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Dataset results
255 results for “peanut”
Intervention to Reduce Early (Peanut) Allergy in Children
ClinicalTrials.gov study NCT04604431. IPD Sharing: NO. Countries: 1. Publications: 3.
Determining the Efficacy and Value of Immunotherapy on the Likelihood of Peanut Tolerance: The DEVIL Study
ClinicalTrials.gov study NCT00932828. IPD Sharing: NO. Countries: 1. Publications: 1.
Peanut Oral Immunotherapy Study of Early Intervention for Desensitization
ClinicalTrials.gov study NCT03736447. IPD Sharing: UNDECIDED. Countries: 4. Publications: 1.
A Study of Sublingual Immunotherapy in Peanut-allergic Children
ClinicalTrials.gov study NCT00597727. IPD Sharing: NO. Countries: 1. Publications: 3.
Follow-up of the VIPES Study to Evaluate Efficacy and Safety of Viaskin Peanut in Adults and Children
ClinicalTrials.gov study NCT01955109. IPD Sharing: Not stated. Countries: 4. Publications: 1.
Peanuts and Glycemic Control
ClinicalTrials.gov study NCT03654651. IPD Sharing: NO. Countries: 1. Publications: 1.
Peanut Allergy Oral Immunotherapy Study of AR101 for Desensitization in Children and Adults (PALISADE)
ClinicalTrials.gov study NCT02635776. IPD Sharing: NO. Countries: 10. Publications: 5.
Persistence of Oral Tolerance to Peanut
ClinicalTrials.gov study NCT01366846. IPD Sharing: YES. Countries: 1. Publications: 4.
Efficacy and Safety of Viaskin Peanut in Children With Immunoglobulin E (IgE)-Mediated Peanut Allergy
ClinicalTrials.gov study NCT02636699. IPD Sharing: Not stated. Countries: 5. Publications: 4.
Predicting Peanut Anaphylaxis and Reducing Epinephrine
ClinicalTrials.gov study NCT05696236. IPD Sharing: NO. Countries: 1. Publications: 1.
A Randomized, Double-Blind, Placebo-Controlled Pilot Study of Sublingual/Oral Immunotherapy for the Treatment of Peanut Allergy
ClinicalTrials.gov study NCT01084174. IPD Sharing: NO. Countries: 1. Publications: 1.
Promoting Tolerance to Peanut in High-Risk Children
ClinicalTrials.gov study NCT00329784. IPD Sharing: YES. Countries: 1. Publications: 9.
Data from: Gastrointestinal gd T cells reveal upregulated T-cell transcripts and signaling pathways during peanut oral immunotherapy
Open the record for dataset details and reuse information.
Data from: Understanding the variability of the peanut-oral immunotherapy response through multi-omics profiling of immune cells
Open the record for dataset details and reuse information.
Novel digital assessment technique used to describe peanut nodulation life history
<p>Biological nitrogen fixation (BNF) in legume root nodules is an important source of N globally. Typically, the quantification of BNF is an estimate of N fixation through techniques such as an acetylene reduction assay or isotopic signatures. However, these methods have large logistical limitations, and do not allow for assessments of the nodule morphology or the nodulation process itself, knowledge that is important to fully understand the BNF process. To provide an updated method of nodulation assessments, peanut (<i>Arachis hypogaea </i>L.) nodules were sampled from a two-year field trial and quantified using scanned images of excised nodules, allowing digital assessment of nodule number and size. Further, a digital color analysis method was developed to assess internal nodule color (a known proxy for nodule BNF activity and senescence). To apply and validate the new method, peanut <span>nodulation</span> trait data was used for an exploration of the relationship between nodulation traits, plant physiological processes, and nitrogen status in the plant, followed by a nodulation life history analysis. The nodulation assessment method was validated by strong correlation with measures of peanut tissue N, biomass, photosynthetic function, and yield. Nodulation life history data suggest novel findings regarding differences in the development of nodules between taproots and lateral roots, contrary to the previous understanding of peanut nodulation life history. This new nodulation assessment method can provide further insight into the BNF process by allowing the collection of detailed data in a relatively simplified manner in field conditions. This dataset represents the nodulation and other variable data collected for this study.</p>
Peanut Box
For more information about this item visit: https://bhpsite.org/ Source: Objaverse 1.0 / Sketchfab
SNPs and InDels identified in 390 peanut accessions
<p>SNPs and InDels identified in 390 peanut accessions</p>
Figure 1 Symptoms seen on peanut leaves inoculated with P. arachidicola conidia suspensions.
