers describe the dysfunction of the sympathetic control and the impairment of the mechanical properties of the heart in the LCOS group.</p>
Data set from the article Vaini E, Bari V, Fantinato A, Pistuddi V, Cairo B, De Maria B, Ranucci M, Porta A. Causality analysis reveals the link between cerebrovascular control and acute kidney dysfunction after coronary artery bypass grafting. Physiol Meas. 2019 Jul 1;40(6):064006. doi: 10.1088/1361-6579/ab21b1. PMID: 31091519.
<p>Data set from the article Vaini E, Bari V, Fantinato A, et al. Causality analysis reveals the link between cerebrovascular control and acute kidney dysfunction after coronary artery bypass grafting. <em>Physiol Meas</em>. 2019;40(6):064006. Published 2019 Jul 1. doi:10.1088/1361-6579/ab21b1</p> <p>This is the abstract:</p> <p><strong>Background: </strong>Patients undergoing coronary artery bypass graft (CABG) surgery might experience postoperative complications and some of them, such as acute kidney dysfunction (AKD), are the likely consequence of hypoperfusion. We hypothesized that an impaired cerebrovascular control is a hallmark of a vascular damage that might favor AKD after CABG.</p> <p><strong>Objective: </strong>Our aim is to characterize cerebrovascular control in CABG patients through the assessment of the relationship between mean arterial pressure (MAP) and mean cerebral blood flow velocity (MCBFV) and to check whether markers describing MCBFV-MAP dynamical interactions could identify subjects at risk to develop postoperative AKD.</p> <p><strong>Approach: </strong>MAP and MCBFV beat-to-beat series were extracted from invasive arterial pressure and transcranial Doppler recordings acquired simultaneously in 23 patients just before CABG after the induction of propofol general anesthesia. Subjects were divided into AKD group (n = 9, age: 68 ± 9, 8 males) and noAKD group (n = 14, age: 65 ± 8, 12 males) according to whether they developed postoperative AKD or not after CABG. We computed MAP and MCBFV time-domain and spectral markers as well as MCBFV-MAP cross-spectral indexes in very-low-frequency (VLF, 0.02-0.07 Hz), low-frequency (LF, 0.07-0.15 Hz) and high-frequency (HF, 0.15-0.30 Hz) bands. We also calculated model-based transfer entropy (TE) to quantify the degree of MCBFV dependence on MAP and vice versa. The null hypothesis of MCBFV-MAP uncoupling was tested via a surrogate approach associating MAP and MCBFV in different patients.</p> <p><strong>Main results: </strong>Time, spectral and cross-spectral markers had a limited power in separating AKD from noAKD individuals. Conversely, TE from MAP to MCBFV was significantly above the level set by surrogates only in AKD groups and significantly larger than that computed in noAKD.</p> <p><strong>Significance: </strong>The reduced cerebrovascular autoregulation in AKD patients suggest a vascular impairment likely making them more at risk of hypoperfusion during CABG and AKD after CABG.</p>
Data set from Barbic F, Minonzio M, Cairo B, Shiffer D, Dipasquale A, Cerina L, Vatteroni A, Urechie V, Verzeletti P, Badilini F, Vaglio M, Iatrino R, Porta A, Santambrogio M, Gatti R, Furlan R. Effects of different classroom temperatures on cardiac autonomic control and cognitive performances in undergraduate students. Physiol Meas. 2019 Jun 4;40(5):054005. doi: 10.1088/1361-6579/ab1816. PMID: 30970334.
<p>Data set from Barbic F, Minonzio M, Cairo B, Shiffer D, Dipasquale A, Cerina L, Vatteroni A, Urechie V, Verzeletti P, Badilini F, Vaglio M, Iatrino R, Porta A, Santambrogio M, Gatti R, Furlan R. Effects of different classroom temperatures on cardiac autonomic control and cognitive performances in undergraduate students. Physiol Meas. 2019 Jun 4;40(5):054005. doi: 10.1088/1361-6579/ab1816. PMID: 30970334.</p> <p> </p> <p>This is the abstract:</p> <p><strong>Objective: </strong> Indoor microclimate may affect students' wellbeing, cardiac autonomic control and cognitive performance with potential impact on learning capabilities. To assess the effects of classroom temperature variations on the autonomic profile and students' cognitive capabilities.</p> <p><strong>Approach: </strong> Twenty students attending Humanitas University School, (14M, age 21 ± 3 years) underwent a single-lead ECG continuous recording by a portable device during a 2 h lecture when classroom temperature was set 'neutral' (20 °C-22 °C, Day 1) and when classroom temperature was set to 24 °C-26 °C (Day 2). ECGs were sent by telemetry to a server for off-line analysis. Spectral analysis of RR variability provided indices of cardiac sympathetic (LF<sub>nu</sub>), vagal (HF, HF<sub>nu</sub>) and cardiac sympatho-vagal modulation (LF/HF). Symbolic analysis of RR variability provided the percentage of sequences of three heart periods with no significant change in RR interval (0V%) and with two significant variations (2V%) reflecting cardiac sympathetic and vagal modulation, respectively. Students' cognitive performance (memory, verbal comprehension and reasoning) was assessed at the end of each lecture using the Cambridge Brain Sciences cognitive evaluation tool.</p> <p><strong>Main results: </strong> Classroom temperature and CO<sub>2</sub> were assessed every 5 min. Classroom temperatures were 22.4 °C ± 0.1 °C (Day 1) and 26.2 °C ± 0.1 °C (Day 2). Student's thermal comfort was lower during Day 2 compared to Day 1. HR, LF/HF and 0V% were greater during Day 2 (79.5 ± 12.1 bpm, 6.9 ± 7.1 and 32.8% ± 10.3%) than during Day 1 (72.6 ± 10.8 bpm, 3.4 ± 3.7, 21.4% ± 9.2%). Conversely, 2V% was lower during Day 2 (23.1% ± 8.1%) than during Day 1 (32.3% ± 11.4%). Short-term memory, verbal ability and the overall cognitive C-score scores were lower during Day 2 (10.3 ± 0.3; 8.1 ± 1.2 and 10.9 ± 2.0) compared to Day 1 (11.7 ± 2.1; 10.7 ± 1.7 and 12.6 ± 1.8).</p> <p><strong>Significance: </strong> During Day 2, a shift of the cardiac autonomic control towards a sympathetic predominance was observed compared to Day 1, in the presence of greater thermal discomfort. Furthermore, during Day 2 reduced cognitive performances were found.</p>
ScienceDex guides
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These curated guides explain access requirements, typical timelines, costs, and reuse considerations for widely used research datasets.
Allen Brain Atlas
Allen Brain Atlas is an Allen Institute collection of brain map atlases, datasets, APIs, and analysis tools covering mouse, human, and non-human primate brain resources.
Annotated Behaviour and Observability Dataset (ABODe)
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DANDI Archive for NWB datasets
DANDI is a BRAIN Initiative archive for publishing and sharing neurophysiology data, including electrophysiology, optophysiology, and behavioral data packaged as NWB and related standards.
International Brain Laboratory public data
The International Brain Laboratory public data releases expose standardized mouse decision-making experiments, including Neuropixels recordings, widefield calcium imaging, behavior, and session metadata accessed through the ONE API.
OpenNeuro
OpenNeuro is a free, open platform for sharing neuroimaging datasets, with public search, dataset pages, and download paths for web, S3, DataLad, and the OpenNeuro CLI.