Find research datasets worth reusing

opencc-by-4.0Apr 2019View details →

Fig. 1 in Evidence of red panda as an intermediate host of Toxoplasma gondii and Sarcocystis species

Fig. 1. Sarcocysts and Toxoplasma gondii cysts in red panda or mice. A: Tissue cysts, leg muscle, red panda. Two oval cysts (arrow) were observed in the skeletal muscle cell. The walls of the two cysts (arrowhead) were deeply stained by eosin. Red panda, H&E. B: Partial magnification of figure A. a, The wall of the cyst was clearly and deeply stained by eosin. b, The bradyzoites were like cresent or banana, they were arranged in packets (arrowhead). c, Necleus of host cell. C: Tissue cyst (arrow) was cross reacted with T. gondii, the cysts were separated by septa and formed many compartments (arrowhead), leg muscle. T. gondii antibody, red panda, IHC. D: Tissue cysts of T. gondii in brain of mouse (arrows), brain squash, unstained, 75DPI. (For interpretation of the references to colour in this figure legend, the reader is referred to the Web version of this article.)

opencc-by-4.0Apr 2019View details →

Fig. 1 in Toxoplasma gondii in four captive kangaroos (Macropus spp.) in China: Isolation of a strain of a new genotype from an eastern grey kangaroo (Macropus giganteus)

Fig. 1. Toxoplasma gondii cysts in kangaroos or mice. A. Toxoplasma gondii cysts in the diaphragm of case 2 kangaroo, H&E. B. Toxoplasma gondii cysts in the tongue of case 2 kangaroo, H&E. C. Toxoplasma gondii cysts in the diaphragm of case 2 kangaroo, IHC. D. Toxoplasma gondii cysts in the tongue of case 2 kangaroo, IHC. E. Toxoplasma gondii-like cysts in the myocardium of case 4 kangaroo, squashed section, unstained. F. Many TgRooCHn1 Toxoplasma gondii cysts were observed in the mouse brain, 27 DPI, squashed section, unstained. Bar = 50 μm.

opencc-by-4.0Aug 2020View details →

Fig. 1 in Serological evidence of Toxoplasma gondii infection in Melanosuchus niger (Spix, 1825) and Caimam crocodilus (Linnaeus, 1758)

Fig. 1. Venn diagram displaying positivity of alligator serum samples to Toxoplasma gondii analyzed by serological methods MAT and IHA.

opencc-by-4.0Aug 2020View details →

Fig. 3 in Comparison of the modified agglutination test and real-time PCR for detection of Toxoplasma gondii exposure in feral cats from Phillip Island, Australia, and risk factors associated with infection

Fig. 3. Predicted lines of fit for the multivariable logistic regression model plotted as probability of Toxoplasma gondii qPCR positivity in feral cats on Phillip Island (Victoria) versus body weight for each season. Dashed lines show 95% confidence intervals.

opencc-by-4.0Aug 2020View details →

Fig. 1 in Comparison of the modified agglutination test and real-time PCR for detection of Toxoplasma gondii exposure in feral cats from Phillip Island, Australia, and risk factors associated with infection

Fig. 1. Location and Toxoplasma gondii infection status, as detected by real-time PCR (qPCR), of feral cats trapped on Phillip Island (Victoria) from July 2016 to December 2017. Map shows the distribution of different location types (Park, Agricultural, Residential) used in multivariable regression analysis. A circular spread of points around a location marked with 'x' indicates multiple animals were sampled at the same site (i.e. same GPS coordinates). Red = T. gondii qPCR positive, white = T. gondii qPCR negative. Map created using Quantum GIS, version 3.8. (For interpretation of the references to colour in this figure legend, the reader is referred to the Web version of this article.)

opencc-by-4.0Dec 2020View details →

Fig. 2 in Toxoplasma gondii infection in European mouflons (Ovis musimon) and captive wild felines from Puebla, M�exico

Fig. 2. Representative PCR-RFLP pattern of T. gondii for SAG3 gene. Single and mixed infections in lions and mouflons tissues were observed. A. PCR for the SAG3 locus of lion 1 (L1, spleen), mouflon 1 (M1, brain) and mouflon 2 (M2, liver) samples. B. A triple infection is highlighted (yellow box). Resulting genotypes are specified at the bottom. In silico digestion was done by www.benchling.com. Reference strains sequences GT1, TGGT1_308020; Me49, TGME49_308020; VEG, TGVEG_308020 are available at www.toxodb.org. MW: molecular weight marker; RH and ME49 are reference strains, type I and II, respectively; M: mouflon, L: lion. (For interpretation of the references to colour in this figure legend, the reader is referred to the Web version of this article.)

opencc-by-4.0Dec 2020View details →

Fig. 1 in Toxoplasma gondii infection in European mouflons (Ovis musimon) and captive wild felines from Puebla, M�exico

Fig. 1. Toxoplasma gondii infection in captive mouflons in 2011 and 2012. A. Anti-T. gondii frequency distribution in fifty-five sera of mouflons sampled in 2011; black arrows indicate positive samples as determined by their position to the right of the normally distributed values of the left population. B. One year later, 41/55 original mouflons were captured, bled, and re-tested for antibodies against this parasite. The cut-off point used was 1.0 RI (dotted lines), which separated the negative population (left) from the positive cases. C. Three mouflons remained positive, five became negative, and four seroconverted positive in 2012 (red, green and blue dots, respectively); one mouflon is on the cut-off (orange dot). D. Immunohistochemistry for T. gondii in tissues from in the spleen of mouflon 1, where an immunopositive cumulus of tachyzoites can be seen (blue arrow). The nuclei of resident lymphocytes and dendritic cells were contrasted with Meyer's hematoxylin. Bar: 50 μm. R = Pearson correlation. (For interpretation of the references to colour in this figure legend, the reader is referred to the Web version of this article.)

opencc-by-4.0Dec 2021View details →

Fig. 2 in Toxoplasma gondii contamination at an animal agriculture facility: Environmental, agricultural animal, and wildlife contamination indicator evaluation

Fig. 2. Soil sampling and animal trapping locations at Little River Animal and Environmental Unit in Walland, Tennessee, United States.

opencc-by-4.0Dec 2021View details →

Fig. 1 in Toxoplasma gondii contamination at an animal agriculture facility: Environmental, agricultural animal, and wildlife contamination indicator evaluation

Fig. 1. The geographic location of the study site, Little River Animal and Environmental Unit in Walland, Tennessee, United States.