<p><strong><span>Figure 1</span></strong><span> Symptoms seen on peanut leaves inoculated with <em>P. arachidicola</em> conidia suspensions. The selected peanut leaves were spray inoculated with <em>P. arachidicola</em> conidia suspensions and their phenotypes were observed at 0, 7, 14, and 21dpi.</span></p> <p><strong><span>Figure 2 </span></strong><span>Gene characterization and analysis for differential expressed genes. (A). Venn dialog of expressed genes from genome analysis. Venn diagrams depict the numbers of expressed genes in peanut leaves that were infected by <em>P. arachidicola </em>at 7 (JZ7), 14 (JZ14), 21(JZ21) dpi. The numbers indicate the counts of common or unique expressed genes. (B). Heatmap of DEGs (differentially expressed genes). Red spots mean up-regulated genes; blue indicates down-regulated genes. Blue spots indicate down-DEGs, Red spots indicate up-DEGs. In the red-blue scale, darker red colors indicate the most upregulated and the darker blue the most downregulated genes. (C). Go analysis for gene functional classifcation shows the number of genes involved in the biological process, cellular component and molecular function, respectively. (D). KEGG pathway enrichments of DEGs. The panel shows 20 enriched KEGG terms for DEGs responding to <em>P. arachidicola </em>infection. The legend is shown on the picture.</span></p> <p><strong><span>Figure 3</span></strong><span> Analysis of IFS protein function (A). Functional domains, phylogenetic analysis, and multiple sequence alignment of the two AhIFS proteins. (B). Functional domain schematic for the AhIFS proteins. The predicted functional domains (http://smart.embl-heidelberg.de/) indicated that both proteins contained P450 domains. (C).Phylogenetic tree analysis of the AhIFS orthologs. Close orthologs from <em>Arachis hypogaea</em> and other plants, including MpIFS1 (<em>Mucuna pruriens</em>, RDX63227.1), PsIFS1 (<em>Pisum sativum</em>, AAQ10282.2), GmIFS1 (<em>Glycine max</em>, NP_001236022.1), and GsIFS1 (<em>Glycine soja</em>, ACA81484.1), were used for IFS protein sequence alignment. An amino acid identity of 100% is indicated by a black background; amino acids with conservation exceeding 50% are in black on a gray background; and similar amino acids are in white on a gray background.</span></p> <p><strong><span>Figure 4 </span></strong><span>(A).The flavonoids and isoflavones biosynthesis pathways. Enzymes are indicated in uppercase letters. PAL, phenylalanine ammonia-lyase; C4H, cinnamate 4-hydroxylase; 4CL, 4-coumarate: CoA ligase; CHS, chalcone synthase; CHI, chalcone isomerase; IFS, isoflavone synthase; The expression of IFS genes refers to the expression of genes at three infection time points(7,14,21day) in the transcriptome. (B, C).Transcriptional expression of AhIFS genes at the infection stage. The y- and x-axes represent the relative expression levels and time course for <em>P. arachidicola</em> infection, respectively. Mean values and standard deviations (SDs) were obtained from three biological and three technical replicates. Error bars are SDs; time points are 0, 7, 14, and 21hpi; and asterisks indicate that the expression level was significantly different from the control (*P< 0.05, **P< 0.01).</span></p> <p><strong><span>Figure 5</span></strong><span> Overexpression of AhIFSs boosts resistance of <em>N. benthamiana </em>to <em>P. parasitica</em>. (A). Representative photographs of <em>N. benthamiana </em>leaves under infection. The constructs, pBINPLUS:AhIFSs and pBINPLUS:GFP, were transiently expressed in <em>N. benthamiana </em>leaves. (A). tumefaciens-mediated transient expression of CK (right) and AhIFSs (left) fusion proteins in <em>N. benthamiana </em>leaves. The infiltrated leaves (after 48 h) were inoculated with <em>P. parasitica </em>zoospores, and photographed at 36 hpi and48 hpi, respectively. (B). Lesion diameters (mm) of <em>N. benthamiana </em>leaves. The data was calculated from three independent biological replicates using at least eight leaves each (** P < 0.01 compared with GFP; Dunnett’s test).