Fig. 5. A data-display network constructed from uncorrected 18S rDNA p in Toxoplasma gondii and related Sarcocystidae parasites in harvested caribou from Nunavik, Canada

Fig. 5. A data-display network constructed from uncorrected 18S rDNA p-distances, using all characters, for tissue dwelling coccidians (mostly Sarcocystis spp.). Group names bear no taxonomic designation but merely assigned for discussion purposes. Bootstrap supports are displayed by the gray curves and associated values imposed on the network. Red dots indicate sequences generated in this study. (For interpretation of the references to colour in this figure legend, the reader is referred to the Web version of this article.)

Fig. 4 in Toxoplasma gondii and related Sarcocystidae parasites in harvested caribou from Nunavik, Canada

Fig. 4. Maximum likelihood topology for tissue dwelling coccidians (mostly Sarcocystis spp.) generated from 18S rDNA sequence data analyzed in RAxML 8 under the GTRCAT approximation. Group names bear no taxonomic designation but merely assigned for discussion purposes. Bootstrap values> 60 are displayed above branches as branch/node support. Red dots indicate sequences generated in this study. (For interpretation of the references to colour in this figure legend, the reader is referred to the Web version of this article.)

Fig. 2 in Toxoplasma gondii and related Sarcocystidae parasites in harvested caribou from Nunavik, Canada

Fig. 2. Observed seroprevalence of antibodies to Toxoplasma gondii and Neospora caninum in caribou harvested from 2 communities in Nunavik. Source: Nunavik Research Centre, Makivik Corporation.

Fig. 1 in Toxoplasma gondii and related Sarcocystidae parasites in harvested caribou from Nunavik, Canada

Fig. 1. General life cycle of Sarcocystidae parasites. 1, For Toxoplasma gondii and Neospora caninum, oocysts are shed in feces by definitive hosts (DH, felids and canids, respectively), sporulate and become infective in the environment, whereas for Sarcocystis spp., sporulation occurs in the intestine of the definitive hosts and sporocysts are immediately infective for intermediate hosts. 2, Intermediate hosts (IH) ingest sporulated oocysts or sporocysts in food, water or soil. 3, Sporozoites are released, divide rapidly, and tachyzoites disseminate to somatic tissues of the IH, and form tissue cysts. 4, DH becomes infected by ingesting prey species with bradyzoites within tissue cysts. Created with BioR ender.com.

Fig. 3 in Toxoplasma gondii and related Sarcocystidae parasites in harvested caribou from Nunavik, Canada

Fig. 3. Sarcocystis DNA prevalence in heart and muscle of caribou harvested by 2 communities in Nunavik, Qu´ebec, Canada.

Fig. 1 in Nasal vaccination of six squirrel monkeys (Saimiri sciureus): Improved immunization protocol against Toxoplasma gondii with a nanoparticle-born vaccine

Fig. 1. Schedule of the vaccinal protocol and of the immunological analysis performed on the 6 Saimiris.

Fig. 2. T in Nasal vaccination of six squirrel monkeys (Saimiri sciureus): Improved immunization protocol against Toxoplasma gondii with a nanoparticle-born vaccine

Fig. 2. T-cell immune response analyzed by IFN-γ ELISPOT on PBMC from 6 Saimiris. The results are presented as Spot Forming Units for 106 PBMC (left), before the immunization (T0), one month after the prime, 5 months after the 1st boost and 2 months after the 2nd boost. A representative picture of the ELISPOT plate after the 2nd boost is presented (right). Only 4 animals were analyzed by ELISPOT after the 2nd boost due to blood coagulation in the sampling tubes. Statistical analyses were made by KruskalWallis test, * p <0.05, ** p <0.01.

Fig. 3 in Nasal vaccination of six squirrel monkeys (Saimiri sciureus): Improved immunization protocol against Toxoplasma gondii with a nanoparticle-born vaccine

Fig. 3. Humoral immune response analyzed by ELISA on serum for each Saimiri. The results are presented as optical density (OD) before the immunization (T0), and 2 months after the 2nd boost. Serum from one seropositive and three seronegative humans were used as positive and negative controls, respectively. Cut-off was determined at each dilution, as the mean + 2.5xSD of the negative controls.

Fig. 1 in Presence of IgG antibodies is not a reliable marker of Toxoplasma gondii infection in feral mice

Fig. 1. Frequency distribution of body weight in Toxoplasma gondii B1 PCRnegative (in green) and positive mice (in red). (For interpretation of the references to colour in this figure legend, the reader is referred to the Web version of this article.)

opencc-by-4.0Dec 2022View details →

Fig. 6 in Role of landscape context in Toxoplasma gondii infection of invasive definitive and intermediate hosts on a World Heritage Island

Fig. 6. Relative density of black rats in different land-use types, as expressed by the capture rate by traps, i.e., number of individuals captured per 100 trapdays. Numerals above the bars indicate total trap-days.