</span></p> <p><strong><span>Figure 6</span></strong><span> Position of target sites on the <em>AhIFS1</em> and <em>AhIFS2</em> genes, and identifcation of the mutant lines produced using the CRISPR/Cas9 system. (A). CRISPR/Cas9 sgRNA-1 and sgRNA-2 target the first exon of <em>AhIFS1</em> and <em>AhIFS2</em> genes.<span> </span>The ATTG is the initial recognition motif</span><span>,</span><span>and the protospacer adjacent motif (PAM) is underlined in red. Gray boxes, blue boxes and black lines indicate UTRs, exons and introns, respectively. (B). The edited sequences at the target site are shown in HY23 and mutation lines. The blue nucleotides indicate SNP in the mutant <em>ahifs1-5/16</em>, <em>ahifs2-5/11</em></span><span>,</span><em><span>ahifs2-26</span></em><span> and the blue ‘-’ indicates the deletions between HY23 and mutant ahifs1-21. The target sequence is shown by the red underline, and nucleotides marked in red represent PAM. (C). Relative expression of <em>AHIFS</em> genes in the mutant. (D). Knockout of AhIFSs genes reduce its disease resistance. The symptoms of peanut mutant leaves inoculated with <em>P. arachidicola </em>conidia suspensions. The detached leaves of peanut were sprayed inoculation with <em>P. arachidicola </em>conidia suspensions and phenotypes were observed at 14 dpi.</span></p>
FIGURE 6. A–B. Lithacrosiphon cristatus. A in Peanut worms of the phylum Sipuncula from the Nha Trang Bay (South China Sea) with a key to species
FIGURE 6. A–B. Lithacrosiphon cristatus. A) lateral view, introvert everted; B) hooks. C–D. Cloesiphon aspergillus. C) lateral view, introvert retracted; D) hook. Scale bars: A, 5 mm; B, 20 µm; C, 2 mm; D, 20 µm.
FIGURE 4. A in Peanut worms of the phylum Sipuncula from the Nha Trang Bay (South China Sea) with a key to species
FIGURE 4. A. Antillesoma antillarum, lateral view, introvert everted. B–D. Apionsoma misakianum. B) lateral view, introvert everted; C) lateral view, introvert retracted; D) hook. E–F. A. trichocephalus. E) head (white arrowhead - rudimental tentacle on dorsal arc.); F) hook. Scale bars: A–B, 5 mm; C, 2 mm; D, 20 µm; E, 100 µm; F, 20 µm.
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Allen Brain Atlas
Allen Brain Atlas is an Allen Institute collection of brain map atlases, datasets, APIs, and analysis tools covering mouse, human, and non-human primate brain resources.
Annotated Behaviour and Observability Dataset (ABODe)
ABODe is a University of Edinburgh DataShare dataset for behavior classification in group-housed mice using home-cage video, identities, bounding boxes, ground-plate positions, and annotator labels.
DANDI Archive for NWB datasets
DANDI is a BRAIN Initiative archive for publishing and sharing neurophysiology data, including electrophysiology, optophysiology, and behavioral data packaged as NWB and related standards.
International Brain Laboratory public data
The International Brain Laboratory public data releases expose standardized mouse decision-making experiments, including Neuropixels recordings, widefield calcium imaging, behavior, and session metadata accessed through the ONE API.
OpenNeuro
OpenNeuro is a free, open platform for sharing neuroimaging datasets, with public search, dataset pages, and download paths for web, S3, DataLad, and the OpenNeuro CLI.